Anaemia in rheumatoid arthritis: role of cytokines.

Anemia develops when your blood lacks enough healthy red blood cells. Anemia of inflammation (AI, also called anemia of chronic disease) is a common, typically normocytic, normochromic anemia that is caused by an underlying inflammatory disease. Here is the latest research on anemia.

Anaemia in rheumatoid arthritis: role of cytokines.

Abstract

References

A human inhibitor of tumor necrosis factor alpha

Interleukin 1 regulates hematopoietic activity, a role previously ascribed to hemopoietin 1.

Specific interleukin-1 inhibitor in serum and urine of children with systemic juvenile chronic arthritis.

Effects of recombinant tumor necrosis factor on proliferation and differentiation of leukemic and normal hemopoietic cells in vitro. Relationship to cell surface receptor.

Regulation of very primitive, multipotent, hemopoietic cells by hemopoietin-1

Mechanism of anaemia in rheumatoid arthritis: demonstration of raised interleukin 1 beta concentrations in anaemic patients and of interleukin 1 mediated suppression of normal erythropoiesis and proliferation of human erythroleukaemia (HEL) cells in vitro

Cachectin/tumor necrosis factor induces cachexia, anemia, and inflammation.

Synergy of interleukin 1 and granulocyte colony-stimulating factor: in vivo stimulation of stem-cell recovery and hematopoietic regeneration following 5-fluorouracil treatment of mice

Interleukin 1 and the pathogenesis of inflammatory diseases

Tumour necrosis factor--an overview

Anaemia in juvenile chronic arthritis: serum inhibition of normal erythropoiesis in vitro

Natural killer (NK) cell-derived hematopoietic colony-inhibiting activity and NK cytotoxic factor. Relationship with tumor necrosis factor and synergism with immune interferon

Interleukin-1

The mechanism of the anaemia in rheumatoid arthritis: effects of bone marrow adherent cells and of serum on in-vitro erythropoiesis

In vitro suppression of erythropoiesis by bone marrow adherent cells from some patients with fungal infection

Humoral suppression of erythropoiesis in systemic lupus erythematosus (SLE) and rheumatoid arthritis

Citations

No detection of macrophage erythropoietin production in bone marrow from rheumatoid arthritis patients with and without anaemia and controls

Correlation of serum cytokine levels with haematological abnormalities in human immunodeficiency virus infection.

The role of tumour necrosis factor-alpha in acquired immunodeficiency syndrome

The role of channel formation in the mechanism of action of tumor necrosis factors

Hemoglobin, erythropoietin and systemic inflammation in exacerbations of chronic obstructive pulmonary disease

Prevalence of anemia in chronic obstructive pulmonary disease: comparison to other chronic diseases

Iron repletion as a contribution to erythropoietin treatment of anemia in rheumatoid arthritis

Prediction of iron deficiency in chronic inflammatory rheumatic disease anaemia

Tumor necrosis factor alpha, its soluble receptor I, and -308 gene promoter polymorphism in patients with rheumatoid arthritis with or without amyloidosis: implications for the pathogenesis of nephropathy and anemia of chronic disease in reactive amyloidosis

Tumor necrosis factor alpha is associated with disease activity and the degree of anemia in patients with rheumatoid arthritis

Pathogenesis of the anemia of chronic disease: a cytokine-mediated anemia

Stimulation of tumour necrosis factor-alpha production by recombinant human erythropoietin may contribute to failure of therapy

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