Parkinson's Disease & Autophagy (MDS)

Autophagy leads to degradation of damaged proteins and organelles by the lysosome. Impaired autophagy has been implicated in several diseases. Here is the role of autophagy in Parkinson’s disease.

November 18, 2020
Review
Open Access

Targets (Metabolic Mediators) of Therapeutic Importance in Pancreatic Ductal Adenocarcinoma

International Journal of Molecular Sciences
Vikrant Rai, Swati Agrawal
November 24, 2020
Review
Open Access

Autophagy and Redox Homeostasis in Parkinson's: A Crucial Balancing Act

Oxidative Medicine and Cellular Longevity
Natalia Jimenez-Moreno, Jon D Lane
November 21, 2020

Daurisoline inhibits hepatocellular carcinoma progression by restraining autophagy and promoting cispaltin-induced cell death

Biochemical and Biophysical Research Communications
Legang Xue, Pei Liu
November 21, 2020
Review
Open Access

A novel autophagy-related lncRNA prognostic risk model for breast cancer

Journal of Cellular and Molecular Medicine
Xiaoying LiYang Li
November 22, 2020
Review
Open Access

The Role of Cholesterol in α-Synuclein and Lewy Body Pathology in GBA1 Parkinson's Disease

Movement Disorders : Official Journal of the Movement Disorder Society
Patricia García-SanzRosario Moratalla
November 22, 2020
Review

The Multifaceted Roles of Nucleophagy in Cancer Development and Therapy

Cell Biology International
Lili ZhaoSurui Sheng
November 17, 2020
Preprint
Open Access

Bioinformatics analysis of autophagy-related lncRNAs in esophageal carcinoma

ResearchSquare
Dan Wujunbai fan
November 18, 2020
Review
Open Access

Current Status and Application of Metformin for Prostate Cancer: A Comprehensive Review

International Journal of Molecular Sciences
Hyun Kyu AhnKyo Chul Koo
November 25, 2020
Review
Open Access

The Anti-Cancer Properties of the HIV Protease Inhibitor Nelfinavir

Cancers
Mahbuba R Subeha, Carlos M Telleria
November 22, 2020
Open Access

Melatonin Can Modulate the Effect of Navitoclax (ABT-737) in HL-60 Cells

Antioxidants
Alexey LomovskyOlga Krestinina
November 24, 2020

Rapamycin inhibits lung squamous cell carcinoma growth by downregulating glypican-3/Wnt/β-catenin signaling and autophagy

Journal of Cancer Research and Clinical Oncology
Yanyu BiKesang Li
November 22, 2020
Open Access

Alternative mitochondrial quality control mediated by extracellular release

Autophagy
Chi-Jing ChoongHideki Mochizuki
November 17, 2020
Review

Mitochondria-ER Tethering in Neurodegenerative Diseases

Cellular and Molecular Neurobiology
Reza Raeisossadati, Merari F R Ferrari
November 18, 2020

Electroacupuncture Improves Motor Symptoms of Parkinson's Disease and Promotes Neuronal Autophagy Activity in Mouse Brain

The American Journal of Chinese Medicine
Wei-Ti HsuShih-Ya Hung
November 22, 2020
Review
Open Access

Potential Anti-Aging Substances Derived from Seaweeds

Marine Drugs
Lei CaoHyeung-Rak Kim

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Alpha-Synuclein Aggregation (MDS)

Alpha-synucleins are small proteins that are believed to restrict the mobility of synpatic vesicles and inhibit neurotransmitter release. Aggregation of these proteins have been linked to several types of neurodegenerative diseases including dementia with Lewy bodies and Parkinson's disease. Here is the latest research on α-synuclein aggregation.

Alpha-Synuclein Antibody Therapeutics (MDS)

Alpha-synuclein is an integral component of Lewy bodies which are comprised of protein clumps and are a pathological hallmark of Parkinson’s disease. They have also been detected in several other neurodegenerative diseases, known as synucleopathies. Here is the latest research on alpha-synuclein antibody therapeutics.

Astrocytes & Huntington’s Disease (MDS)

Astrocytes are abundant within the central nervous system and their dysfunction has been thought to be an important contributor to some neurodegenerative diseases, in particular Huntington’s disease. Damage to these cells may make neurons more susceptible to degeneration. Here is the latest research on astrocytes and Huntington’s disease.

Ataxia telangiectasia (MDS)

Ataxia telangiectasia is a rare neurodegenerative diseases caused by defects in the ATM gene, which is involved in DNA damage recognition and repair pathways. Here is the latest research on this autosomal recessive disease.

Ataxias (MDS)

Ataxia is a neurological condition characterized by lack of voluntary coordination of muscle movements including loss of coordination, balance, and speech. Discover the latest research on ataxia here.

Atypical Parkinsonism (MDS)

Atypical Parkinsonism presents with the same signs and symptoms of Parkinson's disease, but do not respond to typical Parkionson's disease treatment with levodopa. Atypical Parkinsonism is thought to be associated with abnormal protein buildup within brain cells. Here is the latest on Atypical Parkinsonism.

Basal ganglia in Parkinson's disease (MDS)

The basal ganglia is comprised of the neostriatum, the external and internal pallidal segments, the subthalamic nucleus, the substantia nigra pars reticulata, and the pars compacta of the substantia nigra. The basal ganglia circuitry is responsible for the correct execution of voluntary movements and is implicated in Parkinson's disease. Here is the latest research investigating the basal ganglia in Parkinson's disease.

Bovine Spongiform Encephalopathy (MDS)

Bovine spongiform encephalopathy is a neurodegenerative disease belonging to the transmissible spongiform encephalopathies, a group of diseases including sheep scrapie and human Creutzfeldt-Jakob disease. Here is the latest research.

Brain Imaging of Neural Circuits (MDS)

Neural circuits are groups of interconnected neurons which carry out specific functions when activated. Imaging these neural circuits allows researches to further elucidate their mechanisms and functions. Follow this feed to stay up to date on brain imaging of neural circuits.

Cerebral Palsy & Spasticity (MDS)

Spactic cereberal palsy is one of the most common types of cerebral palsy that stems from an upper motor neuron lesion in the brain. Discover the latest research on cerebral palsy and spasticity here.

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