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Decreased Frequency and Secretion of CD26 Promotes Disease Progression in Indian Post Kala-azar Dermal Leishmaniasis

Journal of Clinical Immunology

Dec 9, 2015

Shibabrata MukherjeeMitali Chatterjee

Abstract

Leishmania, the causative organisms for leishmaniasis, reside in host macrophages and survive by modulating the microbicidal pathways via attenuation of the oxidative burst and/or suppression of cell-mediated immunity. As post kala-azar dermal leishmaniasis (PKDL), the dermal sequela of...read more

Mentioned in this Paper

Study
Flow Cytometry
Biochemical Pathway
T-Lymphocyte
Genus: Leishmania
DPP4 protein, human
TGFA protein, human
DPP4
Recombinant Interleukin-10
Dermis
3
1
10
Paper Details
References
  • References23
  • Citations3
123
  • References23
  • Citations3
1

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Decreased Frequency and Secretion of CD26 Promotes Disease Progression in Indian Post Kala-azar Dermal Leishmaniasis

Journal of Clinical Immunology

Dec 9, 2015

Shibabrata MukherjeeMitali Chatterjee

PMID: 26644312

DOI: 10.1007/s10875-015-0215-8

Abstract

Leishmania, the causative organisms for leishmaniasis, reside in host macrophages and survive by modulating the microbicidal pathways via attenuation of the oxidative burst and/or suppression of cell-mediated immunity. As post kala-azar dermal leishmaniasis (PKDL), the dermal sequela of...read more

Mentioned in this Paper

Study
Flow Cytometry
Biochemical Pathway
T-Lymphocyte
Genus: Leishmania
DPP4 protein, human
TGFA protein, human
DPP4
Recombinant Interleukin-10
Dermis
3
1
10

Similar Papers Found In These Feeds

Leishmaniasis

Leishmaniasis is a parasitic infection caused by Leishmania species and transmitted though the bite of phlebotomine sand flies. It is considered to be a neglected tropical disease. Leishmaniasis is often categorized as cutaneous (presenting with skin sores, mucocutaneous (presenting with skin and mucosal lesions), and visceral (affecting multiple organs). Find the latest research on leishmaniasis here.

Related Papers

Paper Details
References
  • References23
  • Citations3
123
  • References23
  • Citations3
1
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