Spinocerebellar Ataxia: miRNAs

microRNAs (miRNAs) are key regulatory RNAs that control modulate gene expression in various biological processes and human diseases such as Spinocerebellar Ataxia. Here are the latest discoveries pertaining to miRNAs and this disease.

June 19, 2019
Open Access

Prediction of Survival With Long-Term Disease Progression in Most Common Spinocerebellar Ataxia

Movement Disorders : Official Journal of the Movement Disorder Society
Alhassane DialloSophie Tezenas du Montcel
October 2, 2019

A neuroscientific approach to increase gender equality

Nature Human Behaviour
Christiane SchreiweisVioletta Zujovic
January 3, 2019
Open Access

A new MRI marker of ataxia with oculomotor apraxia

European Journal of Radiology
Solène RonsinCaroline Tilikete
December 16, 2019
Review

Neurochemical profiles in hereditary ataxias: A meta-analysis of Magnetic Resonance Spectroscopy studies

Neuroscience and Biobehavioral Reviews
Janna KraheSandro Romanzetti
August 14, 2019

Association of CAG Repeats With Long-term Progression in Huntington Disease

JAMA Neurology
Douglas R LangbehnTRACK-HD and Track-On HD Groups
January 10, 2019

Cell Death Mechanisms in a Mouse Model of Retinal Degeneration in Spinocerebellar Ataxia 7

Neuroscience
Cecile LebonAlicia Torriglia
August 27, 2019

Oral mobility reflects rate of progression in advanced Friedreich's ataxia

Annals of Clinical and Translational Neurology
Stéphanie BorelAlexandra Durr
November 21, 2018

Altered Intracortical T1 -Weighted/T2 -Weighted Ratio Signal in Huntington's Disease

Frontiers in Neuroscience
Christopher D RowleyNicholas A Bock
February 19, 2019

Contemporary Dance Practice Improves Motor Function and Body Representation in Huntington's Disease: A Pilot Study

Journal of Huntington's Disease
Iris TrinklerAlexandra Durr
November 10, 2018

Dentatorubral-pallidoluysian Atrophy: An Update

Tremor and Other Hyperkinetic Movements
Liam S CarrollKathryn J Peall
February 18, 2020

Apathy predicts rate of cognitive decline over 24 months in premanifest Huntington's disease

Psychological Medicine
S C AndrewsTRACK-HD Investigators
November 26, 2019

Accelerated 129 Xe MRI morphometry of terminal airspace enlargement: Feasibility in volunteers and those with alpha-1 antitrypsin deficiency

Magnetic Resonance in Medicine : Official Journal of the Society of Magnetic Resonance in Medicine
Alexei OuriadovGrace Parraga

Sign up to follow this feed and discover related papers.

Related Feeds

ALS

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS here.

ALS & FTD: TDP-43

ALS shares with a considerable proportion of FTD cases the same neuropathological substrate, namely, inclusions of abnormally phosphorylated protein tdp-43 (ptdp-43). Here are the latest discoveries pertaining to ptdp-43 and these diseases.

ALS - Pathogenic Mechanisms

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.

ALS - Phenotypes

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating phenotypes associated with this genetically heterogeneous disorder.

ALS: Genetics

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.

ALS: Prions

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here are the latest discoveries pertaining to this disease.

ALS: Stress Granules

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research.

ALS: Therapies

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS therapies here.

ALS: Transposon de-silencing

Transposon silencing is a form of transcriptional gene silencing. These gene silencing mechanisms are impaired in Amyotrophic lateral sclerosis (ALS). Here are the latest discoveries pertaining to transposon silencing and this disease.

Age-related Dementia

Age-related dementia (Alzheimer's disease) results from the destructive impact of the pulse on cerebral vasculature. Evidence is reviewed that the neuropathology of the dementia is caused by the breakdown of small cerebral vessels (silent microbleeds), that the microbleeds result from pulse-induced damage to the cerebral vessels, and that pulse becomes increasingly destructive with age, because of the age-related stiffening of the aorta and great arteries, which causes an increase in the intensity of the pressure pulse. Discover the latest research on age-related dementia here.

© 2020 Meta ULC. All rights reserved
/feed-previews/spinocerebellar-ataxia-mirnas/c3e674fa-59c9-4c12-bf09-160beb819f9e