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Neurodegeneration & Neurodegenerative Diseases


219 Feeds
Brain organoids are three-dimensional in vitro cellular models of the brain that can recapitulate many processes such as the neurodevelopment. In addition, these organoids can be combined with other cell types, such as neurons and astrocytes to study their interactions in assembloids. Disease processes can also be modeled by induced pluripotent stem cell-derived organoids and assembloids from patients with neurodegenerative disorders. Discover the latest research on the models here.
3D Cellular Models of Brain and Neurodegeneration
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Brain organoids are three-dimensional in vitro cellular models of the brain that can recapitulate many processes such as the neurodevelopment. In addition, these organoids can be combined with other cell types, such as neurons and astrocytes to study their interactions in assembloids. Disease processes can also be modeled by induced pluripotent stem cell-derived organoids and assembloids from patients with neurodegenerative disorders. Discover the latest research on the models here.

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TAR DNA-binding protein 43 (TDP-43) is a pathological protein identified in sporadic Amyotrophic Lateral Sclerosis (ALS) and Frontotemporal Dementia (FTD). Here are the latest discoveries pertaining to TDP-43 and these diseases.
ALS & FTD: TDP-43
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TAR DNA-binding protein 43 (TDP-43) is a pathological protein identified in sporadic Amyotrophic Lateral Sclerosis (ALS) and Frontotemporal Dementia (FTD). Here are the latest discoveries pertaining to TDP-43 and these diseases.

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Amyotrophic Lateral Sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.
ALS: Genetics

Amyotrophic Lateral Sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.

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Amyotrophic Lateral Sclerosis is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.
ALS: Pathogenic Mechanisms
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Amyotrophic Lateral Sclerosis is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.

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Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disorder characterized phenotypically by progressive muscle weakness. Clinical phenotypes of ALS can be classified based on the pattern, level, and area of onset (e.g. bulbar, cervical, lumbar). Here is the latest research investigating phenotypes of ALS.
ALS: Phenotypes
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Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disorder characterized phenotypically by progressive muscle weakness. Clinical phenotypes of ALS can be classified based on the pattern, level, and area of onset (e.g. bulbar, cervical, lumbar). Here is the latest research investigating phenotypes of ALS.

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Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here is the latest research on ALS and prions.
ALS: Prions
Going Viral

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here is the latest research on ALS and prions.

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Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research in this field.
ALS: Stress Granules

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research in this field.

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Amyotrophic Lateral Sclerosis (ALS) is associated with the death of neurons that control voluntary muscles. This feed followes the latest research into therapies for this progressive neurodegenerative disease.
ALS: Therapies

Amyotrophic Lateral Sclerosis (ALS) is associated with the death of neurons that control voluntary muscles. This feed followes the latest research into therapies for this progressive neurodegenerative disease.

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Dementias are a group of conditions, including Alzheimer's disease, vascular dementia, and frontotemporal dementia, characterized by deficiencies in cognitive abilities. Age-related dementia refers to dementias that occur in older individuals, usually 60+ years old, in contrast to early-onset dementia. Follow the latest research on age-related dementia here.
Age-related Dementia

Dementias are a group of conditions, including Alzheimer's disease, vascular dementia, and frontotemporal dementia, characterized by deficiencies in cognitive abilities. Age-related dementia refers to dementias that occur in older individuals, usually 60+ years old, in contrast to early-onset dementia. Follow the latest research on age-related dementia here.

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Alexander disease is a rare leukodystrophy caused by mutations in the astrocyte-specific intermediate filament protein glial fibrillary acidic protein (GFAP). Here is the latest research on this disease.
Alexander Disease
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Alexander disease is a rare leukodystrophy caused by mutations in the astrocyte-specific intermediate filament protein glial fibrillary acidic protein (GFAP). Here is the latest research on this disease.

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Alpha-synucleins are small proteins that are believed to restrict the mobility of synpatic vesicles and inhibit neurotransmitter release. Aggregation of these proteins have been linked to several types of neurodegenerative diseases including dementia with Lewy bodies and Parkinson's disease. Here is the latest research on α-synuclein aggregation.
Alpha-Synuclein Aggregation

Alpha-synucleins are small proteins that are believed to restrict the mobility of synpatic vesicles and inhibit neurotransmitter release. Aggregation of these proteins have been linked to several types of neurodegenerative diseases including dementia with Lewy bodies and Parkinson's disease. Here is the latest research on α-synuclein aggregation.

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α-Synuclein is an integral component of Lewy bodies which are comprised of protein clumps and are a pathological hallmark of Parkinson’s disease. Here is the latest research on α-synuclein structure and function.
Alpha-Synuclein Structure & Function
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α-Synuclein is an integral component of Lewy bodies which are comprised of protein clumps and are a pathological hallmark of Parkinson’s disease. Here is the latest research on α-synuclein structure and function.

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Apolipoprotein E (APOE) polymorphic alleles are major genetic risk factors for Alzheimer's disease. Discover the latest research on APOE and other genetic determinants of Alzheimer's disease here.
Alzheimer's Disease: APOE
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Apolipoprotein E (APOE) polymorphic alleles are major genetic risk factors for Alzheimer's disease. Discover the latest research on APOE and other genetic determinants of Alzheimer's disease here.

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Amyloid precursor protein (APP) proteolysis is critical for the development of Alzheimer's disease, a neurodegenerative disease associated with accumulation of amyloid plaques in the brain. Here is the latest research on APP and Alzheimer's disease.
Alzheimer's Disease: APP

Amyloid precursor protein (APP) proteolysis is critical for the development of Alzheimer's disease, a neurodegenerative disease associated with accumulation of amyloid plaques in the brain. Here is the latest research on APP and Alzheimer's disease.

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Alzheimer's disease is a neurodegenerative disease associated with the accumulation of amyloid plaques in the brain; these plaques are comprised of amyloid beta deposits. Here is the latest research in this field.
Alzheimer's Disease: Amyloid Beta

Alzheimer's disease is a neurodegenerative disease associated with the accumulation of amyloid plaques in the brain; these plaques are comprised of amyloid beta deposits. Here is the latest research in this field.

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Alzheimer's disease is a neurodegenerative disease which can be studied using various experimental systems. This feed focuses on animal models used for Alzheimer's disease research.
Alzheimer's Disease: Animal Models
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Alzheimer's disease is a neurodegenerative disease which can be studied using various experimental systems. This feed focuses on animal models used for Alzheimer's disease research.

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Alzheimer's disease (AD) is a neurodegenerative disease characterized by progressive cognitive and behavioral decline. Targeting markers in the earliest stages of the disease may mitigate the progression of AD. This feed focuses on early diagnosis and markers, as well as environmental, pharmacological, and drug-response biomarkers associated with this disease.
Alzheimer's Disease: Early Markers

Alzheimer's disease (AD) is a neurodegenerative disease characterized by progressive cognitive and behavioral decline. Targeting markers in the earliest stages of the disease may mitigate the progression of AD. This feed focuses on early diagnosis and markers, as well as environmental, pharmacological, and drug-response biomarkers associated with this disease.

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Dysfunctional endosomal trafficking may be associated with Alzheimer’s disease (AD) pathology. Targeting the endosome may advance treatment options for AD. Here is the latest research on endosomes and AD.
Alzheimer's Disease: Endosomes

Dysfunctional endosomal trafficking may be associated with Alzheimer’s disease (AD) pathology. Targeting the endosome may advance treatment options for AD. Here is the latest research on endosomes and AD.

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Alzheimer's disease is a neurodegenerative disease. Discover genetic and epigenetic aspects of Alzheimer’s disease, including genetic markers and genomic structural variations with this feed.
Alzheimer's Disease: Genetics

Alzheimer's disease is a neurodegenerative disease. Discover genetic and epigenetic aspects of Alzheimer’s disease, including genetic markers and genomic structural variations with this feed.

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Variants within the membrane-spanning 4-domains subfamily A (MS4A) gene cluster have recently been implicated in Alzheimer's disease in genome-wide association studies. Here is the latest research on Alzheimer's disease and MS4A.
Alzheimer's Disease: MS4A

Variants within the membrane-spanning 4-domains subfamily A (MS4A) gene cluster have recently been implicated in Alzheimer's disease in genome-wide association studies. Here is the latest research on Alzheimer's disease and MS4A.

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Microglia are a type of glial cell found throughout the brain and spinal cord. Microglia have been found to be associated with Alzheimer's disease development and progression. Here are the latest discoveries pertaining to Alzheimer's disease and microglia.
Alzheimer's Disease: Microglia
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Microglia are a type of glial cell found throughout the brain and spinal cord. Microglia have been found to be associated with Alzheimer's disease development and progression. Here are the latest discoveries pertaining to Alzheimer's disease and microglia.

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Neuroimaging can help identify pathological hallmarks of Alzheimer's disease (AD). Here is the latest research on neuroimaging modalities, including magnetic resonance imaging and positron emission tomography, in AD.
Alzheimer's Disease: Neuroimaging

Neuroimaging can help identify pathological hallmarks of Alzheimer's disease (AD). Here is the latest research on neuroimaging modalities, including magnetic resonance imaging and positron emission tomography, in AD.

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RNA regulation involves several mechanisms that are used by cells to decrease or increase the production of RNA. Disruption of RNA regulatory processes has been associated with Alzheimer's disease (AD). Here are the latest discoveries pertaining to RNA regulation and AD.
Alzheimer's Disease: RNA Regulation
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RNA regulation involves several mechanisms that are used by cells to decrease or increase the production of RNA. Disruption of RNA regulatory processes has been associated with Alzheimer's disease (AD). Here are the latest discoveries pertaining to RNA regulation and AD.

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RNA sequencing studies have shed light on the genetic and molecular mechanisms related to the pathophysiology of Alzheimer's disease (AD). This feed follows papers using RNA sequencing technologies in Alzheimer's research.
Alzheimer's Disease: RNA Sequencing
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RNA sequencing studies have shed light on the genetic and molecular mechanisms related to the pathophysiology of Alzheimer's disease (AD). This feed follows papers using RNA sequencing technologies in Alzheimer's research.

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Alzheimer's disease is a neurodegenerative disease. This feed focuses on the underlying role of tau proteins and TAR DNA-binding protein 43, as well as other genetic factors, in Alzheimer's disease.
Alzheimer's Disease: Tau & TDP-43
Influential

Alzheimer's disease is a neurodegenerative disease. This feed focuses on the underlying role of tau proteins and TAR DNA-binding protein 43, as well as other genetic factors, in Alzheimer's disease.

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Impaired transcription is associated with the pathogenesis and progression of conditions such as Alzheimer's disease (AD). Here are the latest discoveries pertaining to transcription and AD.
Alzheimer's Disease: Transcription
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Impaired transcription is associated with the pathogenesis and progression of conditions such as Alzheimer's disease (AD). Here are the latest discoveries pertaining to transcription and AD.

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Amyloid beta is a peptide that is a major component of plaques in Alzheimer’s disease (AD). Accumulation induces neurotoxic effects including oxidative damage and mitochondrial dysfunction. Amyliod plaques are being studied as a drug target in the treatment of AD. Here is the latest research on amyloid beta deposition and degradation.
Amyloid Beta Deposition & Degradation
Influential

Amyloid beta is a peptide that is a major component of plaques in Alzheimer’s disease (AD). Accumulation induces neurotoxic effects including oxidative damage and mitochondrial dysfunction. Amyliod plaques are being studied as a drug target in the treatment of AD. Here is the latest research on amyloid beta deposition and degradation.

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Amyotrophic Lateral Sclerosis (ALS) is a progressive nervous system disease associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS here.
Amyloid Lateral Sclerosis
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Amyotrophic Lateral Sclerosis (ALS) is a progressive nervous system disease associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS here.

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Amyloid deposits around nerves can damage and disrupt smooth information processing between the brain and organs. Here is the latest research on amyloidosis-associated polyneuropathies.
Amyloidogenesis-associated Polyneuropathy
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Amyloid deposits around nerves can damage and disrupt smooth information processing between the brain and organs. Here is the latest research on amyloidosis-associated polyneuropathies.

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Antisense oligonucleotides are synthetic DNA oligomers that hybridize to a target RNA. This feed focuses on antisense oligonucleotide therapies such as Inotersen, Nusinursen, and Patisiran, in neurodegenerative diseases.
Antisense Oligonucleotide Therapies: ND

Antisense oligonucleotides are synthetic DNA oligomers that hybridize to a target RNA. This feed focuses on antisense oligonucleotide therapies such as Inotersen, Nusinursen, and Patisiran, in neurodegenerative diseases.

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Aphasia affects the ability to process language, including formulation and comprehension of language and speech, as well as the ability to read or write. Here is the latest research on aphasia.
Aphasia

Aphasia affects the ability to process language, including formulation and comprehension of language and speech, as well as the ability to read or write. Here is the latest research on aphasia.

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Apolipoprotein E (APOE) is a protein involved in fat metabolism and associated with the pathogenesis of Alzheimer's disease and cardiovascular disease. Here is the latest research on APOE phenotypes.
ApoE Phenotypes
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Apolipoprotein E (APOE) is a protein involved in fat metabolism and associated with the pathogenesis of Alzheimer's disease and cardiovascular disease. Here is the latest research on APOE phenotypes.

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Astrocytes are thought to play a role in amyloid production and have been implicated in neurodegenerative diseases such as Alzheimer’s disease. Here is the latest research on the relationship between astrocytes and amyloid.
Astrocytes & Amyloid

Astrocytes are thought to play a role in amyloid production and have been implicated in neurodegenerative diseases such as Alzheimer’s disease. Here is the latest research on the relationship between astrocytes and amyloid.

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Astrocytes are abundant within the central nervous system and their dysfunction has been thought to be an important contributor to some neurodegenerative diseases, in particular Huntington’s disease. Damage to these cells may make neurons more susceptible to degeneration. Here is the latest research on astrocytes and Huntington’s disease.
Astrocytes & Huntington’s Disease

Astrocytes are abundant within the central nervous system and their dysfunction has been thought to be an important contributor to some neurodegenerative diseases, in particular Huntington’s disease. Damage to these cells may make neurons more susceptible to degeneration. Here is the latest research on astrocytes and Huntington’s disease.

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Parkinson's disease (PD) is a neurodegenerative disorder caused by the progressive loss of dopaminergic neurons. Some PD-genes may be associated with astrocyte dysfunction. Discover the latest research on astrocytes in Parkinson's disease here.
Astrocytes in Parkinson Disease

Parkinson's disease (PD) is a neurodegenerative disorder caused by the progressive loss of dopaminergic neurons. Some PD-genes may be associated with astrocyte dysfunction. Discover the latest research on astrocytes in Parkinson's disease here.

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Astrocytes are glial cells found within the CNS and are able to regenerate new neurons. They become activated during CNS injury and disease. The activation leads to the transcription of new genes and the repair and regeneration of neurons. Discover the latest research on astrocytes in repair and regeneration here.
Astrocytes in Repair & Regeneration
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Astrocytes are glial cells found within the CNS and are able to regenerate new neurons. They become activated during CNS injury and disease. The activation leads to the transcription of new genes and the repair and regeneration of neurons. Discover the latest research on astrocytes in repair and regeneration here.

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Ataxia is a neurological condition characterized by lack of voluntary coordination of muscle movements including loss of coordination, balance, and speech. Discover the latest research on different types of ataxias here.
Ataxias
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Ataxia is a neurological condition characterized by lack of voluntary coordination of muscle movements including loss of coordination, balance, and speech. Discover the latest research on different types of ataxias here.

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Axonal Neuropathy refers to disease or dysfunction of nerves caused as a result of damage or degeneration of axons. Here is the latest research.
Axonal Neuropathies

Axonal Neuropathy refers to disease or dysfunction of nerves caused as a result of damage or degeneration of axons. Here is the latest research.

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Batten Disease is a group of nervous system disorders known as neuronal ceroid lipofuscinosis. This feed focuses on neurobiological and neuropathological aspects of this disease.
Batten Disease
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Batten Disease is a group of nervous system disorders known as neuronal ceroid lipofuscinosis. This feed focuses on neurobiological and neuropathological aspects of this disease.

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Some cytokines are able to cross the blood brain barrier through transport systems and enter the cerebrospinal fluid and interstitial fluid spaces. Here is the latest research on cytokines crossing the blood brain barrier and how this can affect tissues within the CNS.
Blood Brain Barrier & Cytokines

Some cytokines are able to cross the blood brain barrier through transport systems and enter the cerebrospinal fluid and interstitial fluid spaces. Here is the latest research on cytokines crossing the blood brain barrier and how this can affect tissues within the CNS.

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The blood brain barrier is essential in regulating the movement of molecules and substances in and out of the brain. Disruption to the blood brain barrier and changes in permeability allow pathogens and inflammatory molecules to cross the barrier and may play a part in the pathogenesis of neurodegenerative disorders. Here is the latest research in this field.
Blood Brain Barrier Regulation in Health & Disease
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The blood brain barrier is essential in regulating the movement of molecules and substances in and out of the brain. Disruption to the blood brain barrier and changes in permeability allow pathogens and inflammatory molecules to cross the barrier and may play a part in the pathogenesis of neurodegenerative disorders. Here is the latest research in this field.

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The blood brain barrier is important for regulating the movement of biomolecules in and out of the brain. For example, membrane transporters in the blood brain barrier can be essential for regulating drug movement and dysregulation of these processes may play a role in neurodegeneration. This feed follows the latest research on this topic.
Blood-Brain Barrier Transport in Neurodegeneration

The blood brain barrier is important for regulating the movement of biomolecules in and out of the brain. For example, membrane transporters in the blood brain barrier can be essential for regulating drug movement and dysregulation of these processes may play a role in neurodegeneration. This feed follows the latest research on this topic.

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Bovine spongiform encephalopathy is a neurodegenerative disease belonging to the transmissible spongiform encephalopathies, a group of diseases including sheep scrapie and human Creutzfeldt-Jakob disease. Here is the latest research.
Bovine Spongiform Encephalopathy

Bovine spongiform encephalopathy is a neurodegenerative disease belonging to the transmissible spongiform encephalopathies, a group of diseases including sheep scrapie and human Creutzfeldt-Jakob disease. Here is the latest research.

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Here is the latest research on intrinsic and extrinsic factors, as well as pathways and mechanisms that underlie aging in the central nervous system.
Brain Aging
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Here is the latest research on intrinsic and extrinsic factors, as well as pathways and mechanisms that underlie aging in the central nervous system.

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Canavan disease, a type of leukodystrophy, is an autosomal recessive neurodegenerative disorder, and is one of the most common degenerative cerebral diseases of infancy. Discover the latest research on Canavan disease here.
Canavan Disease

Canavan disease, a type of leukodystrophy, is an autosomal recessive neurodegenerative disorder, and is one of the most common degenerative cerebral diseases of infancy. Discover the latest research on Canavan disease here.

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This feed focuses on cellular aging with emphasis on mitochondria, autophagy, and metabolic processes associated with aging and longevity. Here is the latest research on cell aging.
Cell Aging
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This feed focuses on cellular aging with emphasis on mitochondria, autophagy, and metabolic processes associated with aging and longevity. Here is the latest research on cell aging.

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Central Pontine Myelinolysis is a neurologic disorder caused most frequently by rapid correction of hyponatremia and is characterized by demyelination that affects the central portion of the base of the pons. Here is the latest research on this disease.
Central Pontine Myelinolysis
Going Viral

Central Pontine Myelinolysis is a neurologic disorder caused most frequently by rapid correction of hyponatremia and is characterized by demyelination that affects the central portion of the base of the pons. Here is the latest research on this disease.

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Cerebral organoids are an emerging cutting-edge technology to model human brain development and neurodevelopmental disorders. Here is the latest research.
Cerebral Organoids
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Cerebral organoids are an emerging cutting-edge technology to model human brain development and neurodevelopmental disorders. Here is the latest research.

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Apolipoprotein E (APOE) is a protein involved in fat metabolism and associated with the pathogenesis of neurodegenerative and cardiovascular diseases. This feed focuses on the role of this protein in cerebrovasculature and cerebrovascular dysfunction.
Cerebrovasculature & ApoE
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Apolipoprotein E (APOE) is a protein involved in fat metabolism and associated with the pathogenesis of neurodegenerative and cardiovascular diseases. This feed focuses on the role of this protein in cerebrovasculature and cerebrovascular dysfunction.

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Charcot-Marie-Tooth (CMT) disease is a hereditary motor and sensory neuropathy characterized by loss of muscle and touch sensation. Discover the latest research on CMT here.
Charcot-Marie-Tooth Disease
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Charcot-Marie-Tooth (CMT) disease is a hereditary motor and sensory neuropathy characterized by loss of muscle and touch sensation. Discover the latest research on CMT here.

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When demyelination occurs an inflammatory response is activated. Accumulation of excess myelin debris triggers cholesterol crystal formation, and cholesterol deposits in the CNS can overwhelm phagocytes, resulting in impaired tissue regeneration. Here is the latest research on cholesterol synthesis in inflammation and remyelination.
Cholesterol in Inflammation & Remyelination
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When demyelination occurs an inflammatory response is activated. Accumulation of excess myelin debris triggers cholesterol crystal formation, and cholesterol deposits in the CNS can overwhelm phagocytes, resulting in impaired tissue regeneration. Here is the latest research on cholesterol synthesis in inflammation and remyelination.

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Chronic Traumatic Encephalopathy (CTE) is a progressive degenerative disease that occurs in individuals that suffer repetitive brain trauma. Discover the latest research on traumatic encephalopathy here.
Chronic Traumatic Encephalopathy

Chronic Traumatic Encephalopathy (CTE) is a progressive degenerative disease that occurs in individuals that suffer repetitive brain trauma. Discover the latest research on traumatic encephalopathy here.

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Cockayne syndrome is a rare multisystem autosomal recessive disorder characterized by severe photosensitive genodermatosis that is associated with premature aging caused by defects in the UV-induced DNA damage repair system. Follow the latest research with this feed.
Cockayne Syndrome
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Cockayne syndrome is a rare multisystem autosomal recessive disorder characterized by severe photosensitive genodermatosis that is associated with premature aging caused by defects in the UV-induced DNA damage repair system. Follow the latest research with this feed.

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This feed focuses on the role of environmental and genetic factors including environmental policies, disease susceptibility, and socioeconomic status on cognitive decline. Here is the latest research.
Cognitive Decline: Environmental & Genetic Factors
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This feed focuses on the role of environmental and genetic factors including environmental policies, disease susceptibility, and socioeconomic status on cognitive decline. Here is the latest research.

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Computational methods employing machine learning algorithms are powerful tools that can be used to predict the effect of mutations on protein structure. This is important in neurodegenerative disorders, where some mutations can cause the formation of toxic protein aggregations. This feed follows the latests insights into the relationships between mutation and protein structure leading to better understanding of disease.
Computational Methods for Protein Structures

Computational methods employing machine learning algorithms are powerful tools that can be used to predict the effect of mutations on protein structure. This is important in neurodegenerative disorders, where some mutations can cause the formation of toxic protein aggregations. This feed follows the latests insights into the relationships between mutation and protein structure leading to better understanding of disease.

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Corpus Callosum Agenesis Neuronopathy is a neurodegenerative disorder characterized by severe progressive sensorimotor neuropathy beginning in infancy. Here is the latest research.
Corpus Callosum Agenesis Neuronopathy

Corpus Callosum Agenesis Neuronopathy is a neurodegenerative disorder characterized by severe progressive sensorimotor neuropathy beginning in infancy. Here is the latest research.

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Several mechanisms and neural circuits in the cortex enable organisms to move from one place to another. Discover the latest research on cortical control of locomotion here.
Cortical Control of Locomotion

Several mechanisms and neural circuits in the cortex enable organisms to move from one place to another. Discover the latest research on cortical control of locomotion here.

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Corticobasal Degeneration (CBD) is a rare neurodegenerative disease of the cerebral cortex and the basal ganglia, and is characterized by movement disorders. Discover the latest research on CBD here.
Corticobasal Degeneration
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Corticobasal Degeneration (CBD) is a rare neurodegenerative disease of the cerebral cortex and the basal ganglia, and is characterized by movement disorders. Discover the latest research on CBD here.

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Creutzfeldt-Jakob Disease is a fatal neurodegenerative disease caused by aggregation and accumulation of misfolded prion proteins. Here are the latest discoveries pertaining to this disease.
Creutzfeldt-Jakob Disease

Creutzfeldt-Jakob Disease is a fatal neurodegenerative disease caused by aggregation and accumulation of misfolded prion proteins. Here are the latest discoveries pertaining to this disease.

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Demyelinating diseases result in damage to the myelin sheath that surrounds nerves in the brain and spinal cord and can result in neurological conditions such as multiple sclerosis. Here is the latest research on demyelinating disorders of the central nervous system.
Demyelinating Disorders of the CNS
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Demyelinating diseases result in damage to the myelin sheath that surrounds nerves in the brain and spinal cord and can result in neurological conditions such as multiple sclerosis. Here is the latest research on demyelinating disorders of the central nervous system.

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Epidemiological data indicate that a significant portion of the diabetic population exhibits brain changes that are hallmarks of Alzheimer’s disease and vascular dementia. However, direct biological links remain incompletely understood. Here is the latest research in this area.
Diabetes & Alzheimer's Disease
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Epidemiological data indicate that a significant portion of the diabetic population exhibits brain changes that are hallmarks of Alzheimer’s disease and vascular dementia. However, direct biological links remain incompletely understood. Here is the latest research in this area.

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A healthy and balanced diet is essential for controling diseases such as multiple sclerosis. Here are the latest discoveries pertaining to diet and this disease.
Diet & Multiple Sclerosis
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A healthy and balanced diet is essential for controling diseases such as multiple sclerosis. Here are the latest discoveries pertaining to diet and this disease.

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Diffuse myelinoclastic sclerosis is a rare neurodegenerative disorder characterized by pseudotumoral demyelinating lesions. It is sometimes considered a variant of multiple sclerosis while others classify it a distinct disease. Find the latest research on diffuse myelinoclastic sclerosis here.
Diffuse Myelinoclastic Sclerosis
Fast Growing

Diffuse myelinoclastic sclerosis is a rare neurodegenerative disorder characterized by pseudotumoral demyelinating lesions. It is sometimes considered a variant of multiple sclerosis while others classify it a distinct disease. Find the latest research on diffuse myelinoclastic sclerosis here.

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Diffuse Neurofibrillary Tangles with Calcification (DNTC) is a rare form of presenile dementia. Here is the latest research.
Diffuse Neurofibrillary Tangles with Calcification

Diffuse Neurofibrillary Tangles with Calcification (DNTC) is a rare form of presenile dementia. Here is the latest research.

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Embryonic neural stem cells can be used to investigate the effect of different toxins and molecules on the proliferation and differentiation of these cells. Discover the latest research of embryonic neural stem cells here.
Embryonic Neural Stem Cells
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Embryonic neural stem cells can be used to investigate the effect of different toxins and molecules on the proliferation and differentiation of these cells. Discover the latest research of embryonic neural stem cells here.

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Environmental factors play a role in neurological diseases and in particular their contribution in astrocyte proinflammation is of interest. The activation of astrocytes may be involved in neuroinflammation and implicated in neurodegenerative diseases. Here is the latest research on environment and astrocyte proinflammation.
Environment & Astrocyte Proinflammation

Environmental factors play a role in neurological diseases and in particular their contribution in astrocyte proinflammation is of interest. The activation of astrocytes may be involved in neuroinflammation and implicated in neurodegenerative diseases. Here is the latest research on environment and astrocyte proinflammation.

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Microglial cells are instrumental in the maintenance of homeostasis in the CNS and can contribute to neurodegenerative disorders. Epigenetic mechanisms and transcriptional regulation may be potential therapeutic targets for their activation or suppression. Discover the latest research on epigenetic regulation of microglial functions here.
Epigenetic Regulation of Microglia Functions
Going Viral

Microglial cells are instrumental in the maintenance of homeostasis in the CNS and can contribute to neurodegenerative disorders. Epigenetic mechanisms and transcriptional regulation may be potential therapeutic targets for their activation or suppression. Discover the latest research on epigenetic regulation of microglial functions here.

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Myotonic Dystrophy is an autosomal dominant neuromuscular disorder associated with the expansion of CTG trinucleotide repeats in the 3’ untranslated region of the myotonic dystrophy protein kinase (DMPK) gene. Here is the latest research focusing on epigenetic modifications in myotonic dystrophy.
Epigenetics of Myotonic Dystrophy

Myotonic Dystrophy is an autosomal dominant neuromuscular disorder associated with the expansion of CTG trinucleotide repeats in the 3’ untranslated region of the myotonic dystrophy protein kinase (DMPK) gene. Here is the latest research focusing on epigenetic modifications in myotonic dystrophy.

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Extracellular vesicles found in human biological fluids may provide insight into the protein profiles of human neurons in various diseases, including Parkinson's disease. Here is the latest research on extracellular vesicles and Parkinson's disease.
Extracellular Vesicles & Parkinson Disease

Extracellular vesicles found in human biological fluids may provide insight into the protein profiles of human neurons in various diseases, including Parkinson's disease. Here is the latest research on extracellular vesicles and Parkinson's disease.

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Familial Amyloid Neuropathies (FAP) are a rare group of autosomal dominant diseases in which nerves are compromised due to protein aggregation or fibril formation of transthyretin, apolipoprotein A1, or gelsolin proteins, resulting in nervous system dysfunction. Here is the latest research on these diseases.
Familial Amyloid Polyneuropathy
Influential

Familial Amyloid Neuropathies (FAP) are a rare group of autosomal dominant diseases in which nerves are compromised due to protein aggregation or fibril formation of transthyretin, apolipoprotein A1, or gelsolin proteins, resulting in nervous system dysfunction. Here is the latest research on these diseases.

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Fatal familial insomnia is a familial prion disease linked to a mutation of the prion protein gene. Here is the latest research on this rare genetic degenerative brain disorder.
Fatal Familial Insomnia

Fatal familial insomnia is a familial prion disease linked to a mutation of the prion protein gene. Here is the latest research on this rare genetic degenerative brain disorder.

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Fragile X syndrome is caused by an expanded CGG repeat (> 200 repeats) in the 5' untranslated portion of the fragile mental retardation 1 gene (FMR1) within the X chromosome, leading to deficiency of fragile X mental retardation protein (FMRP). It is characterized by cognitive impairment, hyperactivity, seizures, language delay, and enlargement of the ears, head, and testes. Discover the latest research on Fragile X syndrome here.
Fragile X Syndrome
Fast Growing

Fragile X syndrome is caused by an expanded CGG repeat (> 200 repeats) in the 5' untranslated portion of the fragile mental retardation 1 gene (FMR1) within the X chromosome, leading to deficiency of fragile X mental retardation protein (FMRP). It is characterized by cognitive impairment, hyperactivity, seizures, language delay, and enlargement of the ears, head, and testes. Discover the latest research on Fragile X syndrome here.

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Fragile X-associated tremor/ataxia syndrome (FXTAS) is caused by the expansion of CGG triple repeat in the FMR1 gene and characterized by movement problems and cognitive impairment. Here is the latest research on FXTAS.
Fragile X-associated Tremor/Ataxia Syndrome

Fragile X-associated tremor/ataxia syndrome (FXTAS) is caused by the expansion of CGG triple repeat in the FMR1 gene and characterized by movement problems and cognitive impairment. Here is the latest research on FXTAS.

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Friedreich's ataxia is a rare autosomal recessive disease caused by defects in the FXN gene and characterized by progressive damage of the nervous system and movement problems. Here is the latest research on Friedreich's ataxia.
Friedreich's Ataxia
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Friedreich's ataxia is a rare autosomal recessive disease caused by defects in the FXN gene and characterized by progressive damage of the nervous system and movement problems. Here is the latest research on Friedreich's ataxia.

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Frontotemporal dementia (FTD) refers to disorders caused by progressive neuronal loss in the frontal and temporal lobes of the brain. Here is the latest research on FTD and FTD-associated disorders.
Frontotemporal Dementia

Frontotemporal dementia (FTD) refers to disorders caused by progressive neuronal loss in the frontal and temporal lobes of the brain. Here is the latest research on FTD and FTD-associated disorders.

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Genetic screening is a critical tool that can be employed to define and understand gene function and interaction. This feed focuses on genetic screens conducted using induced pluripotent stem cell (iPSC)-derived brain cells. It also follows CRISPR-Cas9 approaches to generating genetic mutants as a means of understanding the effect of genetics on phenotype.
Genetic Screens in iPSC-derived Brain Cells
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Genetic screening is a critical tool that can be employed to define and understand gene function and interaction. This feed focuses on genetic screens conducted using induced pluripotent stem cell (iPSC)-derived brain cells. It also follows CRISPR-Cas9 approaches to generating genetic mutants as a means of understanding the effect of genetics on phenotype.

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Dozens of genes are implicated in lifespan, and epigenetic changes during aging affect cell function. This feed focuses on the genetics and epigenetics of aging.
Genetics & Epigenetics of Aging
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Dozens of genes are implicated in lifespan, and epigenetic changes during aging affect cell function. This feed focuses on the genetics and epigenetics of aging.

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Genome surgery using CRISPR genomic techniques offer the potential to correct disease-causing mutations in many neuronal disorders. Here are the latest discoveries pertaining to genome surgery and neuronal disorders.
Genome Surgery & Neuronal Disorders

Genome surgery using CRISPR genomic techniques offer the potential to correct disease-causing mutations in many neuronal disorders. Here are the latest discoveries pertaining to genome surgery and neuronal disorders.

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Gerstmann-Straussler-Scheinker disease is a rare neurodegenerative disorder caused by prions and characterized by symptoms similar to other prion-associated diseases. Here is the latest research.
Gerstmann-Straussler-Scheinker Disease

Gerstmann-Straussler-Scheinker disease is a rare neurodegenerative disorder caused by prions and characterized by symptoms similar to other prion-associated diseases. Here is the latest research.

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The glymphatic system is a CSF-mediated waste clearance pathway that is formed by astroglial cells. The removal of potentially harmful substances such as amyloid protein is important to prevent toxic levels accumulate in the brain. Discover the latest research on glymphatics here.
Glymphatics
Influential

The glymphatic system is a CSF-mediated waste clearance pathway that is formed by astroglial cells. The removal of potentially harmful substances such as amyloid protein is important to prevent toxic levels accumulate in the brain. Discover the latest research on glymphatics here.

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Patients with neurodegenerative diseases often have an altered composition of the gut microbiome. Novel reports suggest that there is an extensive crosstalk between the gut microbiome and the brain, and this may influence neurodegeneration. Discover the latest research on how the gut microbiome influences neurodegeneration here.
Gut Microbiome & Neurodegeneration
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Patients with neurodegenerative diseases often have an altered composition of the gut microbiome. Novel reports suggest that there is an extensive crosstalk between the gut microbiome and the brain, and this may influence neurodegeneration. Discover the latest research on how the gut microbiome influences neurodegeneration here.

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The gut microbiota has been implicated in neurodevelopment, neuroinflammatory responses, and neurological disorders. Here is the latest research on the role of the gut microbiota in movement disorders.
Gut Microbiota in Movement Disorders
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The gut microbiota has been implicated in neurodevelopment, neuroinflammatory responses, and neurological disorders. Here is the latest research on the role of the gut microbiota in movement disorders.

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Hallervorden-Spatz disease, also known as pantothenate kinase-associated neurodegeneration, is a rare neurodegenerative disease associated with iron accumulation in the brain. Here is the latest research.
Hallervorden-Spatz Disease
Going Viral

Hallervorden-Spatz disease, also known as pantothenate kinase-associated neurodegeneration, is a rare neurodegenerative disease associated with iron accumulation in the brain. Here is the latest research.

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The formation of blood cells, hematopoiesis, has been exploited for the potential use as a therapy in Alzheimer’s disease. Decreased levels of lymphocytes and proteins involved in stem cell recruitment are found in early stages of Alzheimer’s disease. Here is the latest research on hematopoiesis and Alzheimer’s disease.
Hematopoiesis & Alzheimer's Disease

The formation of blood cells, hematopoiesis, has been exploited for the potential use as a therapy in Alzheimer’s disease. Decreased levels of lymphocytes and proteins involved in stem cell recruitment are found in early stages of Alzheimer’s disease. Here is the latest research on hematopoiesis and Alzheimer’s disease.

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Hereditary demyelinating diseases of the central nervous systems are caused primarily due to inherited genetic conditions. Examples include Alexander, Canavan, and Pelizaeus-Merzbacher diseases. Here is the latest research.
Hereditary Demyelinating Diseases of the CNS
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Hereditary demyelinating diseases of the central nervous systems are caused primarily due to inherited genetic conditions. Examples include Alexander, Canavan, and Pelizaeus-Merzbacher diseases. Here is the latest research.

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Hereditary optic atrophy is a mitochondrially inherited disease that results in bilateral subacute loss of central vision due to degeneration of retinal ganglion cells and the optic nerve. Here is the latest research.
Hereditary Optic Atrophy
Going Viral

Hereditary optic atrophy is a mitochondrially inherited disease that results in bilateral subacute loss of central vision due to degeneration of retinal ganglion cells and the optic nerve. Here is the latest research.

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Hereditary Sensory Autonomic Neuropathies are a group of inherited neurodegenerative disorders characterized clinically by loss of sensation and autonomic dysfunction. Here is the latest research on these neuropathies.
Hereditary Sensory Autonomic Neuropathy

Hereditary Sensory Autonomic Neuropathies are a group of inherited neurodegenerative disorders characterized clinically by loss of sensation and autonomic dysfunction. Here is the latest research on these neuropathies.

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Hereditary spastic paraplegia is a neurodegenerative disorder characterized by spastic paraplegia, cognitive impairment, peripheral neuropathy, and progressive spasticity of lower limbs. Here is a latest research.
Hereditary Spastic Paraplegia

Hereditary spastic paraplegia is a neurodegenerative disorder characterized by spastic paraplegia, cognitive impairment, peripheral neuropathy, and progressive spasticity of lower limbs. Here is a latest research.

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Huntington's disease is a hereditary neurodegenerative disease characterized by poor coordination and involuntary body movements. Discover the latest research on Huntington's disease here.
Huntington's Disease

Huntington's disease is a hereditary neurodegenerative disease characterized by poor coordination and involuntary body movements. Discover the latest research on Huntington's disease here.

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Huntington’s disease (HD) is a hereditary neurodegenerative disease caused by mutations in the HTT gene, which encodes the huntingtin protein. HD is characterized by poor coordination and involuntary body movements. Here is the latest research on Huntington's disease genetics.
Huntington’s Disease: Genetics

Huntington’s disease (HD) is a hereditary neurodegenerative disease caused by mutations in the HTT gene, which encodes the huntingtin protein. HD is characterized by poor coordination and involuntary body movements. Here is the latest research on Huntington's disease genetics.

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Huntington’s disease (HD) is a hereditary neurodegenerative disease caused by mutations in the HTT gene which encodes the huntingtin protein. HD is characterized by poor coordination and involuntary body movements. Here is the latest research on the structure and function of huntingtin.
Huntington’s Disease: HTT Structure & Function

Huntington’s disease (HD) is a hereditary neurodegenerative disease caused by mutations in the HTT gene which encodes the huntingtin protein. HD is characterized by poor coordination and involuntary body movements. Here is the latest research on the structure and function of huntingtin.

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Huntington’s disease is a hereditary neurodegenerative disease characterized by poor coordination and involuntary body movements. Here is the latest research on Huntington’s disease therapeutics.
Huntington’s Disease: Therapeutics

Huntington’s disease is a hereditary neurodegenerative disease characterized by poor coordination and involuntary body movements. Here is the latest research on Huntington’s disease therapeutics.

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Multiple sclerosis (MS) was one of the first conditions shown to be associated with HLA class II factors. Find the latest research on MS here, including HLA studies providing insights on the genetics of the susceptibility to MS.
Immunogenetics of Multiple Sclerosis

Multiple sclerosis (MS) was one of the first conditions shown to be associated with HLA class II factors. Find the latest research on MS here, including HLA studies providing insights on the genetics of the susceptibility to MS.

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The end result of the inflammatory process within the brain is neurotoxicity and/or neuroprotection. It is a function of the fine balance between the two cellular systems, i.e., of the complex signaling relationships between anti-inflammatory neuroprotective factors (neurotrophins and other chemical mediators) and proinflammatory neurotoxic factors (tnf, free radicals, certain cytokines, etc). Here are the latest discoveries pertaining to neurotoxicity, inflammation and neuroprotection.
Inflammation in the brain results in neurotoxicity and/or neuroprotection. It is a function of the fine balance between the two cellular systems, i.e., of the complex signaling relationships between anti-inflammatory neuroprotective factors and proinflammatory neurotoxic factors. Here are the latest insights into neurotoxicity, inflammation and neuroprotection.
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The end result of the inflammatory process within the brain is neurotoxicity and/or neuroprotection. It is a function of the fine balance between the two cellular systems, i.e., of the complex signaling relationships between anti-inflammatory neuroprotective factors (neurotrophins and other chemical mediators) and proinflammatory neurotoxic factors (tnf, free radicals, certain cytokines, etc). Here are the latest discoveries pertaining to neurotoxicity, inflammation and neuroprotection.

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The innate immune response is an important regulator of the downstream immune response, and it is becoming increasingly evident that immune dysfunction has impacts on the pathogenesis of multiple disease states, including neurodegeneration. Additionally, mutations in several genes involved in the innate immune response have been associated with neurodegeneration. Discover the latest research on innate immunity and neurodegeneration here.
Innate Immunity & Neurodegeneration

The innate immune response is an important regulator of the downstream immune response, and it is becoming increasingly evident that immune dysfunction has impacts on the pathogenesis of multiple disease states, including neurodegeneration. Additionally, mutations in several genes involved in the innate immune response have been associated with neurodegeneration. Discover the latest research on innate immunity and neurodegeneration here.

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Interactomics is the intersection of bioinformatics and biology concerned with investigating the interaction and the consequences of these interactions between proteins in a cell. Here is the latest research on interactomic data in neurodegeneration.
Interactomics in Neurodegeneration

Interactomics is the intersection of bioinformatics and biology concerned with investigating the interaction and the consequences of these interactions between proteins in a cell. Here is the latest research on interactomic data in neurodegeneration.

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Interneurons are found throughout the central nervous system and create important networks of connections between other sensory and motor neurons. This feed covers the use of stem cells to artificially create interneurons and insights gained into the role of interneurons throughout the CNS.
Interneurons & Stem Cells
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Interneurons are found throughout the central nervous system and create important networks of connections between other sensory and motor neurons. This feed covers the use of stem cells to artificially create interneurons and insights gained into the role of interneurons throughout the CNS.

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Kennedy Disease, also known as X-linked bulbospinal muscular atrophy, is a progressive neuromuscular disorder caused by mutations in the androgen receptor gene and characterized by wasting of proximal and bulbar muscles. Follow the research into Kennedy Disease with this feed.
Kennedy Disease

Kennedy Disease, also known as X-linked bulbospinal muscular atrophy, is a progressive neuromuscular disorder caused by mutations in the androgen receptor gene and characterized by wasting of proximal and bulbar muscles. Follow the research into Kennedy Disease with this feed.

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LRRK2 has been shown to function as a central regulator of vesicular trafficking, infection, immunity, and inflammation. Mutations in the LRRK2 gene are a risk-factor for developing Parkinson’s disease. Here is the latest research on the role of this kinase on immunity during infection.
LRRK2 & Immunity during Infection

LRRK2 has been shown to function as a central regulator of vesicular trafficking, infection, immunity, and inflammation. Mutations in the LRRK2 gene are a risk-factor for developing Parkinson’s disease. Here is the latest research on the role of this kinase on immunity during infection.

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Mutations in the LRRK2 gene are risk-factors for developing Parkinson’s disease (PD). LRRK2 mutations in PD have been shown to enhance its association with microtubules. Here is the latest research.
LRRK2 & Microtubules

Mutations in the LRRK2 gene are risk-factors for developing Parkinson’s disease (PD). LRRK2 mutations in PD have been shown to enhance its association with microtubules. Here is the latest research.

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Mutations in the LRRK2 gene are risk-factors for developing Parkinson’s disease. Increased kinase activity of certain LRRK2 mutants is thought to underlie dopaminergic neuron damage. Thus, LRRK2 inhibitors offer the potential to target an underlying cause of the disease. Here is the latest research.
LRRK2 Inhibitors

Mutations in the LRRK2 gene are risk-factors for developing Parkinson’s disease. Increased kinase activity of certain LRRK2 mutants is thought to underlie dopaminergic neuron damage. Thus, LRRK2 inhibitors offer the potential to target an underlying cause of the disease. Here is the latest research.

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Lafora disease is an inherited neurodegenerative disorder characterized by the presence of abnormal glycogen inclusions, called Lafora bodies, in neurons and other tissues. Here is the latest research on this fatal autosomal recessive disorder.
Lafora Disease
Going Viral

Lafora disease is an inherited neurodegenerative disorder characterized by the presence of abnormal glycogen inclusions, called Lafora bodies, in neurons and other tissues. Here is the latest research on this fatal autosomal recessive disorder.

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Leigh syndrome, caused by a dysfunction in mitochondrial energy metabolism, is an inherited, heterogeneous, and progressive neurodegenerative disorder of infancy and childhood. Discover the latest research on Leigh syndrome here.
Leigh Syndrome
Fast Growing

Leigh syndrome, caused by a dysfunction in mitochondrial energy metabolism, is an inherited, heterogeneous, and progressive neurodegenerative disorder of infancy and childhood. Discover the latest research on Leigh syndrome here.

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Lewy Body Disease is associated with abnormal deposits of proteins like alpha-synuclein in the brain and causes a decline in mental abilities. Here is the latest research on this progressive neurological disorder.
Lewy Body Disease
Influential

Lewy Body Disease is associated with abnormal deposits of proteins like alpha-synuclein in the brain and causes a decline in mental abilities. Here is the latest research on this progressive neurological disorder.

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Lichtheim's disease or subacute combined degeneration of spinal cord is a degenerative disease associated with deficiencies in vitamin B12, vitamin E, and copper. Here is the latest research on this disease.
Lichtheim's Disease

Lichtheim's disease or subacute combined degeneration of spinal cord is a degenerative disease associated with deficiencies in vitamin B12, vitamin E, and copper. Here is the latest research on this disease.

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The mechanisms of axonal degeneration are still poorly understood, but studies in animal models have pointed to specific cellular and molecular mechanisms that may be responsible. The degeneration of axons in the CNS has been found to be characteristic of many neurodegenerative diseases. Discover the latest research on the mechanisms of axon degeneration here.
Mechanisms of Axon Degeneration
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The mechanisms of axonal degeneration are still poorly understood, but studies in animal models have pointed to specific cellular and molecular mechanisms that may be responsible. The degeneration of axons in the CNS has been found to be characteristic of many neurodegenerative diseases. Discover the latest research on the mechanisms of axon degeneration here.

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Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Some key proteins have been identified, but the mechanisms that underlie prion propagation in the brain are still not fully understood. This feed follows the latest research into mechanisms of prion propagation.
Mechanisms of Prion Propagation

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Some key proteins have been identified, but the mechanisms that underlie prion propagation in the brain are still not fully understood. This feed follows the latest research into mechanisms of prion propagation.

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Mesenchymal stem cells are multipotent cells that are a readily available source of stem cells from bone marrow, adipose tissue, and umbilical cord blood. They have shown promise for therapeutic use in neurological diseases. Discover the latest research on mesenchymal stem cells and neurological diseases here.
Mesenchymal Stem Cell & Neurological Diseases
Influential

Mesenchymal stem cells are multipotent cells that are a readily available source of stem cells from bone marrow, adipose tissue, and umbilical cord blood. They have shown promise for therapeutic use in neurological diseases. Discover the latest research on mesenchymal stem cells and neurological diseases here.

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This feed focuses on the impact of insulin resistance, caloric restriction, cytokine singling, and metabolism on dementia and neurodegenerative diseases.
Metabolism & Insulin in Neurodegeneration

This feed focuses on the impact of insulin resistance, caloric restriction, cytokine singling, and metabolism on dementia and neurodegenerative diseases.

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Microglia are resident macrophages of the CNS. They play a role in scavenging the CNS for plaques, phagocytosis, and extracellular signaling. Here is the latest research on their role in CNS diseases.
Microglia & CNS Diseases

Microglia are resident macrophages of the CNS. They play a role in scavenging the CNS for plaques, phagocytosis, and extracellular signaling. Here is the latest research on their role in CNS diseases.

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Misfolding and aggregation of proteins can lead to several diseases. For instance, misfolding of prion or tau proteins are associated with several neurodegenerative diseases. Here is the latest research on diseases caused by protein misfolding and aggregation.
Misfolding & Aggregation Diseases

Misfolding and aggregation of proteins can lead to several diseases. For instance, misfolding of prion or tau proteins are associated with several neurodegenerative diseases. Here is the latest research on diseases caused by protein misfolding and aggregation.

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Mitochondrial quality control mechanisms operate at the protein, organelle, and cell level and are key to organismal health. Perturbance of these pathways can lead to a variety of pathologies, including Parkinson's disease. This feed follows the latest research in this area.
Mitochondrial Quality Control in Neurodegeneration
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Mitochondrial quality control mechanisms operate at the protein, organelle, and cell level and are key to organismal health. Perturbance of these pathways can lead to a variety of pathologies, including Parkinson's disease. This feed follows the latest research in this area.

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Mutations in RNA-binding proteins, including TARDBP, have been found in neurodegenerative diseases such as amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). This feed focuses on molecular mechanisms that underlie ALS and FTD.
Molecular Mechanisms of Disease in ALS & FTD

Mutations in RNA-binding proteins, including TARDBP, have been found in neurodegenerative diseases such as amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). This feed focuses on molecular mechanisms that underlie ALS and FTD.

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The motor cortex is associated with planning, control, and execution of voluntary movements. This feed focuses on bi-directional communication pathways between an external device and the motor cortex.
Motor Cortex & Brain-Machine Interfaces

The motor cortex is associated with planning, control, and execution of voluntary movements. This feed focuses on bi-directional communication pathways between an external device and the motor cortex.

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Motor neuron diseases such as amyotrophic lateral sclerosis and spinal muscular atrophy are progressive neurodegenerative diseases that result in the death of motor neurons. Discover the latest research on motor neuron disease here.
Motor Neuron Disease
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Motor neuron diseases such as amyotrophic lateral sclerosis and spinal muscular atrophy are progressive neurodegenerative diseases that result in the death of motor neurons. Discover the latest research on motor neuron disease here.

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Multiple Sclerosis is a demyelinating autoimmune disease of the central nervous system. Symptoms include optic neuritis and motor weakness. Discover the latest research on Multiple Sclerosis with this feed.
Multiple Sclerosis

Multiple Sclerosis is a demyelinating autoimmune disease of the central nervous system. Symptoms include optic neuritis and motor weakness. Discover the latest research on Multiple Sclerosis with this feed.

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Multiple Sclerosis (MS) is the most common chronic inflammatory disease of the central nervous system which is characterized by inflammatory demyelination and neurodegeneration. Here is the latest research MS and aging.
Multiple Sclerosis: Aging
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Multiple Sclerosis (MS) is the most common chronic inflammatory disease of the central nervous system which is characterized by inflammatory demyelination and neurodegeneration. Here is the latest research MS and aging.

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Magnetic resonance imaging (MRI) has been used to diagnose and monitor disease progression in multiple sclerosis (MS). MRI has improved the management of MS patients and provided information on the level of atrophy in the brain and the severity of the disease. Discover the latest research on multiple sclerosis imaging here.
Multiple Sclerosis: Imaging

Magnetic resonance imaging (MRI) has been used to diagnose and monitor disease progression in multiple sclerosis (MS). MRI has improved the management of MS patients and provided information on the level of atrophy in the brain and the severity of the disease. Discover the latest research on multiple sclerosis imaging here.

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Multiple sclerosis (MS) is the most common chronic inflammatory disease of the central nervous system; it is characterized by inflammatory demyelination and neurodegeneration. Here is the latest research into the influence of the human microbiome on this demyelinating disorder.
Multiple Sclerosis: Microbiome
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Multiple sclerosis (MS) is the most common chronic inflammatory disease of the central nervous system; it is characterized by inflammatory demyelination and neurodegeneration. Here is the latest research into the influence of the human microbiome on this demyelinating disorder.

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Multiple sclerosis (MS) is the most common chronic inflammatory disease of the central nervous system which is characterized by inflammatory demyelination and neurodegeneration. Here is the latest research on neuroprotective mechanisms in MS.
Multiple Sclerosis: Neuroprotection

Multiple sclerosis (MS) is the most common chronic inflammatory disease of the central nervous system which is characterized by inflammatory demyelination and neurodegeneration. Here is the latest research on neuroprotective mechanisms in MS.

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Multiple Sclerosis (MS) is a demyelinating autoimmune disease of the central nervous system. Symptoms include optic neuritis and motor weakness. Here is the latest research on the pathology of this complex neurodegenerative disease.
Multiple Sclerosis: Pathology

Multiple Sclerosis (MS) is a demyelinating autoimmune disease of the central nervous system. Symptoms include optic neuritis and motor weakness. Here is the latest research on the pathology of this complex neurodegenerative disease.

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Multiple Sclerosis is a demyelinating autoimmune disease of the central nervous system, resulting in symptoms including optic neuritis and motor weakness. The feed focuses on therapeutic interventions used to treat Multiple Sclerosis.
Multiple Sclerosis: Therapies

Multiple Sclerosis is a demyelinating autoimmune disease of the central nervous system, resulting in symptoms including optic neuritis and motor weakness. The feed focuses on therapeutic interventions used to treat Multiple Sclerosis.

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Multiple System Atrophy is a rare neurodegenerative disorder which affects the body’s autonomic functions, including breathing and muscle control. Discover the latest research on Multiple System Atrophy here.
Multiple System Atrophy

Multiple System Atrophy is a rare neurodegenerative disorder which affects the body’s autonomic functions, including breathing and muscle control. Discover the latest research on Multiple System Atrophy here.

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Myelin surrounds axons and is important for efficient axonal function. Formation of myelin is regulated by many signaling pathways. Degeneration can result in multiple diseases such as multiple sclerosis, and regeneration is associated with oligodendrocyte progenitor cells. Here is the latest research on myelin formation and repair.
Myelin Formation & Repair

Myelin surrounds axons and is important for efficient axonal function. Formation of myelin is regulated by many signaling pathways. Degeneration can result in multiple diseases such as multiple sclerosis, and regeneration is associated with oligodendrocyte progenitor cells. Here is the latest research on myelin formation and repair.

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Myeloid cells in the CNS are innate immune cells that are essential for brain development and maintenance. Here is the latest research on myeloid cells, including microglia, in neuroinflammation.
Myeloid Cells & Neuroinflammation

Myeloid cells in the CNS are innate immune cells that are essential for brain development and maintenance. Here is the latest research on myeloid cells, including microglia, in neuroinflammation.

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Myopathies are genetic or acquired disorders of skeletal muscle that lead to varying degrees of weakness, atrophy, and exercise intolerance. Here are the latest discoveries pertaining to this disease.
Myopathies
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Myopathies are genetic or acquired disorders of skeletal muscle that lead to varying degrees of weakness, atrophy, and exercise intolerance. Here are the latest discoveries pertaining to this disease.

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Myotonia congenita is a disorder that causes sustained tensing of skeletal muscles, preventing them from relaxing. This feed focuses on mechanisms that underlie this condition and therapeutic strategies used for its treatment.
Myotonia Congenita

Myotonia congenita is a disorder that causes sustained tensing of skeletal muscles, preventing them from relaxing. This feed focuses on mechanisms that underlie this condition and therapeutic strategies used for its treatment.

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Myotonic Dystrophy is an autosomal dominant neuromuscular disorder associated with the expansion of CTG trinucleotide repeats in the 3’ untranslated region of the myotonic dystrophy protein kinase (DMPK) gene. Here is the latest research.
Myotonic Dystrophy

Myotonic Dystrophy is an autosomal dominant neuromuscular disorder associated with the expansion of CTG trinucleotide repeats in the 3’ untranslated region of the myotonic dystrophy protein kinase (DMPK) gene. Here is the latest research.

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Neural stem cells are self-renewing, multipotent cells that are present in both the embryonic and adult brain. Several factors, including metabolism, have been shown to regulate proliferation, differentiation, and quiescence of neural stem cells. Here is the latest research.
Neural Stem Cell Regulation
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Neural stem cells are self-renewing, multipotent cells that are present in both the embryonic and adult brain. Several factors, including metabolism, have been shown to regulate proliferation, differentiation, and quiescence of neural stem cells. Here is the latest research.

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Neuroacanthocytosis is a heterogeneous group of hereditary syndromes characterized by the association of neurological abnormalities with acanthocytosis. Discover the latest research on neuroacanthocytosis here.
Neuroacanthocytosis

Neuroacanthocytosis is a heterogeneous group of hereditary syndromes characterized by the association of neurological abnormalities with acanthocytosis. Discover the latest research on neuroacanthocytosis here.

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Aging is the primary risk factor for most neurodegenerative diseases, including Alzheimer's and Parkinson's disease. Biological mechanisms of aging - for example genomic instability - are being explored as therapeutic targets for neurodegenerative disease This feed follows the latest research into neurodegeneration and aging.
Neurodegeneration & Aging
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Aging is the primary risk factor for most neurodegenerative diseases, including Alzheimer's and Parkinson's disease. Biological mechanisms of aging - for example genomic instability - are being explored as therapeutic targets for neurodegenerative disease This feed follows the latest research into neurodegeneration and aging.

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Functional genomics techniques such as CRISPR/RNAi screens, microarrays, etc. can be employed to better understand complex mechanisms that underlie neurodegeneration. Here is the latest research.
Neurodegeneration & Functional Genomics
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Functional genomics techniques such as CRISPR/RNAi screens, microarrays, etc. can be employed to better understand complex mechanisms that underlie neurodegeneration. Here is the latest research.

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Given that progression of neurodegenerative diseases can be driven by aggregation of misfolded proteins, autophagic activity is through to modulate the severity of neurodegenerative diseases. Here is the latest research on the influence of autophagy on neurodegeneration.
Neurodegeneration: Autophagy

Given that progression of neurodegenerative diseases can be driven by aggregation of misfolded proteins, autophagic activity is through to modulate the severity of neurodegenerative diseases. Here is the latest research on the influence of autophagy on neurodegeneration.

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Neurodegeneration is the progressive loss of structure or function of neurons, including neuronal death, and leads to several diseases, including Alzheimer’s and Parkinson’s. This feed focuses on the genetic alterations that underlie neurodegeneration.
Neurodegeneration: Genetics
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Neurodegeneration is the progressive loss of structure or function of neurons, including neuronal death, and leads to several diseases, including Alzheimer’s and Parkinson’s. This feed focuses on the genetic alterations that underlie neurodegeneration.

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microRNAs (miRNAs) are key regulatory RNAs that modulate gene expression in various biological processes and neurodegenerative disorders. Here are the latest discoveries pertaining to miRNAs and neurodegeneration.
Neurodegeneration: MicroRNAs
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microRNAs (miRNAs) are key regulatory RNAs that modulate gene expression in various biological processes and neurodegenerative disorders. Here are the latest discoveries pertaining to miRNAs and neurodegeneration.

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TDP-43 is implicated in several neurodegenerative disorders including Alzheimer's disease, amyotrophic lateral sclerosis and frontotemporal lobe dementia. It has also been shown to regulate microglial phagocytosis. Here is the latest research on the role of TDP-43 and microglia in neurodegeneration.
Neurodegeneration: Microglia & TDP-43

TDP-43 is implicated in several neurodegenerative disorders including Alzheimer's disease, amyotrophic lateral sclerosis and frontotemporal lobe dementia. It has also been shown to regulate microglial phagocytosis. Here is the latest research on the role of TDP-43 and microglia in neurodegeneration.

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Protein misfolding is the primary cause of several neurodegenerative diseases, including Alzheimer’s and Parkinson’s disease. This feed follows studies investigating protein misfolding in neurodegeneration.
Neurodegeneration: Misfolded Proteins
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Protein misfolding is the primary cause of several neurodegenerative diseases, including Alzheimer’s and Parkinson’s disease. This feed follows studies investigating protein misfolding in neurodegeneration.

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Progranulin is a secreted growth factor involved in inflammation, wound healing, cancer, and known for its neurotrophic properties. Along with lipids, these molcules are associated with neurodegenerative processes. Here are the latest discoveries pertaining to these molecules and neurodegeneration.
Neurodegeneration: Progranulin & Lipids
Fast Growing

Progranulin is a secreted growth factor involved in inflammation, wound healing, cancer, and known for its neurotrophic properties. Along with lipids, these molcules are associated with neurodegenerative processes. Here are the latest discoveries pertaining to these molecules and neurodegeneration.

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Neurodegeneration is the progressive loss of structure or function of neurons, including neuronal death, and leads to several diseases, including Alzheimer’s and Parkinson’s. This feed focuses on therapeutic targets for neurodegenerative disorders.
Neurodegeneration: Therapeutic Targets

Neurodegeneration is the progressive loss of structure or function of neurons, including neuronal death, and leads to several diseases, including Alzheimer’s and Parkinson’s. This feed focuses on therapeutic targets for neurodegenerative disorders.

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Neurodegenerative disorders and obesity appear to have common pathologies including neuroinflammation. Targeting common pathways may prove to be a useful treatment strategy for neurodegenerative disease. Discover the latest research on neurodegenerative disorders and obesity here.
Neurodegenerative Disease & Obesity

Neurodegenerative disorders and obesity appear to have common pathologies including neuroinflammation. Targeting common pathways may prove to be a useful treatment strategy for neurodegenerative disease. Discover the latest research on neurodegenerative disorders and obesity here.

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Neurodegeneration is the progressive loss of structure or function of neurons, including neuronal death, and leads to several diseases, including Alzheimer’s and Parkinson’s. This feed focuses on the genetics and pathology of neurodegenerative diseases.
Neurodegenerative Disease: Genetics & Pathology
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Neurodegeneration is the progressive loss of structure or function of neurons, including neuronal death, and leads to several diseases, including Alzheimer’s and Parkinson’s. This feed focuses on the genetics and pathology of neurodegenerative diseases.

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Gene therapy is a powerful tool that can potentially be used for treating neurodegenerative diseases such as amyotrophic lateral sclerosis, spinal muscular atrophy, Parkinson's disease, and Alzheimer's disease. Follow the latest research in this field with this feed.
Neurodegenerative Diseases: Gene Therapies

Gene therapy is a powerful tool that can potentially be used for treating neurodegenerative diseases such as amyotrophic lateral sclerosis, spinal muscular atrophy, Parkinson's disease, and Alzheimer's disease. Follow the latest research in this field with this feed.

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Neurofibromatosis is a genetic disorder that causes tumors to form in the central nervous system. While these tumors are typically benign, they can become malignant. Other complications of neurofibromatosis include hearing loss, learning impairment, vision loss, etc. Here is the latest research.
Neurofibromatosis

Neurofibromatosis is a genetic disorder that causes tumors to form in the central nervous system. While these tumors are typically benign, they can become malignant. Other complications of neurofibromatosis include hearing loss, learning impairment, vision loss, etc. Here is the latest research.

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Neuroinflammation is a complex process that has been noted in multiple neurological disease states such as brain injuries, infectious disease, and neurodegenerative disorders. Microglia and other cell types are activated and initiate a pro inflammatory response. Here is the latest research on neuroinflammation.
Neuroinflammation
Fast Growing

Neuroinflammation is a complex process that has been noted in multiple neurological disease states such as brain injuries, infectious disease, and neurodegenerative disorders. Microglia and other cell types are activated and initiate a pro inflammatory response. Here is the latest research on neuroinflammation.

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Neuromodulation alters nerve activity by delivering electrical or pharmaceutical agents directly to a target area. This feed focuses on different types of light therapies and methods, including optogenetics and low-level laser applications.
Neuromodulation
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Neuromodulation alters nerve activity by delivering electrical or pharmaceutical agents directly to a target area. This feed focuses on different types of light therapies and methods, including optogenetics and low-level laser applications.

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Neuromuscular disorders result in impaired functioning of the muscles. Discover the latest research on these disorders, including Amyotrophic Lateral Sclerosis, and the underlying genetic aspects here.
Neuromuscular Disorders
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Neuromuscular disorders result in impaired functioning of the muscles. Discover the latest research on these disorders, including Amyotrophic Lateral Sclerosis, and the underlying genetic aspects here.

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This feed focuses on neuronal and network function and dysfunction in neurodegenerative disorders and the effects of tauopathies on synapses and memory.
Neuronal and Network Dysfunction in ND

This feed focuses on neuronal and network function and dysfunction in neurodegenerative disorders and the effects of tauopathies on synapses and memory.

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Induced pluripotent stem cells (iPSCs) may be obtained by direct reprogramming of different somatic cells to a pluripotent state by forced expression of a handful of transcription factors. This feed focuses on the generation and application of neurons differentiated from/to iPSCs.
Neurons and iPSCs
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Induced pluripotent stem cells (iPSCs) may be obtained by direct reprogramming of different somatic cells to a pluripotent state by forced expression of a handful of transcription factors. This feed focuses on the generation and application of neurons differentiated from/to iPSCs.

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Neuroregeneration is the growth or repair of neural cells and tissues following damage or injury. Discover the latest research on neuroregeneration here.
Neuroregeneration

Neuroregeneration is the growth or repair of neural cells and tissues following damage or injury. Discover the latest research on neuroregeneration here.

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Alterations in nucleocytoplasmic transport, controlled by the nuclear pore complex, may be involved in the pathomechanism underlying multiple neurodegenerative diseases including Amyotrophic Lateral Sclerosis and Frontotemporal Dementia. Here is the latest research on the nuclear pore complex in ALS and FTD.
Nuclear Pore Complex in ALS/FTD

Alterations in nucleocytoplasmic transport, controlled by the nuclear pore complex, may be involved in the pathomechanism underlying multiple neurodegenerative diseases including Amyotrophic Lateral Sclerosis and Frontotemporal Dementia. Here is the latest research on the nuclear pore complex in ALS and FTD.

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Olivopontocerebellar atrophy (OPCA) refers to neurodegeneration of the inferior olivary nucleus, pons, and the cerebellum, and is characterized by cerebellar ataxia and other neurological problems. Here is the latest research.
Olivopontocerebellar Atrophy
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Olivopontocerebellar atrophy (OPCA) refers to neurodegeneration of the inferior olivary nucleus, pons, and the cerebellum, and is characterized by cerebellar ataxia and other neurological problems. Here is the latest research.

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Opsoclonus-Myoclonus Syndrome (OMS) is a rare inflammatory neurological disorder characterized by ocular, sleep, and behavioural disorders, as well as ataxia. Here is the latest research on OMS.
Opsoclonus-Myoclonus Syndrome
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Opsoclonus-Myoclonus Syndrome (OMS) is a rare inflammatory neurological disorder characterized by ocular, sleep, and behavioural disorders, as well as ataxia. Here is the latest research on OMS.

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Each cellular organelle plays a specific role in keeping a cell healthy. Different organelles, such as lysosomes and mitochondria, are implicated in neurodegenerative diseases. This feed focuses on cellular organelles and their role in neurodegeneration.
Organelles in Neurodegeneration
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Each cellular organelle plays a specific role in keeping a cell healthy. Different organelles, such as lysosomes and mitochondria, are implicated in neurodegenerative diseases. This feed focuses on cellular organelles and their role in neurodegeneration.

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Cerebral organoids derived from induced pluripotent stem (IPS) cells have illuminated early developmental events altered by disease processes, and may be a promising avenue for future therapies in neurodegenerative disorders. Here is the latest research on organoids and neurodegeneration.
Organoids & Neurodegeneration
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Cerebral organoids derived from induced pluripotent stem (IPS) cells have illuminated early developmental events altered by disease processes, and may be a promising avenue for future therapies in neurodegenerative disorders. Here is the latest research on organoids and neurodegeneration.

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P2Y12 is a G-protein-coupled receptor known for its role in platelet activation. However, its role in the CNS and involvement in microglial activation and renewal is less well understood. Discover the latest research on P2Y12 and microglial self-renewal here.
P2Y12 & Microglial Self-Renewal

P2Y12 is a G-protein-coupled receptor known for its role in platelet activation. However, its role in the CNS and involvement in microglial activation and renewal is less well understood. Discover the latest research on P2Y12 and microglial self-renewal here.

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Paraneoplastic disorders of the nervous system are syndromes that affect diverse organs systems (including endocrine, neurologic, dermatologic systems, etc.) that occur in association with systemic cancer. Here is the latest research.
Paraneoplastic Syndromes of the Nervous System
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Paraneoplastic disorders of the nervous system are syndromes that affect diverse organs systems (including endocrine, neurologic, dermatologic systems, etc.) that occur in association with systemic cancer. Here is the latest research.

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Parkinson's Disease is a progressive neurodegenerative disorder characterized by loss of muscle control, including trembling of the limbs and impaired balance. Here is the latest research on this disease.
Parkinson's Disease

Parkinson's Disease is a progressive neurodegenerative disorder characterized by loss of muscle control, including trembling of the limbs and impaired balance. Here is the latest research on this disease.

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Autophagy leads to degradation of damaged proteins and organelles by the lysosome. Impaired autophagy has been implicated in several diseases. Here is the role of autophagy in Parkinson’s disease.
Parkinson's Disease & Autophagy
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Autophagy leads to degradation of damaged proteins and organelles by the lysosome. Impaired autophagy has been implicated in several diseases. Here is the role of autophagy in Parkinson’s disease.

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Alpha-synuclein is an integral component of Lewy bodies which are comprised of protein clumps and are a pathological hallmark of Parkinson’s disease. Here is the latest research on alpha-synuclein and Parkinson's disease.
Parkinson's Disease: Alpha-Synuclein

Alpha-synuclein is an integral component of Lewy bodies which are comprised of protein clumps and are a pathological hallmark of Parkinson’s disease. Here is the latest research on alpha-synuclein and Parkinson's disease.

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Parkinson’s disease is a progressive neurodegenerative disorder that affects movement. This feed focuses on Parkinson's disease research using animal models.
Parkinson's Disease: Animal Models

Parkinson’s disease is a progressive neurodegenerative disorder that affects movement. This feed focuses on Parkinson's disease research using animal models.

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Parkinson’s disease is a neurodegenerative disease characterized by involuntary movement, decreased movement, rigidity, and abnormal walking and posture. Targeting markers in the earliest stages of the disease may mitigate its progression. This feed focuses on biomarkers for Parkinson’s disease.
Parkinson's Disease: Biomarkers

Parkinson’s disease is a neurodegenerative disease characterized by involuntary movement, decreased movement, rigidity, and abnormal walking and posture. Targeting markers in the earliest stages of the disease may mitigate its progression. This feed focuses on biomarkers for Parkinson’s disease.

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Parkinson’s disease (PD) is a neurodegenerative disorder that affects movement. This feed focuses on therapeutic aspects, diagnostic tools including medical imaging, and clinical trials in PD.
Parkinson's Disease: Diagnostics & Clinical Trials

Parkinson’s disease (PD) is a neurodegenerative disorder that affects movement. This feed focuses on therapeutic aspects, diagnostic tools including medical imaging, and clinical trials in PD.

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Parkinson’s disease is a progressive neurodegenerative disorder that affects movement. This feed focuses on genetic screening approaches and genetic predispositions in Parkinson’s disease.
Parkinson's Disease: Genetics

Parkinson’s disease is a progressive neurodegenerative disorder that affects movement. This feed focuses on genetic screening approaches and genetic predispositions in Parkinson’s disease.

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Parkinson’s disease (PD) is a neurodegenerative disorder that affects movement. This feed focuses on leucine-rich repeat kinase 2 (LRRK2) which is the most prominent genetic contributor to PD.
Parkinson's Disease: LRRK2

Parkinson’s disease (PD) is a neurodegenerative disorder that affects movement. This feed focuses on leucine-rich repeat kinase 2 (LRRK2) which is the most prominent genetic contributor to PD.

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The gut microbiota shares a symbiotic relationship with the human host. Dysbiosis is linked with several neurological pathologies including Parkinson’s disease (PD). Here is the latest research on the modulation of PD by the gut microbiota.
Parkinson's Disease: Microbiome
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The gut microbiota shares a symbiotic relationship with the human host. Dysbiosis is linked with several neurological pathologies including Parkinson’s disease (PD). Here is the latest research on the modulation of PD by the gut microbiota.

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Neurodegenerative diseases may be caused in part by triggers from peripheral body systems or inflammation mediated by brain cells other than neurons. Understanding these peripheral factors is necessary for designing treatments. Additionally, genetic tools allowing for delivery of transgenes past the blood-brain barrier are important for treatments of any CNS diseases. Discover the latest research on neurodegenerative disorders, their peripheral causes, and viral vectors for CNS delivery here.
Peripheral Causes of Neurodegeneration
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Neurodegenerative diseases may be caused in part by triggers from peripheral body systems or inflammation mediated by brain cells other than neurons. Understanding these peripheral factors is necessary for designing treatments. Additionally, genetic tools allowing for delivery of transgenes past the blood-brain barrier are important for treatments of any CNS diseases. Discover the latest research on neurodegenerative disorders, their peripheral causes, and viral vectors for CNS delivery here.

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This feed focuses on the pharmacology of proteinopathies - diseases in which proteins abnormally aggregate (i.e. Alzheimer’s, Parkinson’s, etc.). Discover the latest research in this field with this feed.
Pharmacology of Proteinopathies

This feed focuses on the pharmacology of proteinopathies - diseases in which proteins abnormally aggregate (i.e. Alzheimer’s, Parkinson’s, etc.). Discover the latest research in this field with this feed.

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Pontocerebellar hypoplasias are a group of neurodegenerative autosomal recessive disorders with prenatal onset, atrophy or hypoplasia of the cerebellum, hypoplasia of the ventral pons, microcephaly, variable neocortical atrophy and severe mental and motor impairments. Here is the latest research on pontocerebellar hypoplasia.
Pontocerebellar Hypoplasia

Pontocerebellar hypoplasias are a group of neurodegenerative autosomal recessive disorders with prenatal onset, atrophy or hypoplasia of the cerebellum, hypoplasia of the ventral pons, microcephaly, variable neocortical atrophy and severe mental and motor impairments. Here is the latest research on pontocerebellar hypoplasia.

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Postpoliomyelitis syndrome is characterized by a sudden or progressive loss of muscle strength, muscle atrophy, muscle pain, fatigue, intolerance to cold, after a period of at least 15 years from the acute polio virus infection, a period of neurological and functional stability. Here is the latest research.
Postpoliomyelitis Syndrome
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Postpoliomyelitis syndrome is characterized by a sudden or progressive loss of muscle strength, muscle atrophy, muscle pain, fatigue, intolerance to cold, after a period of at least 15 years from the acute polio virus infection, a period of neurological and functional stability. Here is the latest research.

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Prion diseases or transmissible spongiform encephalopathies are rare progressive neurogenerative diseases caused by misfolded prion proteins. Here is the latest research in this domain.
Prion Disease

Prion diseases or transmissible spongiform encephalopathies are rare progressive neurogenerative diseases caused by misfolded prion proteins. Here is the latest research in this domain.

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Prion diseases are fatal neurodegenerative and infectious disorders characterized by structural transition of the host-encoded cellular prion protein into the aberrantly folded pathologic isoform of the protein. Here is the latest research on diagnostic strategies for prion diseases.
Prion Disease: Diagnostic Strategies

Prion diseases are fatal neurodegenerative and infectious disorders characterized by structural transition of the host-encoded cellular prion protein into the aberrantly folded pathologic isoform of the protein. Here is the latest research on diagnostic strategies for prion diseases.

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Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion proteins, as an important biomarker of prion disease, are responsible for the transmissible spongiform encephalopathies. Here is the latest research investigating biomarkers in prion diseases.
Prion Diseases: Biomarkers
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Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion proteins, as an important biomarker of prion disease, are responsible for the transmissible spongiform encephalopathies. Here is the latest research investigating biomarkers in prion diseases.

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Misfolded prion proteins characterize a number of fatal neurodegenerative diseases. Distinct prion strains are associated with different prion protein conformations and glycosylation patterns and associate with disease progression and severity. Here is the latest research on strain diversity and neurotoxicity in prion diseases.
Prion Diseases: Strain Diversity & Neurotoxicity
Fast Growing

Misfolded prion proteins characterize a number of fatal neurodegenerative diseases. Distinct prion strains are associated with different prion protein conformations and glycosylation patterns and associate with disease progression and severity. Here is the latest research on strain diversity and neurotoxicity in prion diseases.

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Parkinson's disease is a progressive, neurodegenerative disorder that affects both motor and cognitive function. The etiology of the disease is mostly unknown, but it probably involves both environmental and genetic. This feed focuses on profiling and defining Parkinson’s disease using biological and computational methods.
Profiling Parkinson's Disease

Parkinson's disease is a progressive, neurodegenerative disorder that affects both motor and cognitive function. The etiology of the disease is mostly unknown, but it probably involves both environmental and genetic. This feed focuses on profiling and defining Parkinson’s disease using biological and computational methods.

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Progressive Multifocal Leukoencephalopathy (PML) is a progressive demyelinating disease of the CNS caused by JC polyoma virus (JCV) reactivation. It emerges in individuals with impaired cell-mediated immunity. This feed follows research into this disease.
Progressive Multifocal Leukoencephalopathy

Progressive Multifocal Leukoencephalopathy (PML) is a progressive demyelinating disease of the CNS caused by JC polyoma virus (JCV) reactivation. It emerges in individuals with impaired cell-mediated immunity. This feed follows research into this disease.

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Progressive Supranuclear Palsy (PSP) is a rare neurodegenerative disorder that affects movement, gait, balance, and speech. Discover the latest research on PSP here.
Progressive Supranuclear Palsy
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Progressive Supranuclear Palsy (PSP) is a rare neurodegenerative disorder that affects movement, gait, balance, and speech. Discover the latest research on PSP here.

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This feed focuses on proteotoxicity, which plays an important role in aging-associated inflammation of the CNS and is a major hallmark of different neurodegenerative disorders. Discover the latest research on proteotoxicity in inflammation and neurodegeneration here.
Proteotoxicity in Inflammation & Dementia

This feed focuses on proteotoxicity, which plays an important role in aging-associated inflammation of the CNS and is a major hallmark of different neurodegenerative disorders. Discover the latest research on proteotoxicity in inflammation and neurodegeneration here.

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Tau mutations in frontotemporal dementia and parkinsonism linked to chromosome 17 (ftdp-17) are associated with changes in alternative RNA splicing. Here is the latest research on RNA & Tau in Frontotemporal Dementia.
RNA & Tau in Frontotemporal Dementia
Going Viral

Tau mutations in frontotemporal dementia and parkinsonism linked to chromosome 17 (ftdp-17) are associated with changes in alternative RNA splicing. Here is the latest research on RNA & Tau in Frontotemporal Dementia.

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While a link has been established between RNA, RNA-binding proteins, and motor proteins, mechanisms underlying RNA trafficking are still unclear. Furthermore, interactions between cargo and motor proteins are dysregulated in microsatellite repeat expansion diseases, including Huntington’s disease. Here is the latest research focusing on RNA trafficking and neurodegeneration.
RNA in Neurodegeneration
Going Viral

While a link has been established between RNA, RNA-binding proteins, and motor proteins, mechanisms underlying RNA trafficking are still unclear. Furthermore, interactions between cargo and motor proteins are dysregulated in microsatellite repeat expansion diseases, including Huntington’s disease. Here is the latest research focusing on RNA trafficking and neurodegeneration.

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Some neurodegenerative disorders are believed to be due to RNA-induced cellular toxicity caused by the expansion of nucleotide-repeat sequences in DNA. Discover the latest research on RNA-mediated toxicity in neurodegeneration here.
RNA-mediated Toxicity in Neurodegeneration
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Some neurodegenerative disorders are believed to be due to RNA-induced cellular toxicity caused by the expansion of nucleotide-repeat sequences in DNA. Discover the latest research on RNA-mediated toxicity in neurodegeneration here.

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Reactive oxygen species (ROS) are reactive chemicals containing oxygen which may have complex roles in promoting the development of neurodegenerative diseases. They are generated as a byproduct of oxidative energy metabolism and play a role in regulating several intracellular signaling pathways. Here is the latest research on ROS metabolism and signaling in neurodegeneration.
ROS: Metabolism & Signaling in Neurodegeneration

Reactive oxygen species (ROS) are reactive chemicals containing oxygen which may have complex roles in promoting the development of neurodegenerative diseases. They are generated as a byproduct of oxidative energy metabolism and play a role in regulating several intracellular signaling pathways. Here is the latest research on ROS metabolism and signaling in neurodegeneration.

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Refsum disease is an autosomal recessive disorder of lipid metabolism characterized by adult-onset retinitis pigmentosa, anosmia, sensory neuropathy, ataxia, and an accumulation of phytanic acid in plasma and tissues. Here is the latest research on this peroxisomal disorder.
Refsum Disease

Refsum disease is an autosomal recessive disorder of lipid metabolism characterized by adult-onset retinitis pigmentosa, anosmia, sensory neuropathy, ataxia, and an accumulation of phytanic acid in plasma and tissues. Here is the latest research on this peroxisomal disorder.

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Risk factors for neurodegeneration include trauma, lifestyle, and allelic variants of disease-associated genes with incomplete penetrance. Here is the latest research.
Risk Factors for Neurodegeneration
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Risk factors for neurodegeneration include trauma, lifestyle, and allelic variants of disease-associated genes with incomplete penetrance. Here is the latest research.

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Scrapie is one of the several transmissible spongiform encephalopathies that affect the nervous system of sheep and goats. Here is the latest research on this fatal degenerative disease.
Scrapie
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Scrapie is one of the several transmissible spongiform encephalopathies that affect the nervous system of sheep and goats. Here is the latest research on this fatal degenerative disease.

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Secondary Parkinson disease refers to a group of disorders that present similarly to Parkinson disease but have different etiology. Secondary Parkinson disease is caused due to the blockade of or interference in dopamine’s action in the basal ganglia. Here is the latest research.
Secondary Parkinson Disease
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Secondary Parkinson disease refers to a group of disorders that present similarly to Parkinson disease but have different etiology. Secondary Parkinson disease is caused due to the blockade of or interference in dopamine’s action in the basal ganglia. Here is the latest research.

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There are sex differences in prevalence or outcome of neurodevelopmental and neurodegenerative disorders. For instance, Multiple Sclerosis is more common in women, whereas Parkinson’s disease is more common in men. Here is the latest research on sexual dimorphism in neurodegeneration.
Sexual Dimorphism in Neurodegeneration

There are sex differences in prevalence or outcome of neurodevelopmental and neurodegenerative disorders. For instance, Multiple Sclerosis is more common in women, whereas Parkinson’s disease is more common in men. Here is the latest research on sexual dimorphism in neurodegeneration.

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Combining genomic, transcriptomic, proteomic, and metabolomic data of the human spinal cord at the single cell level will help build a single cell atlas to give insight into the health and disease of the spinal cord. Discover the latest research on the single cell atlas of the human spinal cord here.
Single Cell Atlas of the Human Spinal Cord
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Combining genomic, transcriptomic, proteomic, and metabolomic data of the human spinal cord at the single cell level will help build a single cell atlas to give insight into the health and disease of the spinal cord. Discover the latest research on the single cell atlas of the human spinal cord here.

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Single-cell RNA sequencing is a technique that is used to study isolated single cells and obtain their transcripts, which are then used to generate sequence libraries. This approach can be used to learn more about the cellular diversity in the brain and understand neurological diseases. Here is the latest research on single-cell RNA sequencing in the brain.
Single-Cell RNA Sequencing: Brain
Fast Growing

Single-cell RNA sequencing is a technique that is used to study isolated single cells and obtain their transcripts, which are then used to generate sequence libraries. This approach can be used to learn more about the cellular diversity in the brain and understand neurological diseases. Here is the latest research on single-cell RNA sequencing in the brain.

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Spinal cord injury is a serious type of trauma and can cause temporary or permanent damage, including loss of muscle function, sensation, or autonomic functions. Here is the latest research on spinal cord injuries.
Spinal Cord Injuries
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Spinal cord injury is a serious type of trauma and can cause temporary or permanent damage, including loss of muscle function, sensation, or autonomic functions. Here is the latest research on spinal cord injuries.

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This feed describes latest advances in spinal cord regeneration research and potential nerve regeneration treatments after spinal cord injuries.
Spinal Cord Injury: Regeneration
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This feed describes latest advances in spinal cord regeneration research and potential nerve regeneration treatments after spinal cord injuries.

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Spinal muscular atrophy (SMA) is a neuromuscular disorder caused by genetic defects in the SMN1 gene, which encodes the survival motor neuron 1 (SMN1) protein. SMA is characterized by loss of lower motor neurons and progressive muscle wasting. Here is the latest research on this condition.
Spinal Muscular Atrophy

Spinal muscular atrophy (SMA) is a neuromuscular disorder caused by genetic defects in the SMN1 gene, which encodes the survival motor neuron 1 (SMN1) protein. SMA is characterized by loss of lower motor neurons and progressive muscle wasting. Here is the latest research on this condition.

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Spinocerebellar ataxias (SCA) are hereditary and characterized by cerebellar degeneration, which affects muscle coordination and balance. Discover the latest research on SCA and related ataxias here.
Spinocerebellar & Related Ataxias
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Spinocerebellar ataxias (SCA) are hereditary and characterized by cerebellar degeneration, which affects muscle coordination and balance. Discover the latest research on SCA and related ataxias here.

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microRNAs (miRNAs) are key regulatory RNAs that control gene expression in various biological processes and human diseases such as Spinocerebellar Ataxia. Here are the latest discoveries pertaining to miRNAs and this disease.
Spinocerebellar Ataxia: miRNAs

microRNAs (miRNAs) are key regulatory RNAs that control gene expression in various biological processes and human diseases such as Spinocerebellar Ataxia. Here are the latest discoveries pertaining to miRNAs and this disease.

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The use of stem cells is being explored as a treatment option for neuroimmune disorders, including Multiple Sclerosis. The immune system has been demonstrated to have negative impacts on cognitive function and neurogenesis and is a potential target for therapeutic intervention. Discover the latest research on stem cells and neuroimmunology.
Stem Cells & Neuroimmunology
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The use of stem cells is being explored as a treatment option for neuroimmune disorders, including Multiple Sclerosis. The immune system has been demonstrated to have negative impacts on cognitive function and neurogenesis and is a potential target for therapeutic intervention. Discover the latest research on stem cells and neuroimmunology.

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Stem cells have the potential to develop into many different types of cells in the body and may be useful as a therapy for several diseases including neurodegenerative diseases. Discover the latest research on stem cells and neurodegenerative diseases here.
Stem Cells in Neurodegenerative Diseases
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Stem cells have the potential to develop into many different types of cells in the body and may be useful as a therapy for several diseases including neurodegenerative diseases. Discover the latest research on stem cells and neurodegenerative diseases here.

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Striatonigral degeneration is a sporadic, progressive neurodegenerative disorder resulting from a disrupted connection between the striatum and substantia nigra, affecting balance and movement. This feed follows the research intop this disease.
Striatonigral Degeneration

Striatonigral degeneration is a sporadic, progressive neurodegenerative disorder resulting from a disrupted connection between the striatum and substantia nigra, affecting balance and movement. This feed follows the research intop this disease.

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As we age, the number of synapses present in the human brain starts to decline, but in neurodegenerative diseases this occurs at a much more rapid rate. A reduction in synaptic density has been observed in neurodegenerative disorders, including Multiple Sclerosis, providing a potential area to target for treatment. Here is the latest research on synapse loss in neurodegenerative disorders and as a potential therapeutic target.
Synapse Loss in Neurodegenerative Disorders
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As we age, the number of synapses present in the human brain starts to decline, but in neurodegenerative diseases this occurs at a much more rapid rate. A reduction in synaptic density has been observed in neurodegenerative disorders, including Multiple Sclerosis, providing a potential area to target for treatment. Here is the latest research on synapse loss in neurodegenerative disorders and as a potential therapeutic target.

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Synaptic alterations are emerging as critical determinants of neurodegeneration. Here is the latest research on alterations of synaptic transmission and plasticity in neurodegeneration and neurodegenerative disorders.
Synaptic Transmission in Neurodegeneration

Synaptic alterations are emerging as critical determinants of neurodegeneration. Here is the latest research on alterations of synaptic transmission and plasticity in neurodegeneration and neurodegenerative disorders.

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Synucleinopathies are neurodegenerative diseases characterized by accumulation of alpha-synuclein protein aggregates in neurons and glial cells. Discover the latest research on synucleinopathies here.
Synucleinopathies

Synucleinopathies are neurodegenerative diseases characterized by accumulation of alpha-synuclein protein aggregates in neurons and glial cells. Discover the latest research on synucleinopathies here.

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Tau and alpha-synuclein are proteins that form aggregates which characterize various neurodegenerative disorders including Parkinson’s disease. This feed focuses on the T cell response to tau and alpha-synuclein.
T Cell Reactivity to Tau & Alpha-Synuclein

Tau and alpha-synuclein are proteins that form aggregates which characterize various neurodegenerative disorders including Parkinson’s disease. This feed focuses on the T cell response to tau and alpha-synuclein.

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Aberrant T cell activity is known to mediate considerable damage in a variety of disease states, and the immune system has been recognized to contribute to the development of neurodegenerative disorders. It is therefore important to identify the characteristics of T cells involved in the pathogenesis of neurodegenerative diseases. Discover the latest research on T cells and neurodegeneration here.
T Lymphocytes & Neurodegenerative Disorders

Aberrant T cell activity is known to mediate considerable damage in a variety of disease states, and the immune system has been recognized to contribute to the development of neurodegenerative disorders. It is therefore important to identify the characteristics of T cells involved in the pathogenesis of neurodegenerative diseases. Discover the latest research on T cells and neurodegeneration here.

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TAR DNA binding protein (TDP43) is a DNA and RNA binding protein that is implicated in several neurodegenerative disorders known as “TDP43 proteinopathies" including Alzheimer's disease, amyotrophic lateral sclerosis and frontotemporal lobe dementia. Here is the latest research.
TDP-43 Proteinopathies
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TAR DNA binding protein (TDP43) is a DNA and RNA binding protein that is implicated in several neurodegenerative disorders known as “TDP43 proteinopathies" including Alzheimer's disease, amyotrophic lateral sclerosis and frontotemporal lobe dementia. Here is the latest research.

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Triggering receptor expressed on myeloid cells 2 (TREM2) is an important gene implicated in neurodegenerative disorders like Alzheimer's Disease, Parkinson's Disease, and Frontotemporal Lobar Degeneration. Discover the latest research studying the molecular mechanisms of TREM2 in neurodegeneration here.
TREM2 & Neurodegeneration

Triggering receptor expressed on myeloid cells 2 (TREM2) is an important gene implicated in neurodegenerative disorders like Alzheimer's Disease, Parkinson's Disease, and Frontotemporal Lobar Degeneration. Discover the latest research studying the molecular mechanisms of TREM2 in neurodegeneration here.

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Tau proteins include six highly soluble protein isoforms produced by alternative splicing from the gene MAPT (microtubule-associated protein tau). Combating tau-associated pathologies is a key focus of neurodegeneration research. Here is the latest research on tau and neurodegeneration.
Tau & Neurodegeneration
Influential

Tau proteins include six highly soluble protein isoforms produced by alternative splicing from the gene MAPT (microtubule-associated protein tau). Combating tau-associated pathologies is a key focus of neurodegeneration research. Here is the latest research on tau and neurodegeneration.

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Tau aggregation is an important characteristic in some neurodegenerative disorders, including Alzheimer’s Disease. Discover the latest research using genomics to understand Tau aggregation.
Tau Aggregation: Genomics
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Tau aggregation is an important characteristic in some neurodegenerative disorders, including Alzheimer’s Disease. Discover the latest research using genomics to understand Tau aggregation.

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Tau vaccination and administration of anti-tau antibodies can prevent pathology and cognitive impairment in transgenic mouse models of tauopathy, suggesting that therapies which increase anti-tau antibodies might slow the development and/or progression of tauopathies including Alzheimer's disease. Here is the latest research.
Tau Antibody Therapy in Neurodegenerative Diseases

Tau vaccination and administration of anti-tau antibodies can prevent pathology and cognitive impairment in transgenic mouse models of tauopathy, suggesting that therapies which increase anti-tau antibodies might slow the development and/or progression of tauopathies including Alzheimer's disease. Here is the latest research.

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Tau toxicity has been implicated in the emergence of synaptic dysfunction in Alzheimer's disease. Here is the latest research on Tau Toxicity in the Synapses.
Tau Toxicity in the Synapse

Tau toxicity has been implicated in the emergence of synaptic dysfunction in Alzheimer's disease. Here is the latest research on Tau Toxicity in the Synapses.

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Tauopathies are neurodegenerative disorders caused due to misfolding and aggregation of the tau protein in neurofibrillary tangles in the brain. Discover the latest research on tauopathies here.
Tauopathies
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Tauopathies are neurodegenerative disorders caused due to misfolding and aggregation of the tau protein in neurofibrillary tangles in the brain. Discover the latest research on tauopathies here.

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Tauopathies are neurodegenerative disorders caused due to misfolding and aggregation of the tau protein in neurofibrillary tangles in the brain. This feed focuses on the structural basis of protein pathology in tauopathies. Here is the latest research.
Tauopathies: Structural Basis of Protein Pathology

Tauopathies are neurodegenerative disorders caused due to misfolding and aggregation of the tau protein in neurofibrillary tangles in the brain. This feed focuses on the structural basis of protein pathology in tauopathies. Here is the latest research.

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Tauopathies are neurodegenerative disorders that are caused due to the accumulation of abnormal tau proteins in neurons and/or the glia. Here is the latest research on tauopathy biochemistry.
Tauopathy Biochemistry
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Tauopathies are neurodegenerative disorders that are caused due to the accumulation of abnormal tau proteins in neurons and/or the glia. Here is the latest research on tauopathy biochemistry.

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Torsion dystonia is a movement disorder characterized by loss of control of voluntary movements appearing as sustained muscle contractions and/or abnormal postures. Here is the latest research.
Torsion Dystonia

Torsion dystonia is a movement disorder characterized by loss of control of voluntary movements appearing as sustained muscle contractions and/or abnormal postures. Here is the latest research.

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Tourette Syndrome is a neuropsychiatric disorder characterized by multiple motor and vocal tics and often associated with neurobehavioral comorbidities, such as obsessive-compulsive disorder. Here is the latest research on this childhood-onset neurodevelopmental disorder.
Tourette Syndrome

Tourette Syndrome is a neuropsychiatric disorder characterized by multiple motor and vocal tics and often associated with neurobehavioral comorbidities, such as obsessive-compulsive disorder. Here is the latest research on this childhood-onset neurodevelopmental disorder.

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Triplet Repeat Disorders are genetic disorders that occur as a result of expansion of trinucleotide repeats in certain genes. Examples include Huntington’s disease, certain types of spinocerebellar ataxia, and Fragile X syndrome, among others. Here is the latest research.
Triplet Repeat Disorders
Fast Growing

Triplet Repeat Disorders are genetic disorders that occur as a result of expansion of trinucleotide repeats in certain genes. Examples include Huntington’s disease, certain types of spinocerebellar ataxia, and Fragile X syndrome, among others. Here is the latest research.

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Tuberous Sclerosis (TSC) is an autosomal dominant genetic disorder caused by mutations in TSC1 or TSC2 tumor suppressor genes and characterized by the development of benign hamartomas in multiple organs. Here is the latest research.
Tuberous Sclerosis

Tuberous Sclerosis (TSC) is an autosomal dominant genetic disorder caused by mutations in TSC1 or TSC2 tumor suppressor genes and characterized by the development of benign hamartomas in multiple organs. Here is the latest research.

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Vascular Dementia refers to the decline in thinking ability caused by cerebrovascular diseases. Discover the latest research on aspects underlying vascular dementia here.
Vascular Dementia

Vascular Dementia refers to the decline in thinking ability caused by cerebrovascular diseases. Discover the latest research on aspects underlying vascular dementia here.

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Wallerian degeneration refers to axonal degeneration in response to nerve fiber damage, where the axon distal to the injury site degenerates. Here is the latest research on Wallerian degeneration.
Wallerian Degeneration

Wallerian degeneration refers to axonal degeneration in response to nerve fiber damage, where the axon distal to the injury site degenerates. Here is the latest research on Wallerian degeneration.

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Wilson’s disease is an autosomal recessive disorder caused by mutation in the ATP7B gene and is characterized by copper build-up in the body. Discover the latest research on Wilson’s disease here.
Wilson Disease

Wilson’s disease is an autosomal recessive disorder caused by mutation in the ATP7B gene and is characterized by copper build-up in the body. Discover the latest research on Wilson’s disease here.

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Induced pluripotent stem cell (iPSCs) models can be generated to study mechanisms involved in neurodegeneration. The iPSCs can be selectively differentiated into specific neurons such as dopaminergic neurons that are susceptible to neurodegeneration. Discover the latest research on iPSC models in neurodegeneration here.
iPSC Models in Neurodegeneration
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Induced pluripotent stem cell (iPSCs) models can be generated to study mechanisms involved in neurodegeneration. The iPSCs can be selectively differentiated into specific neurons such as dopaminergic neurons that are susceptible to neurodegeneration. Discover the latest research on iPSC models in neurodegeneration here.

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Induced pluripotent stem cell-derived microglia have recently been developed and provide unlimited access to patient-derived material. These cells can be used to model and investigate the mechanism that underlie neurodegenerative diseases. This feed follows the latest research in this area using iPSC-derived microglia.
iPSC-derived Microglia in Neurodegeneration

Induced pluripotent stem cell-derived microglia have recently been developed and provide unlimited access to patient-derived material. These cells can be used to model and investigate the mechanism that underlie neurodegenerative diseases. This feed follows the latest research in this area using iPSC-derived microglia.

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