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Increased prevalence of non-secretors in patients with Graves' disease: evidence for an infective aetiology?

British Medical Journal

Apr 23, 1988

A CollierD M Weir

PMID: 3132249

Abstract

No abstract listed.

Mentioned in this Paper

Disease Susceptibility
Graves Disease
Thyrotropin Receptor
Blood Group Antigens
Bacterial Infections
Paper Details
References
  • References2
  • Citations9
  • References2
  • Citations9
1

Similar Papers Found In These Feeds

Graves Disease

Graves disease is common form of hyperthyroidism with a diffuse hyperplastic goiter. It is an autoimmune disorder that produces antibodies against thyroid stimulating hormone receptor. These autoantibodies activate the TSH receptor, thereby stimulating the thyroid gland and hypersecretion of thyroid hormones.Discover the latest research on Graves disease here.

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Secretor state of patients with insulin dependent or non-insulin-dependent diabetes mellitus

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Increased prevalence of non-secretors in patients with Graves' disease: evidence for an infective aetiology?

British Medical Journal

Apr 23, 1988

A CollierD M Weir

PMID: 3132249

DOI:

Abstract

No abstract listed.

Mentioned in this Paper

Disease Susceptibility
Graves Disease
Thyrotropin Receptor
Blood Group Antigens
Bacterial Infections

Similar Papers Found In These Feeds

Graves Disease

Graves disease is common form of hyperthyroidism with a diffuse hyperplastic goiter. It is an autoimmune disorder that produces antibodies against thyroid stimulating hormone receptor. These autoantibodies activate the TSH receptor, thereby stimulating the thyroid gland and hypersecretion of thyroid hormones.Discover the latest research on Graves disease here.

Related Papers

British Medical Journal

Secretor state of patients with insulin dependent or non-insulin-dependent diabetes mellitus

British Medical JournalOctober 24, 1987
C C BlackwellB F Clarke
British Medical Journal

Secretor state of patients with insulin dependent or non-insulin dependent diabetes mellitus

British Medical JournalDecember 12, 1987
P J LameyT W MacFarlane
Paper Details
References
  • References2
  • Citations9
  • References2
  • Citations9
1
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