Feed Preview

Frontotemporal Dementia

Frontotemporal Dementia diagram by Atthapon Raksthaput, Shutterstock
Atthapon Raksthaput, Shutterstock

Frontotemporal dementia (FTD) refers to disorders caused by progressive neuronal loss in the frontal and temporal lobes of the brain. Here is the latest research on FTD and FTD-associated disorders.

Top 20 most recent papers
Acta Neuropathologica Communications

Analysis of neurodegenerative disease-causing genes in dementia with Lewy bodies

Acta Neuropathologica CommunicationsJanuary 31, 2020
Tatiana OrmeJose Bras
7
1
bioRxiv

Phase separation of toxic dipeptide repeat proteins related to C9orf72 ALS/FTD

bioRxivFebruary 1, 2020
Hamidreza JafariniaPatrick Onck
12
Journal of Geriatric Psychiatry and Neurology

Distinguishing Frontotemporal Dementia From Alzheimer Disease Through Everyday Function Profiles: Trajectories of Change

Journal of Geriatric Psychiatry and NeurologyFebruary 15, 2020
Clarissa M GiebelMizanur Khondoker
6
Journal of Alzheimer's Disease : JAD

Longitudinal Amyloid-β PET in Atypical Alzheimer's Disease and Frontotemporal Lobar Degeneration

Journal of Alzheimer's Disease : JADFebruary 11, 2020
Jennifer L WhitwellKeith A Josephs
4
1
Clinical Neuropathology

History of Pick's disease

Clinical NeuropathologyFebruary 1, 2020
Jacqueline Mikol
1
Brain : a Journal of Neurology

Morphometric network differences in ageing versus Alzheimer's disease dementia

Brain : a Journal of NeurologyFebruary 11, 2020
Alexa Pichet BinettePREVENT-AD Research Group
53
1
International Journal of Molecular Sciences

Innate Immunity: A Common Denominator between Neurodegenerative and Neuropsychiatric Diseases

International Journal of Molecular SciencesFebruary 13, 2020
Fabiana NovellinoGiuseppe Donato
3
1
Frontiers in Neurology

Functional Characterization of Atrophy Patterns Related to Cognitive Impairment

Frontiers in NeurologyFebruary 11, 2020
Gereon J SchnellbächerImis Dogan
1
1

See more papers from this feed

Related Feeds

ALS

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS here.

ALS & FTD: TDP-43

ALS shares with a considerable proportion of FTD cases the same neuropathological substrate, namely, inclusions of abnormally phosphorylated protein tdp-43 (ptdp-43). Here are the latest discoveries pertaining to ptdp-43 and these diseases.

ALS - Pathogenic Mechanisms

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.

ALS - Phenotypes

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating phenotypes associated with this genetically heterogeneous disorder.

ALS: Genetics

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.

ALS: Prions

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here are the latest discoveries pertaining to this disease.

ALS: Stress Granules

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research.

ALS: Therapies

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS therapies here.

ALS: Transposon de-silencing

Transposon silencing is a form of transcriptional gene silencing. These gene silencing mechanisms are impaired in Amyotrophic lateral sclerosis (ALS). Here are the latest discoveries pertaining to transposon silencing and this disease.

Age-related Dementia

Age-related dementia (Alzheimer's disease) results from the destructive impact of the pulse on cerebral vasculature. Evidence is reviewed that the neuropathology of the dementia is caused by the breakdown of small cerebral vessels (silent microbleeds), that the microbleeds result from pulse-induced damage to the cerebral vessels, and that pulse becomes increasingly destructive with age, because of the age-related stiffening of the aorta and great arteries, which causes an increase in the intensity of the pressure pulse. Discover the latest research on age-related dementia here.

© 2020 Meta ULC. All rights reserved
/feed-previews/frontotemporal-dementia/806b2e01-8987-48ff-a599-f71effb4d394