Hepatitis C virus and the threshold of natural killer cell inhibition

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Hepatitis C virus and the threshold of natural killer cell inhibition

Abstract

References

Identification of four subsets of human CD3-CD16+ natural killer (NK) cells by the expression of clonally distributed functional surface molecules: correlation between subset assignment of NK clones and ability to mediate specific alloantigen recognition

In search of the 'missing self': MHC molecules and NK cell recognition

Peptide binding to MHC class I molecules: implications for antigenic peptide prediction

Peptide specificity in the recognition of MHC class I by natural killer cell clones

A p70 killer cell inhibitory receptor specific for several HLA-B allotypes discriminates among peptides bound to HLA-B*2705

Self and viral peptides can initiate lysis by autologous natural killer cells.

The direct binding of a p58 killer cell inhibitory receptor to human histocompatibility leukocyte antigen (HLA)-Cw4 exhibits peptide selectivity

Peptides isolated from HLA-Cw*0304 confer different degrees of protection from natural killer cell-mediated lysis

HLA gene and haplotype frequencies in the North American population: the National Marrow Donor Program Donor Registry

Functionally and structurally distinct NK cell receptor repertoires in the peripheral blood of two human donors

Human diversity in killer cell inhibitory receptor genes

NK cell inhibitory receptors prevent tyrosine phosphorylation of the activation receptor 2B4 (CD244)

Racial differences in HLA class II associations with hepatitis C virus outcomes

Natural killer cell immunoglobulin-like receptor (KIR) locus profiles in African and South Asian populations

Kinetics of the immune response during HBV and HCV infection

Host genetic determinants in hepatitis C virus infection

HLA and NK cell inhibitory receptor genes in resolving hepatitis C virus infection

Persistence of hepatitis C virus in a white population: associations with human leukocyte antigen class 1

Citations

Immune responses in hepatitis C virus infection and mechanisms of hepatitis C virus persistence

Innate immunity and hepatitis C virus infection: a microarray's view.

Chronic ethanol consumption inhibits hepatic natural killer cell activity and accelerates murine cytomegalovirus-induced hepatitis

Natural killer cells ameliorate liver fibrosis by killing activated stellate cells in NKG2D-dependent and tumor necrosis factor-related apoptosis-inducing ligand-dependent manners

Related Concepts

Related Feeds

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