PMID: 9421810Jan 9, 1998Paper

Could exogenous melatonin prevent sudden infant death syndrome?

Medical Hypotheses
C P Maurizi

Abstract

Hypoplastic pineal glands and decreased melatonin levels are found in sudden infant death syndrome. Excess oxygen radical production and inadequate radical quenching is suggested to cause oxidative stress in the brain of sudden infant death syndrome victims. Loss of intraventricular cerebrospinal fluid melatonin could play an important role in the disease process. Infants at risk might be protected by exogenous melatonin.

References

Sep 1, 1990·Neurobiology of Aging·L Volicer, P B Crino
Jun 1, 1994·Neurology·W D ParkerB K Kleinschmidt-DeMasters
Jun 15, 1996·Annals of the New York Academy of Sciences·R J ReiterJ M Guerrero

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