Peripheral blood lymphocytes from six patients with congenital hypoplastic anemia suppressed erythroid cell formation by normal human bone marrow cells in response to erythropoietin in vitro. The results suggest that the anemia in these children has an immunologic basis.
Immunoregulation in hypersensitivity pneumonitis. I. Differences in T-cell and macrophage suppressor activity in symptomatic and asymptomatic pigeon breeders
T-lymphocyte--mediated granulopoietic failure. In vitro identification of prednisone-responsive patients
Sustained cyclosporine-induced erythropoietic response in identical male twins with diamond-blackfan anemia
Elevation of pyrimidine enzyme activities in the RBC of patients with congenital hypoplastic anaemia and their parents
Diamond-Blackfan syndrome: evidence for T-cell mediated suppression of erythroid development and a serum blocking factor associated with remission
The production of steel factor mRNA in Diamond-Blackfan anaemia long-term cultures and interactions of steel factor with erythropoietin and interleukin-3
Neutropenia in three patients with rheumatic disorders. Suppression of granulopoiesis by control-sensitive thymus-dependent lymphocytes
Aplastic anemia associated with bone marrow suppressor T-cell hyperactivity: successful treatment with antithymocyte globulin
Pure red cell aplasia: relationship between inhibitory activity of T cells to CFU-E and erythropoiesis
Bone marrow cells associated with heightened eosinophilopoiesis: an electron microscope study of murine bone marrow stimulated by Ascaris suum
Treatment of refractory aplastic anemia with plasmapheresis: report of a case in childhood with review of the literature
Partial restoration of local GVH reaction in cancer patients by depletion of theophylline-sensitive suppressor T-cells
Lymphocytes suppressing both immunoglobulin production and erythroid differentiation in hypogammaglobulinaemia
Regulation of autologous T-lymphocyte clones on in vitro growth of BFU-e: enhancing effects of both CD4- and CD8-positive clones
Anemia develops when your blood lacks enough healthy red blood cells. Anemia of inflammation (AI, also called anemia of chronic disease) is a common, typically normocytic, normochromic anemia that is caused by an underlying inflammatory disease. Here is the latest research on anemia.