Axonal Neuropathies

Axonal Neuropathy refers to disease or dysfunction of nerves caused as a result of damage or degeneration of axons. Here is the latest research.

September 16, 2020

Comparison between plasma and cerebrospinal fluid biomarkers for the early diagnosis and association with survival in prion disease

Journal of Neurology, Neurosurgery, and Psychiatry
Samir Abu-RumeilehPiero Parchi
September 9, 2020

Delayed Onset of Central Pain due to Traumatic Axonal Injury of the Spinothalamic Tract in a Patient with Mild Traumatic Brain Injury

Pain Medicine : the Official Journal of the American Academy of Pain Medicine
Sung Ho Jang, You Sung Seo
August 31, 2020
Case Report
Open Access

Parkin Pleiotropy: Extremely Atypical Phenotypes in Patients With Compound Heterozygous Mutations

Tremor and Other Hyperkinetic Movements
Patricio Millar VernettiMarcelo Merello
September 5, 2020
Preprint
Open Access

rab-27 acts in an intestinal secretory pathway to inhibit axon regeneration in C. elegans

BioRxiv : the Preprint Server for Biology
A. T. Lin-MooreMarc Hammarlund
September 15, 2020

Traumatic Injury Reduces Amyloid Plaque Burden in the Transgenic 5xFAD Alzheimer's Mouse Spinal Cord

Journal of Alzheimer's Disease : JAD
Tak-Ho ChuPeter K Stys
September 18, 2020
Preprint

Drug library screen identifies inhibitors of toxic astrogliosis

MedRxiv : the Preprint Server for Health Sciences
R. R. MasvekarBibiana Bielekova
September 17, 2020
Review

Neuromuscular presentations in patients with COVID-19

Neurological Sciences : Official Journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology
Vimal Kumar PaliwalNidhi Tejan
September 18, 2020

A rare case of acute motor axonal neuropathy and myelitis related to SARS-CoV-2 infection

Journal of Neurology
Fabio Giuseppe MasuccioSolaro Claudio
September 13, 2020
Open Access

Autonomic dysfunction heralding acute motor axonal neuropathy in COVID-19

Journal of Neurovirology
Ritwik GhoshJulián Benito-León
August 31, 2020

Verification and characterisation of human digital Ruffini's sensory corpuscles

Journal of Anatomy
Ramón CoboJosé A Vega
September 9, 2020

Bi-allelic mutations in EGR2 cause autosomal recessive demyelinating neuropathy by disrupting the EGR2-NAB complex

European Journal of Neurology : the Official Journal of the European Federation of Neurological Societies
Vincenzo LupoTeresa Sevilla
September 4, 2020
Open Access

C9orf72 poly(GR) aggregation induces TDP-43 proteinopathy

Science Translational Medicine
Casey N CookLeonard Petrucelli
September 15, 2020
Open Access

Diffuse axonal injury associated with COVID-19 infection

Medicina Clínica
Alba Lopez-FernándezManuel Sánchez-Sánchez

Sign up to follow this feed and discover related papers.

Related Feeds

ALS

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS here.

ALS & FTD: TDP-43

ALS shares with a considerable proportion of FTD cases the same neuropathological substrate, namely, inclusions of abnormally phosphorylated protein tdp-43 (ptdp-43). Here are the latest discoveries pertaining to ptdp-43 and these diseases.

ALS - Pathogenic Mechanisms

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.

ALS - Phenotypes

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating phenotypes associated with this genetically heterogeneous disorder.

ALS: Genetics

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.

ALS: Prions

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here are the latest discoveries pertaining to this disease.

ALS: Stress Granules

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research.

ALS: Therapies

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS therapies here.

ALS: Transposon de-silencing

Transposon silencing is a form of transcriptional gene silencing. These gene silencing mechanisms are impaired in Amyotrophic lateral sclerosis (ALS). Here are the latest discoveries pertaining to transposon silencing and this disease.

Age-related Dementia

Age-related dementia (Alzheimer's disease) results from the destructive impact of the pulse on cerebral vasculature. Evidence is reviewed that the neuropathology of the dementia is caused by the breakdown of small cerebral vessels (silent microbleeds), that the microbleeds result from pulse-induced damage to the cerebral vessels, and that pulse becomes increasingly destructive with age, because of the age-related stiffening of the aorta and great arteries, which causes an increase in the intensity of the pressure pulse. Discover the latest research on age-related dementia here.

© 2020 Meta ULC. All rights reserved
/feed-previews/axonal-neuropathies/090e78de-67fd-4413-9339-1a414fb90d72