Multiple System Atrophy

Multiple System Atrophy is a rare neurodegenerative disorder which affects the body’s autonomic functions, including breathing and muscle control. Discover the latest research on Multiple System Atrophy here.

December 29, 2020

MOBP and HIP1 in multiple system atrophy: new α-synuclein partners in glial cytoplasmic inclusions implicated in the disease pathogenesis

Neuropathology and Applied Neurobiology
Conceição BettencourtJanice L Holton
January 6, 2021

Sighs during sleep in multiple system atrophy.

Sleep Medicine
Sara ParreiraRita Peralta
November 26, 2020
Open Access

Sleep-related symptoms in multiple system atrophy: determinants and impact on disease severity

Chinese Medical Journal
Jun-Yu LinHui-Fang Shang
November 19, 2020

Parkinsonism-associated speech disorders

Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova
N A Skripkina, A P Arefyeva
January 9, 2021

Chemotherapy and Radiation-Associated Cardiac Autonomic Dysfunction

Current Oncology Reports
Alexandra E TengEric H Yang
January 3, 2021
Open Access

Distinct cryo-EM Structure of α-synuclein Filaments derived by Tau

BioRxiv : the Preprint Server for Biology
A. HojjatianKwang Hun Lim
November 28, 2020
Open Access

A distinct neuromelanin magnetic resonance imaging pattern in parkinsonian multiple system atrophy

BMC Neurology
Rita Moiron SimõesSofia Reimão
January 19, 2021

[Clinical and biochemical heterogeneity of Parkinson's disease].

Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova
Kh I KhadzievaA A Plotnikov
November 19, 2020

Palliative care in Parkinson's disease and neurodegenerative diseases

Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova
O S LevinN V Fedorova
December 18, 2020
Open Access

Voxel-Based Meta-Analysis of Gray Matter Abnormalities in Multiple System Atrophy

Frontiers in Aging Neuroscience
Junyu LinHuifang Shang
December 8, 2020
Open Access

REM sleep behavior in Parkinson disease: Frequent, particularly with higher age

PloS One
Heide Baumann-VogelChristian R Baumann
December 2, 2020

Identification and Evaluation of Bisquinoline Scaffold as a New Candidate for α-Synuclein-PET Imaging

ACS Chemical Neuroscience
Sho KaideMasahiro Ono
January 12, 2021

Myoclonus-Ataxia Syndromes: A Diagnostic Approach

Movement Disorders Clinical Practice
Malco RossiBart van de Warrenburg
December 30, 2020
Open Access

Insoluble proteins catch heterologous soluble proteins into inclusion bodies by intermolecular interaction of aggregating peptides.

Jose Vicente CarrataláNeus Ferrer-Miralles
January 23, 2021
Open Access

Cerebrospinal Fluid α-Synuclein Species in Cognitive and Movements Disorders.

Brain Sciences
Vasilios C ConstantinidesElisabeth Kapaki
November 16, 2020

Association of mitochondrial genomic background with risk of Multiple System Atrophy

Parkinsonism & Related Disorders
Rebecca R ValentinoOwen A Ross

Sign up to follow this feed and discover related papers.

Related Feeds

3D Cellular Models of Brain and Neurodegeneration

Brain organoids are three-dimensional in vitro cellular models of the brain that can recapitulate many processes such as the neurodevelopment. In addition, these organoids can be combined with other cell types, such as neurons and astrocytes to study their interactions in assembloids. Disease processes can also be modeled by induced pluripotent stem cell-derived organoids and assembloids from patients with neurodegenerative disorders. Discover the latest research on the models here.


TAR DNA-binding protein 43 (TDP-43) is a pathological protein identified in sporadic Amyotrophic Lateral Sclerosis (ALS) and Frontotemporal Dementia (FTD). Here are the latest discoveries pertaining to TDP-43 and these diseases.

ALS: Genetics

Amyotrophic Lateral Sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.

ALS: Pathogenic Mechanisms

Amyotrophic Lateral Sclerosis is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.

ALS: Phenotypes

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disorder characterized phenotypically by progressive muscle weakness. Clinical phenotypes of ALS can be classified based on the pattern, level, and area of onset (e.g. bulbar, cervical, lumbar). Here is the latest research investigating phenotypes of ALS.

ALS: Prions

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here is the latest research on ALS and prions.

ALS: Stress Granules

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research in this field.

ALS: Therapies

Amyotrophic Lateral Sclerosis (ALS) is associated with the death of neurons that control voluntary muscles. This feed followes the latest research into therapies for this progressive neurodegenerative disease.

Age-related Dementia

Dementias are a group of conditions, including Alzheimer's disease, vascular dementia, and frontotemporal dementia, characterized by deficiencies in cognitive abilities. Age-related dementia refers to dementias that occur in older individuals, usually 60+ years old, in contrast to early-onset dementia. Follow the latest research on age-related dementia here.

Alexander Disease

Alexander disease is a rare leukodystrophy caused by mutations in the astrocyte-specific intermediate filament protein glial fibrillary acidic protein (GFAP). Here is the latest research on this disease.

© 2021 Meta ULC. All rights reserved