Experimental autoimmune thymitis. An animal model of human myasthenia gravis
No abstract listed.
Desensitization of the nicotinic acetylcholine receptor: molecular mechanisms and effect of modulators
Electrophysiological studies of thymectomized and nonthymectomized acetylcholine receptor-immunized animal models of myasthenia gravis
Combined effects of a thymic peptide, thymopoietin and myasthenic patient sera in rat myotube culture
Induction of T-cell differentiation in vitro by thymin, a purified polypeptide hormone of the thymus
Contrasting biological activities of thymopoietin and splenin, two closely related polypeptide products of thymus and spleen
Calcium-dependent effect of the thymic polypeptide thymopoietin on the desensitization of the nicotinic acetylcholine receptor
Electrophysiological changes similar to those of myasthenia gravis in rats with experimental autoimmune thymitis
The role of the thymus in myasthenia gravis: immunohistological and immunological studies in 115 cases
Complete amino acid sequences of bovine thymopoietins I, II, and III: closely homologous polypeptides
Clinical features, pathogenesis, and treatment of myasthenia gravis: a supplement to the Guidelines of the German Neurological Society
Immunopathology of myasthenia gravis. Comparison of myasthenic sceletal muscle antibodies with rabbit and rat antibodies produced by immunization with skeletal muscle proteins
Comparative efficacy of various routes of administration of thymopentin (TP-5) with consideration of degradative mechanisms
Damage of skeletal muscle in rats by immunoglobulins. I. Pathophysiological data and lightmicroscopic observations
Autoimmune diseases occur as a result of an attack by the immune system on the body’s own tissues resulting in damage and dysfunction. There are different types of autoimmune diseases, in which there is a complex and unknown interaction between genetics and the environment. Discover the latest research on autoimmune diseases here.