Inotropic Agents & Heart Disease

Inotropic agents alter the force or energy of muscular contractions. Negatively inotropic agents weaken the force of muscular contractions. Positively inotropic agents increase the strength of muscular contraction. Inotropic agents are used in hospitalized patients with acute decompensated heart failure, with reduced left ventricular ejection fraction and with signs of end-organ dysfunction in the setting of a low cardiac output, or with severe systolic heart failure awaiting heart transplant to maintain hemodynamic stability or as a bridge to decision. Discover the latest research on inotrpic agents and heart diseases here.

December 6, 2021

The mid-term outcome of interventricular septal hematoma after ventricular septal defect closure.

Asian Cardiovascular & Thoracic Annals
Masaya AokiNaoki Yoshimura
December 6, 2021
Open Access

Periplocymarin Alleviates Doxorubicin-Induced Heart Failure and Excessive Accumulation of Ceramides.

Frontiers in Cardiovascular Medicine
Weijing YunHu Xu
December 5, 2021
Correction

Correction to: Re-examining digoxin bioavailability after half a century: Time for changes in the bioavailability concepts.

Pharmaceutical Research
Athanasios A Tsekouras, Panos Macheras
December 4, 2021
Review
Open Access

Recent and Upcoming Drug Therapies for Pediatric Heart Failure.

Frontiers in Pediatrics
Karla L LossPaul F Kantor
December 3, 2021

Digoxin targets low density lipoprotein receptor-related protein 4 and protects against osteoarthritis.

Annals of the Rheumatic Diseases
Kai-di WangChuan-Ju Liu
December 2, 2021
Review

Therapeutic Effects and Safe Uses of Plant-Derived Polyphenolic Compounds in Cardiovascular Diseases: A Review.

Drug Design, Development and Therapy
Badriyah Shadid AlotaibiGhulam Murtaza
December 2, 2021
Case Report

A successful treatment with intravenous lipid emulsion therapy in a child with verapamil poisoning.

Turkish Journal of Emergency Medicine
Merve HavanTayfun Uçar
December 2, 2021

Effects of ischaemic postconditioning in aortic valve replacement: a multicenter randomized controlled trial.

European Journal of Cardio-thoracic Surgery : Official Journal of the European Association for Cardio-thoracic Surgery
Mari-Liis KaljustoJarle Vaage
December 1, 2021
Open Access

The cellular prion protein interacts with and promotes the activity of Na,K-ATPases.

PloS One
Declan WilliamsGerold Schmitt-Ulms
November 30, 2021

Moringa oleifera: A Tree of Life as a Promising Medicinal Plant for Neurodegenerative Diseases.

Journal of Agricultural and Food Chemistry
Saurav GhimireBhakta Prasad Gaire
November 29, 2021
Preprint
Open Access

Epistasis is not a strong constraint on the recurrent evolution of toxin-resistant Na+,K+-ATPases among tetrapods

BioRxiv : the Preprint Server for Biology
S. MohammadiPeter Andolfatto
November 29, 2021

Hormetic-like dose-response induced by alternagin-C, a protein isolated from urutu snake (Rhinocerophis alternatus) venom, in fish (Hoplias malabaricus) cardiac contractility.

Toxicon : Official Journal of the International Society on Toxinology
Diana Amaral MonteiroFrancisco Tadeu Rantin
November 28, 2021
Review

The Overview on the Pharmacokinetic and Pharmacodynamic Interactions of Triazoles.

Pharmaceutics
Andrzej CzyrskiFranciszek K Główka
November 28, 2021
Review

Phytochemistry, Pharmacology, and Nutraceutical Profile of Carissa Species: An Updated Review.

Molecules : a Journal of Synthetic Chemistry and Natural Product Chemistry
Jyoti DhatwaliaRyszard Amarowicz
November 28, 2021
Open Access

Flavonoids and Omega3 Prevent Muscle and Cardiac Damage in Duchenne Muscular Dystrophy Animal Model.

Cells
Luana TripodiYvan Torrente

Sign up to follow this feed and discover related papers.

Related Feeds

22q11 Deletion Syndrome

22q11.2 deletion syndrome, also known as DiGeorge syndrome, is a congenital disorder caused by a partial deletion of chromosome 22. Symptoms include heart defects, poor immune system function, a cleft palate, complications related to low levels of calcium in the blood, and delayed development. Discover the latest research on this disease here.

Alagille Syndrome

Alagille syndrome is a multi-system genetic disorder that can affect the liver, heart, and other parts of the body. It’s characterization includes heart problems, liver damage, jaundice, and xanthomas. Here is the latest research.

Aneurysm

Aneurysms are outward distensions or bulges that occurs in a weakened wall of blood vessels. Discover the latest research on aneurysms here.

Antianginal Drugs: Mechanisms of Action

Antianginal drugs, including nitrates, beta-blockers, and calcium channel blockers, are used in the treatment of angina pectoris. Here is the latest research on their use and their mechanism of action.

Antiarrhythmic Agents: Mechanisms of Action

Understanding the mechanism of action of antiarrhythmic agents is essential in developing new medications as treatment of cardiac arrhythmias is currently limited by the reduced availability of safe and effective drugs. Discover the latest research on Antiarrhythmic Agents: Mechanism of Action here.

Antihypertensive Agents: Mechanisms of Action

Antihypertensive drugs are used to treat hypertension (high blood pressure) which aims to prevent the complications of high blood pressure, such as stroke and myocardial infarction. Discover the latest research on antihypertensive drugs and their mechanism of action here.

Aortic Aneurysm

An aortic aneurysm is the weakening and bulging of the blood vessel wall in the aorta. This causes dilatation of the aorta, which is usually asymptomatic but carries the risk of rupture and hemorrhage. Find the latest research on aortic aneurysms here.

Aortic Coarctation

Aortic coarctation is a congenital condition characterized by narrowing of the aorta. Discover the latest research on this disease here.

ApoE Phenotypes

Apolipoprotein E (APOE) is a protein involved in fat metabolism and associated with the pathogenesis of Alzheimer's disease and cardiovascular disease. Here is the latest research on APOE phenotypes.

Arrhythmia

Arrhythmias are abnormalities in heart rhythms, which can be either too fast or too slow. They can result from abnormalities of the initiation of an impulse or impulse conduction or a combination of both. Here is the latest research on arrhythmias.

© 2021 Meta ULC. All rights reserved
/feed-previews/inotropic-agents-heart-disease/ac3dcd52-2258-4b2c-b6cc-2ab2f99bae46