Feed Preview

Fast Growing

Parkinson's Disease: Biomarkers

Parkinson's Disease: Biomarkers diagram by Santiago et al, PLOS One
Santiago et al, PLOS One

Parkinson’s disease is a progressive neurodegenerative disorder that affects movement. This feed focuses on genomic and drug-response biomarkers in Parkinson’s disease.

Top 20 most recent papers
Movement Disorders Clinical Practice

Nosology and Phenomenology of Psychosis in Movement Disorders

Movement Disorders Clinical PracticeFebruary 20, 2020
Malco RossiMarcelo Merello
bioRxiv

Protein-protein interactions in neurodegenerative diseases: a conspiracy theory.

bioRxivFebruary 11, 2020
Travis B ThompsonAlain Goriely
11
Europace : European Pacing, Arrhythmias, and Cardiac Electrophysiology : Journal of the Working Groups on Cardiac Pacing, Arrhythmias, and Cardiac Cellular Electrophysiology of the European Society of Cardiology

Diagnostic yield of hypertrophic cardiomyopathy in first-degree relatives of decedents with idiopathic left ventricular hypertrophy

Europace : European Pacing, Arrhythmias, and Cardiac Electrophysiology : Journal of the Working Groups on Cardiac Pacing, Arrhythmias, and Cardiac Cellular Electrophysiology of the European Society of CardiologyFebruary 5, 2020
Gherardo FinocchiaroSanjay Sharma
81
Current Pharmaceutical Design

Current Therapeutic Strategies and Perspectives for Neuroprotection in Parkinson's Disease

Current Pharmaceutical DesignFebruary 18, 2020
Mohan Krishna Ghanta Bhaskar L V K S
Parkinsonism & Related Disorders

A comparison of auditory and vestibular dysfunction in Parkinson's disease and Multiple System Atrophy

Parkinsonism & Related DisordersFebruary 8, 2020
Alfonso ScarpaMaria Teresa Pellecchia
1
Frontiers in Neuroscience

A Role of Cholesterol in Modulating the Binding of α-Synuclein to Synaptic-Like Vesicles

Frontiers in NeuroscienceFebruary 18, 2020
Wing K ManGiuliana Fusco
4
Movement Disorders : Official Journal of the Movement Disorder Society

Prodromal Dementia with Lewy Bodies: Clinical Characterization and Predictors of Progression

Movement Disorders : Official Journal of the Movement Disorder SocietyFebruary 13, 2020
Marleen van de BeekAfina W Lemstra
12
1
Journal of the American Heart Association

Role of DJ-1 in Modulating Glycative Stress in Heart Failure

Journal of the American Heart AssociationFebruary 19, 2020
Yuuki ShimizuJohn W Calvert
1
Parkinsonism & Related Disorders

Gastrointestinal symptoms are predictive of trajectories of cognitive functioning in de novo Parkinson's disease

Parkinsonism & Related DisordersFebruary 15, 2020
Jacob D JonesJonathan P Jacobs
20
2
Acta Neuropathologica Communications

Early-onset impairment of the ubiquitin-proteasome system in dopaminergic neurons caused by α-synuclein

Acta Neuropathologica CommunicationsFebruary 16, 2020
Chris McKinnonSuneil K Kalia
7
Redox Biology

Redox-dependent regulation of mitochondrial dynamics by DJ-1 paralogs in Saccharomyces cerevisiae

Redox BiologyFebruary 20, 2020
Kondalarao BankapalliPatrick D'Silva
Frontiers in Neuroscience

Alpha-Synuclein Physiology and Pathology: A Perspective on Cellular Structures and Organelles

Frontiers in NeuroscienceFebruary 11, 2020
Luis D Bernal-CondeMagdalena Guerra-Crespo
11
1
Parkinsonism & Related Disorders

Reply to J. Dulski and J. Slawek's "Fibrodysplasia ossificans progressiva as a form of pseudodystonia"

Parkinsonism & Related DisordersFebruary 7, 2020
Rok BerlotMaja Kojović
2
Journal of Medical Genetics

Excess of singleton loss-of-function variants in Parkinson's disease contributes to genetic risk

Journal of Medical GeneticsFebruary 15, 2020
Dheeraj Reddy BobbiliPatrick May
6

See more papers from this feed

Related Feeds

ALS

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS here.

ALS & FTD: TDP-43

ALS shares with a considerable proportion of FTD cases the same neuropathological substrate, namely, inclusions of abnormally phosphorylated protein tdp-43 (ptdp-43). Here are the latest discoveries pertaining to ptdp-43 and these diseases.

ALS - Pathogenic Mechanisms

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.

ALS - Phenotypes

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating phenotypes associated with this genetically heterogeneous disorder.

ALS: Genetics

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.

ALS: Prions

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here are the latest discoveries pertaining to this disease.

ALS: Stress Granules

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research.

ALS: Therapies

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS therapies here.

ALS: Transposon de-silencing

Transposon silencing is a form of transcriptional gene silencing. These gene silencing mechanisms are impaired in Amyotrophic lateral sclerosis (ALS). Here are the latest discoveries pertaining to transposon silencing and this disease.

Age-related Dementia

Age-related dementia (Alzheimer's disease) results from the destructive impact of the pulse on cerebral vasculature. Evidence is reviewed that the neuropathology of the dementia is caused by the breakdown of small cerebral vessels (silent microbleeds), that the microbleeds result from pulse-induced damage to the cerebral vessels, and that pulse becomes increasingly destructive with age, because of the age-related stiffening of the aorta and great arteries, which causes an increase in the intensity of the pressure pulse. Discover the latest research on age-related dementia here.

© 2020 Meta ULC. All rights reserved
/feed-previews/parkinsons-disease-biomarkers/28ca42d2-e7b5-4c4f-9932-3384c44e101f