PMID: 7518201Jun 17, 1994Paper

Cytokines, neuropeptides, and reperfusion injury during magnesium deficiency

Annals of the New York Academy of Sciences
William B WeglickiJ H Kramer

Abstract

In summary, hypomagnesemia enhances reperfusion injury. We postulate that neurogenic inflammation, which occurs very early during hypomagnesemia, predisposes the myocardium to reperfusion injury by depleting endogenous antioxidants and recruiting inflammatory cells, which can participate in enhanced free radical production during postischemic reperfusion. Vitamin E supplements can prevent the occurrence of this enhanced injury possibly through the restoration of endogenous antioxidant defenses.

References

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Citations

Jul 5, 2006·Heart Failure Reviews·Maria Isabel Tejero-TaldoWilliam Bernard Weglicki
Sep 10, 2013·Journal of Inorganic Biochemistry·Regina M M OliveiraRose M Carlos
Jun 6, 2000·Biochimica Et Biophysica Acta·C Malpuech-BrugèreY Rayssiguier
Mar 12, 1999·Cardiovascular Surgery : Official Journal of the International Society for Cardiovascular Surgery·A ParolariP Biglioli
Mar 30, 2002·Critical Reviews in Oncology/hematology·J Anastassopoulou, T Theophanides
Mar 13, 2012·Annual Review of Nutrition·William B Weglicki
Apr 2, 2015·British Journal of Pharmacology·F CorriganR J Turner
Feb 6, 2008·The Veterinary Clinics of North America. Small Animal Practice·Peter Vajdovich
May 24, 2015·Transplantation Reviews·Steven Van Laecke, Wim Van Biesen

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