PMID: 24526778DOI: 10.1182/blood-2013-12-542506Feb 15, 2014Paper

In APS, two A1's are better than one!

Blood
Bill Giannakopoulos, Steven A Krilis

Abstract

No abstract listed.

References

Anti-phospholipid antibodies are directed against a complex antigen that includes a lipid-binding inhibitor of coagulation: beta 2-glycoprotein I (apolipoprotein H)

Jun 1, 1990·Proceedings of the National Academy of Sciences of the United States of America·H P McNeilS A Krilis

Epitope studies with anti-beta 2-glycoprotein I antibodies from autoantibody and immunized sources

Sep 2, 2000·Journal of Autoimmunity·S W ReddelS A Krilis

International consensus statement on an update of the classification criteria for definite antiphospholipid syndrome (APS)

Jan 20, 2006·Journal of Thrombosis and Haemostasis : JTH·S MiyakisS A Krilis

Platelets express three different splice variants of ApoER2 that are all involved in signaling

May 2, 2007·Journal of Thrombosis and Haemostasis : JTH·M T T PenningsP G de Groot

Apolipoprotein E receptor 2 is involved in the thrombotic complications in a murine model of the antiphospholipid syndrome

Dec 2, 2010·Blood·Zurina Romay-PenabadSilvia S Pierangeli

A novel dimeric inhibitor targeting Beta2GPI in Beta2GPI/antibody complexes implicated in antiphospholipid syndrome

Dec 24, 2010·PloS One·Alexey KolyadaNatalia Beglova

The pathogenesis of the antiphospholipid syndrome

Mar 15, 2013·The New England Journal of Medicine·Bill Giannakopoulos, Steven A Krilis

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Antiphospholipid Syndrome

Antiphospholipid syndrome or antiphospholipid antibody syndrome (APS or APLS), is an autoimmune, hypercoagulable state caused by the presence of antibodies directed against phospholipids.

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