PMID: 7028312Jul 1, 1981Paper

A double-blind trial of the H2 receptor antagonist cimetidine, and the H1 receptor antagonist promethazine hydrochloride in the treatment of atopic dermatitis

Clinical Allergy
I S Foulds, R M MacKie

Abstract

Twenty young adults with atopic dermatitis were allocated randomly in a clinical trial to compare the action of the H2 receptor antagonist cimetidine (Tagamet) and the H1 receptor antagonist promethazine hydrochloride (Phenergan), singly and in combination. No significant differences were found, clinically or in the laboratory investigations, between the patients on either drug alone or the combination of the two.

References

Sep 1, 1978·Clinical Allergy·P PhanuphakP F Kohler
Aug 1, 1977·The British Journal of Dermatology·M GreavesI Robertson
Dec 1, 1978·The British Journal of Dermatology·C A Commens, M W Greaves
Mar 1, 1979·Clinical and Experimental Dermatology·R Corbett, M W Greaves
Jul 1, 1979·The British Journal of Dermatology·C N MatthewsF Storari
Dec 1, 1979·The British Journal of Dermatology·M G Cook, H A Ridgway
Feb 1, 1980·The British Journal of Dermatology·R Marks
Feb 1, 1980·Annals of Internal Medicine·J L JorizzoA J Olansky

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Citations

Jan 1, 1984·Archives of Dermatological Research·P J FroschE Macher
Jan 6, 2011·Clinical Reviews in Allergy & Immunology·Ulf DarsowSonja Ständer
Nov 1, 1990·Clinical and Experimental Allergy : Journal of the British Society for Allergy and Clinical Immunology·H Behrendt, J Ring
Dec 30, 2015·Journal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG·Thomas WerfelUNKNOWN Association of Scientific Medical Societies of Germany
Mar 2, 2013·The Cochrane Database of Systematic Reviews·Christian J ApfelbacherElke Weisshaar
Jan 23, 2019·The Cochrane Database of Systematic Reviews·Uwe MatterneChristian J Apfelbacher
Aug 23, 2016·The British Journal of Dermatology·H NankervisH C Williams

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Atopic dermatitis is a chronic inflammatory genetically determined disease of the skin marked by increased ability to form reagin (IgE), with increased susceptibility to allergic rhinitis and asthma, and hereditary disposition to a lowered threshold for pruritus. Discover the latest research on atopic dermatitis here.

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