HPA axis response to stress predicts short-term snack intake in obese women.

Appetite
Bradley M AppelhansBonnie J Spring

Abstract

Prior research has linked heightened cortisol reactivity to stress with increased food consumption. This pilot study tested corollaries of the hypothesis that cortisol stress reactivity promotes obesity. Thirty-four lean and obese women completed an acute stress task and a non-stressful control task in counterbalanced order. Contrary to expectations, higher post-stress cortisol was associated with decreased post-stress snack intake in obese women but was unrelated to snack intake in lean women. Stress also blunted an expected rise in hunger only among obese women. Findings suggest that some obese women may be more sensitive to short-term anorectic effects of HPA axis activation.

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Citations

Feb 16, 2016·Toxicological Research·Ki-Woong KimSeong-Kyu Kang
May 15, 2012·Physiology & Behavior·A Janet TomiyamaElissa S Epel
Nov 1, 2011·Appetite·Tatjana van StrienCarolina de Weerth
Oct 21, 2010·Appetite·Kristin L SchneiderSherry L Pagoto
Dec 28, 2010·Eating Behaviors·Bradley M AppelhansSherry L Pagoto
Mar 2, 2012·Psychoneuroendocrinology·Vasiliki MichopoulosMark E Wilson
Sep 25, 2012·Psychoneuroendocrinology·Tatjana van StrienCarolina de Weerth
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Feb 19, 2020·Translational Psychiatry·Benedict HerhausKatja Petrowski
Aug 7, 2019·Psychosomatic Medicine·Matthew R NagyRebecca E Hasson

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