Tauopathy Biochemistry

Tauopathies are neurodegenerative disorders that are caused due to the accumulation of abnormal tau proteins in neurons and/or the glia. Here is the latest research on tauopathy biochemistry.

December 6, 2021

With No Lysine Kinase 1 Promotes Metabolic Derangements and RV Dysfunction in Pulmonary Arterial Hypertension.

JACC. Basic to Translational Science
Sasha Z PriscoKurt W Prins
December 6, 2021
Open Access

Recent High-Resolution Structures of Amyloids Involved in Neurodegenerative Diseases.

Frontiers in Aging Neuroscience
Rodrigo Diaz-Espinoza
December 6, 2021
Open Access

Exploration of Aberrant E3 Ligases Implicated in Alzheimer's Disease and Development of Chemical Tools to Modulate Their Function.

Frontiers in Cellular Neuroscience
Frances M Potjewyd, Alison D Axtman
December 6, 2021

Prions and Neurodegenerative Diseases: A Focus on Alzheimer's Disease.

Journal of Alzheimer's Disease : JAD
Alessio CrestiniVincenzo Mattei
December 5, 2021

β-arrestin1 promotes tauopathy by transducing GPCR signaling, disrupting microtubules and autophagy.

Life Science Alliance
Jung-Aa WooDavid E Kang
December 4, 2021
Open Access

NMR Studies of Tau Protein in Tauopathies.

Frontiers in Molecular Biosciences
Kristine KitokaKristaps Jaudzems
December 4, 2021
Open Access

Human Induced Pluripotent Stem Cell Models of Frontotemporal Dementia With Tau Pathology.

Frontiers in Cell and Developmental Biology
Rebekka KühnGunnar Hargus
December 3, 2021

Microtubule-modulating agents in the fight against neurodegeneration: Will it ever work?

Current Neuropharmacology
Ahmed SolimanRoland Brandt
December 3, 2021

Tau phosphorylation sites serine202 and serine396 are differently altered in chronic traumatic encephalopathy and Alzheimer's disease.

Alzheimer's & Dementia : the Journal of the Alzheimer's Association
SpiroAnthony StathasThor D Stein
December 3, 2021

Hydromethylthionine enhancement of central cholinergic signalling is blocked by rivastigmine and memantine.

Journal of Neurochemistry
Constantin KondakJochen Klein
December 3, 2021

The metabolic signaling of the nucleoredoxin-like 2 gene supports brain function.

Redox Biology
Céline JaillardThierry Léveillard
November 29, 2021

Post-translational modifications within tau paired helical filament nucleating motifs perturb microtubule interactions and oligomer formation.

The Journal of Biological Chemistry
Diana M AcostaDavid Eliezer
November 28, 2021

Examining the Toxicity of α-Synuclein in Neurodegenerative Disorders.

Frank Y ShanJason H Huang

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