Alzheimer's Disease: Amyloid Beta

Alzheimer's disease is a neurodegenerative disease associated with the accumulation of amyloid plaques in the brain; these plaques are comprised of amyloid beta deposits. Here is the latest research in this field.

January 20, 2021

Improving the inhibition of β-amyloid aggregation by withanolide and withanoside derivatives

International Journal of Biological Macromolecules
Shreya DubeyRajagopal Subramanyam
January 20, 2021

The Structural Basis of Amyloid Strains in Alzheimer's Disease

ACS Biomaterials Science & Engineering
Lee Makowski
January 20, 2021

ATP-sensitive potassium channel subunits in the neuroinflammation: novel drug targets in neurodegenerative disorders

CNS & Neurological Disorders Drug Targets
Fatima MaqoudDomenico Tricarico
January 19, 2021

Alzheimer's Disease, Neural Plasticity, and Functional Recovery

Journal of Alzheimer's Disease : JAD
Daymara Mercerón-MartínezAdrian G Palacios
January 18, 2021
Open Access

High temperature promotes amyloid β-protein production and γ-secretase complex formation via Hsp90

The Journal of Biological Chemistry
Arshad Ali NooraniMakoto Michikawa
January 20, 2021

Microglial store-operated calcium signaling in health and in Alzheimer's disease

Current Alzheimer Research
James G McLarnon
January 20, 2021

Caterpillar Medicinal Mushroom, Cordyceps militaris (Ascomycetes), Protects Aβ1-42-Induced Neurologic Damage in C6 Glial Cells

International Journal of Medicinal Mushrooms
Mei Tong HeEun Ju Cho
January 19, 2021
Open Access

Plasma Lipids as Biomarkers for Alzheimer's Disease: A Systematic Review

Mehak Agarwal, Safeera Khan
January 16, 2021
Open Access

Inhibitory Neural Network's Impairments at Hippocampal CA1 LTP in an Aged Transgenic Mouse Model of Alzheimer's Disease

International Journal of Molecular Sciences
Hyeon Jeong SeoByeong C Kim
January 18, 2021

Inflammation and Parkinson's disease pathogenesis: Mechanisms and therapeutic insight

Progress in Molecular Biology and Translational Science
Gianluigi ForloniClaudia Balducci
January 19, 2021

Effect of Alzheimer's Disease and Lewy Body Disease on Metabolic Changes

Journal of Alzheimer's Disease : JAD
Yang Hyun LeeByoung Seok Ye
January 20, 2021

Early-Onset Alzheimer's Disease: What Is Missing in Research?

Current Neurology and Neuroscience Reports
Temitope AyodeleChristiane Reitz
January 20, 2021
Open Access

An "epitomic" analysis of the specificity of conformation-dependent, anti-Aß amyloid monoclonal antibodies

The Journal of Biological Chemistry
Jorge Mauricio Reyes-RuizCharles Glabe
January 19, 2021
Open Access

Enhancing top-down proteomics of brain tissue with FAIMS

BioRxiv : the Preprint Server for Biology
J. M. FulcherVladislav A Petyuk
January 18, 2021

The cellular machinery of post-endocytic APP trafficking in Alzheimer's disease: A future target for therapeutic intervention?

Progress in Molecular Biology and Translational Science
Lawrence S B Goldstein, Utpal Das

Sign up to follow this feed and discover related papers.

Related Feeds

22q11 Deletion Syndrome

22q11.2 deletion syndrome, also known as DiGeorge syndrome, is a congenital disorder caused as a result of a partial deletion of chromosome 22. Here is the latest research.

3D Cellular Models of Brain and Neurodegeneration

Brain organoids are three-dimensional in vitro cellular models of the brain that can recapitulate many processes such as the neurodevelopment. In addition, these organoids can be combined with other cell types, such as neurons and astrocytes to study their interactions in assembloids. Disease processes can also be modeled by induced pluripotent stem cell-derived organoids and assembloids from patients with neurodegenerative disorders. Discover the latest research on the models here.

4H Leukodystrophy

4H Lekodystrophy involves hypomyelination, hypogonadotropic hypogonadism and hypodontia. It is also known as POLR-3Related Leukodystrophy. 4H syndrome symptoms include delayed or abnormal puberty, issues with central nervous system signal transmission, abnormal development of teeth, and symptoms tend to progressively get worse over time. There is not yet a known cure.


TAR DNA-binding protein 43 (TDP-43) is a pathological protein identified in sporadic Amyotrophic Lateral Sclerosis (ALS) and Frontotemporal Dementia (FTD). Here are the latest discoveries pertaining to TDP-43 and these diseases.

ALS: Genetics

Amyotrophic Lateral Sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.

ALS: Pathogenic Mechanisms

Amyotrophic Lateral Sclerosis is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.

ALS: Phenotypes

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disorder characterized phenotypically by progressive muscle weakness. Clinical phenotypes of ALS can be classified based on the pattern, level, and area of onset (e.g. bulbar, cervical, lumbar). Here is the latest research investigating phenotypes of ALS.

ALS: Prions

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here is the latest research on ALS and prions.

ALS: Stress Granules

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research in this field.

ALS: Therapies

Amyotrophic Lateral Sclerosis (ALS) is associated with the death of neurons that control voluntary muscles. This feed followes the latest research into therapies for this progressive neurodegenerative disease.

© 2021 Meta ULC. All rights reserved