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ROS: Metabolism & Signaling
StudioMolekuul, ShutterstockReactive oxygen species (ROS) are reactive chemicals containing oxygen. They generated as a byproduct of oxidative energy metabolism and play a role in regulating several intracellular signaling pathways. Here is the latest research on ROS metabolism and signaling.
ROS: Trichothecenes' handy weapon?
Teriflunomide Does Not Change Dynamics of Nadph Oxidase Activation and Neuronal Dysfunction During Neuroinflammation
CTRP9 Mediates Protective Effects in Cardiomyocytes via AMPK- and Adiponectin Receptor-Mediated Induction of Anti-Oxidant Response
Transcriptome analysis on responses of orchardgrass (Dactylis glomerata L.) leaves to a short term flooding
Putting the brakes on tumorigenesis with snake venom toxins: New molecular insights for cancer drug discovery
The Early Effects of Uninephrectomy on Rat Kidney Metabolic State Using Optical Imaging
Insoluble tau aggregates induce neuronal death through modification of membrane ion conductance, activation of voltage-gated calcium channels and NADPH oxidase
Immuno-metabolic impact of the multiple sclerosis patients' sera on endothelial cells of the blood-brain barrier
Hepatokine selenoprotein P-mediated reductive stress causes resistance to intracellular signal transduction
Luteolin inhibits melanoma growth in vitro and in vivo via regulating ECM and oncogenic pathways but not ROS
miRNAs, oxidative stress, and cancer: A comprehensive and updated review
Bisoprolol, a β1 antagonist, protects myocardial cells from ischemia reperfusion injury via PI3K/AKT/GSK3β pathway
NADPH Oxidases in Bone and Cartilage Homeostasis and Disease: A Promising Therapeutic Target
Redox signaling in the pathogenesis of human disease and the regulatory role of autophagy
Total antioxidant capacity-Relevance, methods and clinical implications
APPA (apocynin and paeonol) modulates pathological aspects of human neutrophil function, without supressing antimicrobial ability, and inhibits TNFα expression and signalling
Deactivation of mitochondrial NADH:ubiquinone oxidoreductase (respiratory complex I): Extrinsically affecting factors
Epidermal Growth Factor (EGF) Augments the Invasive Potential of Human Glioblastoma Multiforme Cells via the Activation of Collaborative EGFR/ROS-Dependent Signaling
Reactive oxygen species in male reproduction: A boon or a bane?
Seminal oxidation-reduction potential levels are not influenced by the presence of leucocytospermia
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