No abstract listed.
Lymphocyte transformation induced by autologous cells. IV. Human T-lymphocyte proliferation induced by autologous or allogeneic non-T lymphocytes
Suppressor-cell antibody in systemic lupus erythematosus. Possible mechanism for suppressor-cell dysfunction
Increased spontaneous activity of antibody-forming cells in the peripheral blood of patients with active SLE
Editorial: Immune activation of B cells: evidence for 'one nonspecific triggering signal' not delivered by the Ig receptors
The interaction of alpha 2-macroglobulin with proteinases. Characteristics and specificity of the reaction, and a hypothesis concerning its molecular mechanism
IgM in bone marrow-derived lymphocytes. Synthesis, surface deposition, turnover and carbohydrate composition in unstimulated mouse B cells
Release of DNA in circulating blood and induction of anti-DNA antibodies after injection of bacterial lipopolysaccharides
A suggested mechanism for T lymphocyte activation: implications on the acquisition of functional reactivities
Clones of killer and helper T cells: growth requirements, specificity and retention of function in long-term culture
Mitogenic activation of human lymphocytes: a protein A plaque assay evaluation of polyclonal B-cell activators
Pathogenesis of lupus dermatoses in autoimmune mice. XIX. Attempts to induce subepidermal immunoglobulin deposition in MRL/Mp- +/+ mice
The effect of protease inhibitors on the polyclonal B cell activator from the serum of patients with rheumatoid arthritis
Monoreactive and polyreactive rheumatoid factors produced by in vitro Epstein-Barr virus-transformed peripheral blood and synovial B lymphocytes from rheumatoid arthritis patients
Identification of protein disulfide isomerase and calreticulin as autoimmune antigens in LEC strain of rats
Kinetics of inhibition of mitogen-induced proliferation of human lymphocytes by alpha 2-macroglobulin in serum-free medium
Plasma from rheumatoid arthritis patients does not contain abnormally high levels of alpha 2-macroglobulin-proteinase complexes
Degradation of a chromogenic substrate by alpha 2-macroglobulin from plasma of patients with rheumatoid arthritis
Polyclonal B-cell response to stimulation with Escherichia coli lipopolysaccharide in dietary protein restriction.
Autoimmune diseases occur as a result of an attack by the immune system on the body’s own tissues resulting in damage and dysfunction. There are different types of autoimmune diseases, in which there is a complex and unknown interaction between genetics and the environment. Discover the latest research on autoimmune diseases here.