Autophagy in the dentin-pulp complex against inflammation

Oral Diseases
L Zhang, Z Chen

Abstract

The dentin-pulp complex is a highly specialized tissue for protecting the dental pulp. Odontoblasts are long-lived, hard-tissue-forming cells in the dentin-pulp complex and critically involved in inflammatory responses against invading pathogens. Autophagy is a highly conserved homeostasis mechanism of living cells under various stress conditions. Growing evidence in the literature addresses the role of autophagy in odontoblast differentiation and aging. This review summarizes the current knowledge about autophagy for the dentin-pulp complex in resisting inflammation.

References

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Mar 14, 2008·The EMBO Journal·Mónica A DelgadoVojo Deretic
Jan 8, 2011·Journal of Dental Research·E Couve, O Schmachtenberg
Jan 21, 2011·Nature·Beth LevineHerbert W Virgin
Aug 10, 2012·Autophagy·David R Soto-PantojaDavid D Roberts
Oct 12, 2012·Histochemistry and Cell Biology·Jing-wen YangZhi Chen
Nov 26, 2013·Frontiers in Physiology·Jean-Christophe FargesFlorence Carrouel
Oct 10, 2014·Journal of Dental Research·F PeiZ Chen
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Feb 24, 2016·Oral Diseases·T HuiL Ye
Feb 26, 2016·Journal of Dental Research·H S WangL Zhang

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Citations

Sep 28, 2021·Frontiers in Bioengineering and Biotechnology·Hanxin XuJiarong Liu
Oct 23, 2021·Journal of Biochemistry·Pujan JoshiDashzeveg Bayarsaihan

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