Prion Neurodegeneration

Prion diseases are fatal, transmissible, neurodegenerative diseases caused by the misfolding of the prion protein (prp). Here is the latest research on prion diseases.

January 16, 2021
Review

FUS and TDP-43 Phases in Health and Disease

Trends in Biochemical Sciences
Bede PortzJames Shorter
January 8, 2021

2,3,5,6-Tetramethylpyrazine protects retinal photoreceptors against endoplasmic reticulum stress by modulating ATF4-mediated inhibition of PRP aggregation

Journal of Molecular Medicine : Official Organ of the Gesellschaft Deutscher Naturforscher Und Ärzte
Hao HuangHuangxuan Shen
December 31, 2020
Open Access

Regional Differences in Neuroinflammation-Associated Gene Expression in the Brain of Sporadic Creutzfeldt-Jakob Disease Patients

International Journal of Molecular Sciences
Aušrinė AreškevičiūtėEva L Lund
January 15, 2021
Open Access

Structural and molecular basis of cross-seeding barriers in amyloids

Proceedings of the National Academy of Sciences of the United States of America
Asen DaskalovAntoine Loquet
December 23, 2020
Review
Open Access

Exposure Risk of Chronic Wasting Disease in Humans

Viruses
Satish K NemaniValerie L Sim
December 22, 2020

Familial Prion Disease: First Indian Kindred with Gerstmann-Sträussler-Scheinker Syndrome

Neurology India
Sameer BhatiaMandaville Gourie-Devi
December 22, 2020

Aβ Plaques

Free Neuropathology
Lary C Walker
January 21, 2021
Review
Open Access

From Cell Culture to Organoids-Model Systems for Investigating Prion Strain Characteristics.

Biomolecules
Hailey Pineau, Valerie L Sim
January 19, 2021
Review
Open Access

Heterotypic interactions in amyloid function and disease

The FEBS Journal
Katerina KonstantouleaJoost Schymkowitz
January 4, 2021
Preprint
Open Access

The engineered peptide construct NCAM1-Aβ inhibits aggregation of the human prion protein (PrP)

BioRxiv : the Preprint Server for Biology
M. GielnikSebastian K.T.S. Warmlander
January 6, 2021
Preprint
Open Access

Complete microglia deficiency accelerates prion disease without enhancing CNS prion accumulation

BioRxiv : the Preprint Server for Biology
B. M. BradfordNeil A Mabbott
January 22, 2021
Review

Retromer dysfunction at the nexus of tauopathies.

Cell Death and Differentiation
Julian M CarosiTimothy J Sargeant
December 18, 2020
Open Access

Amyotrophic Lateral Sclerosis: Proteins, Proteostasis, Prions, and Promises

Frontiers in Cellular Neuroscience
Luke McAlaryNeil R Cashman
January 18, 2021

Inflammation and Parkinson's disease pathogenesis: Mechanisms and therapeutic insight

Progress in Molecular Biology and Translational Science
Gianluigi ForloniClaudia Balducci
January 12, 2021
Review

Myoclonus-Ataxia Syndromes: A Diagnostic Approach

Movement Disorders Clinical Practice
Malco RossiBart van de Warrenburg
January 22, 2021

Advances in genetics research in the pathogenesis of amyotrophic lateral sclerosis.

Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences
Yan WangFangfang Bi
December 30, 2020
Preprint
Open Access

Insoluble proteins catch heterologous soluble proteins into inclusion bodies by intermolecular interaction of aggregating peptides.

ResearchSquare
Jose Vicente CarrataláNeus Ferrer-Miralles

Sign up to follow this feed and discover related papers.

Related Feeds

ALS: Prions

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here is the latest research on ALS and prions.

Age-related Dementia

Dementias are a group of conditions, including Alzheimer's disease, vascular dementia, and frontotemporal dementia, characterized by deficiencies in cognitive abilities. Age-related dementia refers to dementias that occur in older individuals, usually 60+ years old, in contrast to early-onset dementia. Follow the latest research on age-related dementia here.

Alzheimer's Disease: APOE

Apolipoprotein E (APOE) polymorphic alleles are major genetic risk factors for Alzheimer's disease. Discover the latest research on APOE and other genetic determinants of Alzheimer's disease here.

Alzheimer's Disease: APP

Amyloid precursor protein (APP) proteolysis is critical for the development of Alzheimer's disease, a neurodegenerative disease associated with accumulation of amyloid plaques in the brain. Here is the latest research on APP and Alzheimer's disease.

Alzheimer's Disease: Amyloid Beta

Alzheimer's disease is a neurodegenerative disease associated with the accumulation of amyloid plaques in the brain; these plaques are comprised of amyloid beta deposits. Here is the latest research in this field.

Alzheimer's Disease: Animal Models

Alzheimer's disease is a neurodegenerative disease which can be studied using various experimental systems. This feed focuses on animal models used for Alzheimer's disease research.

Alzheimer's Disease: Early Markers

Alzheimer's disease (AD) is a neurodegenerative disease characterized by progressive cognitive and behavioral decline. Targeting markers in the earliest stages of the disease may mitigate the progression of AD. This feed focuses on early diagnosis and markers, as well as environmental, pharmacological, and drug-response biomarkers associated with this disease.

Alzheimer's Disease: Endosomes

Dysfunctional endosomal trafficking may be associated with Alzheimer’s disease (AD) pathology. Targeting the endosome may advance treatment options for AD. Here is the latest research on endosomes and AD.

Alzheimer's Disease: Genetics

Alzheimer's disease is a neurodegenerative disease. Discover genetic and epigenetic aspects of Alzheimer’s disease, including genetic markers and genomic structural variations with this feed.

Alzheimer's Disease: MS4A

Variants within the membrane-spanning 4-domains subfamily A (MS4A) gene cluster have recently been implicated in Alzheimer's disease in genome-wide association studies. Here is the latest research on Alzheimer's disease and MS4A.

© 2021 Meta ULC. All rights reserved
/feed-previews/prion-neurodegeneration/94e00538-6a65-418e-a7a2-7aeb6d5a3f0b