P2Y12 & Microglial Self-Renewal

P2Y12 is a G-coupled receptor that is known for its role in platelet activation. However, the expression and involvement in the CNS and microglial activation and renewal is less known. Discover the latest research on P2Y12 and microglial self-renewal here.

August 18, 2020
Preprint
Open Access

MORPHIOUS: A Machine Learning Workflow to Naively Detect the Activation of Microglia and Astrocytes.

BioRxiv : the Preprint Server for Biology
Joseph L Silburt, I. Aubert
April 24, 2020
Preprint
Open Access

Repetitive Transcranial Magnetic Stimulation Activates Glial Cells and Inhibits Neurogenesis after Pneumococcal Meningitis

BioRxiv : the Preprint Server for Biology
M. LukasStephen L Leib
June 18, 2020
Open Access

Glia-Mediated Regenerative Response Following Acute Excitotoxic Damage in the Postnatal Squamate Retina

Frontiers in Cell and Developmental Biology
Julia Eymann, Nicolas Di-Poï
May 28, 2020
Open Access

Tetramethylpyrazine attenuates endotoxin-induced retinal inflammation by inhibiting microglial activation via the TLR4/NF-κB signalling pathway

Biomedicine & Pharmacotherapy = Biomédecine & Pharmacothérapie
Xiaokun HanKeming Yu
April 19, 2020

Mechanisms of brain injury in newborn infants associated with the fetal inflammatory response syndrome

Seminars in Fetal & Neonatal Medicine
Vivien Yap, Jeffrey M Perlman
June 1, 2020

Homeostatic state of microglia in a rat model of chronic sleep restriction

Sleep
Shannon HallKazue Semba
May 31, 2020
Open Access

Brief Electrical Stimulation Triggers an Effective Regeneration of Leech CNS

ENeuro
Sharon CohenOrit Shefi
August 28, 2020
Open Access

Overview of General and Discriminating Markers of Differential Microglia Phenotypes

Frontiers in Cellular Neuroscience
Agnieszka M JurgaKatarzyna Z Kuter
September 2, 2020
Review
Open Access

Cell Therapies under Clinical Trials and Polarized Cell Therapies in Pre-Clinical Studies to Treat Ischemic Stroke and Neurological Diseases: A Literature Review

International Journal of Molecular Sciences
Masahiro HatakeyamaMasato Kanazawa
May 29, 2020

Neuroprotective effect of quercetin nanoparticles: A possible prophylactic and therapeutic role in alzheimer's disease

Journal of Chemical Neuroanatomy
Rehab Ahmed RifaaiNashwa Fathy Gamal El-Tahawy
August 28, 2020
Review
Open Access

Role of Chondroitin Sulfation Following Spinal Cord Injury

Frontiers in Cellular Neuroscience
Rowan K HusseinHerbert M Geller
September 3, 2020
Preprint
Open Access

Microglia-neuron communication at nodes of Ranvier depends on neuronal activity through potassium release and contributes to myelin repair.

BioRxiv : the Preprint Server for Biology
R. RonzanoAnne Desmazieres

Sign up to follow this feed and discover related papers.

Related Feeds

ALS

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS here.

ALS & FTD: TDP-43

ALS shares with a considerable proportion of FTD cases the same neuropathological substrate, namely, inclusions of abnormally phosphorylated protein tdp-43 (ptdp-43). Here are the latest discoveries pertaining to ptdp-43 and these diseases.

ALS - Pathogenic Mechanisms

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.

ALS - Phenotypes

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating phenotypes associated with this genetically heterogeneous disorder.

ALS: Genetics

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.

ALS: Prions

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here are the latest discoveries pertaining to this disease.

ALS: Stress Granules

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research.

ALS: Therapies

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS therapies here.

ALS: Transposon de-silencing

Transposon silencing is a form of transcriptional gene silencing. These gene silencing mechanisms are impaired in Amyotrophic lateral sclerosis (ALS). Here are the latest discoveries pertaining to transposon silencing and this disease.

Age-related Dementia

Age-related dementia (Alzheimer's disease) results from the destructive impact of the pulse on cerebral vasculature. Evidence is reviewed that the neuropathology of the dementia is caused by the breakdown of small cerebral vessels (silent microbleeds), that the microbleeds result from pulse-induced damage to the cerebral vessels, and that pulse becomes increasingly destructive with age, because of the age-related stiffening of the aorta and great arteries, which causes an increase in the intensity of the pressure pulse. Discover the latest research on age-related dementia here.

© 2020 Meta ULC. All rights reserved
/feed-previews/p2y12-microglial-self-renewal/a1c6708a-0e0d-4134-a131-add15c67b192