PMID: 6412966Sep 5, 1983Paper
Antianaphylactic effect of naloxone in mice is mediated by increased central sympathetic outflow to sympathetic nerve endings and adrenal medulla
Brain Research
S Amir
Abstract
Intravenous naloxone, 1 or 10 mg/kg, protects sensitized mice from lethal anaphylaxis. The protective effect is reversed by pretreatment with the ganglionic blocker, chlorisondamine chloride, peripheral chemical sympathectomy with 6-hydroxydopamine or bilateral adrenal gland denervation. The possible involvement of the sympathetic nervous system in naloxone's antianaphylactic action, suggested by these findings, is discussed.
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Citations
Mar 11, 1985·Brain Research·S Amir, J M van Ree
Nov 11, 1983·European Journal of Pharmacology·S Amir
Mar 1, 1984·Pharmacology, Biochemistry, and Behavior·S Amir
Sep 30, 1983·Neuroscience Letters·S Amir
May 4, 1984·Neuroscience Letters·S Amir
Jan 1, 1989·Annals of the New York Academy of Sciences·J W HoladayN J Gurll
Apr 30, 1984·Brain Research·S AmirA Schachar
Jul 1, 1986·Resuscitation·W G BarsanS A Syverud
Apr 9, 1984·Brain Research·S Amir
Jan 2, 1984·Brain Research·S Amir
Jan 1, 1988·Research in Experimental Medicine. Zeitschrift Für Die Gesamte Experimentelle Medizin Einschliesslich Experimenteller Chirurgie·J D WatsonC J Hinds
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Anaphylaxis
Anaphylaxis is a serious allergic reaction that is rapid in onset and may cause death.
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