Synucleinopathies

Synucleinopathies are neurodegenerative diseases characterized by accumulation of alpha-synuclein protein aggregates in neurons and glial cells. Discover the latest research on synucleinopathies here.

June 25, 2020
Open Access

Parkinson's disease: etiopathogenesis and treatment

Journal of Neurology, Neurosurgery, and Psychiatry
Joseph Jankovic, Eng King Tan
July 1, 2020
Open Access

Appendectomy, Tonsillectomy and Parkinson's Disease Risk: A Swedish Register-Based Study

Frontiers in Neurology
Bojing LiuKarin Wirdefeldt
July 2, 2020

Enhancing the Activity of Glucocerebrosidase as a Treatment for Parkinson Disease

CNS Drugs
Elisa Menozzi, Anthony H V Schapira
June 26, 2020
Open Access

Translation Imaging in Parkinson's Disease: Focus on Neuroinflammation

Frontiers in Aging Neuroscience
Sara BelloliMaria Carla Gilardi
July 1, 2020
Open Access

Characterization by Nano-Infrared Spectroscopy of Individual Aggregated Species of Amyloid Proteins

Molecules : a Journal of Synthetic Chemistry and Natural Product Chemistry
Jehan WaeytensVincent Raussens
July 1, 2020
Review

REM sleep behavior disorder (RBD)

Neurobiology of Disease
Michele T M Hu
June 28, 2020

Kinetic and protective role of autophagy in manganese-exposed BV-2 cells

Biochimica Et Biophysica Acta. Molecular Cell Research
Soledad Porte AlconMónica Lidia Kotler
July 1, 2020
Comment / Editorial

Small-molecule inhibitors of α-synuclein for the treatment of Parkinson's disease: a patent landscape

Pharmaceutical Patent Analyst
Jacques Joubert
July 2, 2020

Parkinsonian and Cerebellar Phenotypes of Probable MSA: An Insight in to Prognostic Factors Based on Autonomic Functions

Annals of Indian Academy of Neurology
Malligurki Raghurama RukmaniTalakad N Sathyaprabha
July 1, 2020

Monoamine oxidase A inhibition as monotherapy reverses parkinsonism in the MPTP-lesioned marmoset

Naunyn-Schmiedeberg's Archives of Pharmacology
Adjia HamadjidaPhilippe Huot
June 24, 2020

Amyloid beta, tau, synaptic, neurodegeneration, and glial biomarkers in the preclinical stage of the Alzheimer's continuum

Alzheimer's & Dementia : the Journal of the Alzheimer's Association
Marta Milà-AlomàALFA study
July 2, 2020
Review

Vesicle trafficking and lipid metabolism in synucleinopathy

Acta Neuropathologica
Saranna FanningUlf Dettmer
June 27, 2020
Case Report
Open Access

Parkinsonian Syndrome with Frontal Lobe Involvement and Anti-Glycine Receptor Antibodies

Brain Sciences
Dominique EndresLudger Tebartz van Elst
June 25, 2020

Delta Oscillations Are a Robust Biomarker of Dopamine Depletion Severity and Motor Dysfunction in Awake Mice

Journal of Neurophysiology
Timothy C WhalenAryn H Gittis

Sign up to follow this feed and discover related papers.

Related Feeds

ALS

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS here.

ALS & FTD: TDP-43

ALS shares with a considerable proportion of FTD cases the same neuropathological substrate, namely, inclusions of abnormally phosphorylated protein tdp-43 (ptdp-43). Here are the latest discoveries pertaining to ptdp-43 and these diseases.

ALS - Pathogenic Mechanisms

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating pathogenic mechanisms that underlie this genetically heterogeneous disorder.

ALS - Phenotypes

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. Here is the latest research investigating phenotypes associated with this genetically heterogeneous disorder.

ALS: Genetics

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder characterized by muscle weakness. ALS is a genetically heterogeneous disorder with several causative genes. Here are the latest discoveries pertaining to the genetics of this disease.

ALS: Prions

Prions are misfolded proteins which characterize several fatal neurodegenerative diseases. Prion-like mechanisms are associated with the pathogenesis of Amyotrophic Lateral Sclerosis (ALS). Here are the latest discoveries pertaining to this disease.

ALS: Stress Granules

Amyotrophic Lateral Sclerosis (ALS) is a neurodegenerative disease characterized by cytoplasmic protein aggregates within motor neurons. TDP-43 is an ALS-linked protein that is known to regulate splicing and storage of specific mRNAs into stress granules, which have been implicated in formation of ALS protein aggregates. Here is the latest research.

ALS: Therapies

Amyotrophic Lateral Sclerosis (ALS), also known as motor neuron disease, is associated with the death of neurons that control voluntary muscles. Discover the latest research on ALS therapies here.

ALS: Transposon de-silencing

Transposon silencing is a form of transcriptional gene silencing. These gene silencing mechanisms are impaired in Amyotrophic lateral sclerosis (ALS). Here are the latest discoveries pertaining to transposon silencing and this disease.

Age-related Dementia

Age-related dementia (Alzheimer's disease) results from the destructive impact of the pulse on cerebral vasculature. Evidence is reviewed that the neuropathology of the dementia is caused by the breakdown of small cerebral vessels (silent microbleeds), that the microbleeds result from pulse-induced damage to the cerebral vessels, and that pulse becomes increasingly destructive with age, because of the age-related stiffening of the aorta and great arteries, which causes an increase in the intensity of the pressure pulse. Discover the latest research on age-related dementia here.

© 2020 Meta ULC. All rights reserved
/feed-previews/synucleinopathies/076a22d3-bacb-4253-8763-8b9001de2c68