Cardiomyopathy mimicking left ventricular noncompaction in a patient with lupus nephritis

CEN Case Reports
Abdullah OzkokAli Riza Odabas

Abstract

A 37-year-old female patient was admitted with exertional dyspnea. Her serum creatinine was 2.4 mg/dL and anti-nuclear antibody was positive in a titer of 1/320. Renal biopsy revealed diffuse proliferative lupus nephritis. Echocardiography and cardiac magnetic resonance (MR) imaging showed increased apical trabeculations compatible with left ventricular noncompaction (LVNC), which is a rare genetic cardiomyopathy. The patient expressed a marked improvement in exertional dyspnea after the immune-suppressive treatment for systemic lupus erythematosus (SLE). Control echocardiography revealed a significant increase of ejection fraction. SLE may cause a kind of cardiomyopathy with high resemblance to LVNC. Discrimination of these two similar clinical entities is important because SLE-induced cardiomyopathy is potentially reversible after the immune-suppressive treatment for SLE.

References

Feb 2, 2002·Journal of the American College of Cardiology·Rolf JenniPhilipp A Kaufmann
Nov 6, 2002·Cardiology·Josef FinstererHans Feichtinger
May 1, 2004·Circulation·Josef FinstererGerhard Blazek
Jun 24, 2004·Circulation·Brian C WeifordKevin M Mulhern
Oct 13, 2005·Lupus·A DoriaM Petri
Mar 31, 2009·European Journal of Radiology·Memduh DursunEnsar Yekeler

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Citations

Jul 23, 2015·Journal of Clinical Imaging Science·Cristina I Olivas-ChaconLuis R Ramos-Duran
Nov 1, 2018·Romanian Journal of Internal Medicine = Revue Roumaine De Médecine Interne·Mihaela DobraniciGheorghe-Andrei Dan

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