Acute on chronic bronchitis: A model of mucosal immunology

Immunology and Cell Biology
R ClancyA W Cripps

Abstract

Acute bronchitis has been studied as a model of disturbed mucosal immunoregulation. A new hypothesis relating to the pathogenesis of acute bronchitis has been developed, based on altered host response as the prime mover. Infection-prone subjects had low levels of lysozyme. Effective oral immunization, especially if early, reduced levels of bacterial colonization. Future attention focuses on intra bronchial inflammation and its link to the host-parasite relationship.

References

Mar 1, 1978·Clinics in Endocrinology and Metabolism·R Volpé
Mar 13, 1975·The New England Journal of Medicine·I Tager, F E Speizer
Jun 1, 1992·The Journal of Infectious Diseases·R L Clancy, A W Cripps
Aug 1, 1991·Australian and New Zealand Journal of Medicine·M K Tandon, V Gebski
Oct 1, 1990·Immunology and Cell Biology·D C TaylorR L Clancy
Feb 1, 1990·Australian and New Zealand Journal of Medicine·H L ButtM J Hensley

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Citations

May 24, 2000·Immunopharmacology·M A Jagels, T E Hugli
Mar 21, 2001·Vaccine·S ElahiG Pang
Jan 12, 2008·Journal of the American College of Nutrition·Robert L Clancy, Gerald Pang
Aug 26, 1999·American Journal of Respiratory Cell and Molecular Biology·M A JagelsT E Hugli
Mar 1, 2012·Internal Medicine Journal·R L Clancy
Apr 23, 2010·Clinical and Experimental Immunology·R L Clancy, M L Dunkley
Aug 6, 2003·FEMS Immunology and Medical Microbiology·Robert Clancy
Jun 1, 1997·Infection and Immunity·E M PorterT Ganz

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