Iron and lymphocytes: reciprocal regulatory interactions

Anemia develops when your blood lacks enough healthy red blood cells. Anemia of inflammation (AI, also called anemia of chronic disease) is a common, typically normocytic, normochromic anemia that is caused by an underlying inflammatory disease. Here is the latest research on anemia.

Iron and lymphocytes: reciprocal regulatory interactions

Abstract

Citations

Decreased CD8-p56lck activity in peripheral blood T-lymphocytes from patients with hereditary haemochromatosis

Hepcidin: the missing link between hemochromatosis and infections.

Transfusion-related biologic effects and free hemoglobin, heme, and iron

Iron metabolism and HIV infection: reciprocal interactions with potentially harmful consequences?

Human cytomegalovirus protein US2 interferes with the expression of human HFE, a nonclassical class I major histocompatibility complex molecule that regulates iron homeostasis

Cytokine-mediated regulation of iron transport in human monocytic cells.

Diagnosis and management of iron-related anemias in critical illness

Lymphocyte gene expression signatures from patients and mouse models of hereditary hemochromatosis reveal a function of HFE as a negative regulator of CD8+ T-lymphocyte activation and differentiation in vivo

The macrophage: a cellular factory at the interphase between iron and immunity for the control of infections

Iron and inflammatory bowel disease

Iron and the risk of infection

Iron and immunity: a double-edged sword

Benefits and risks of iron therapy for chronic anaemias

The hemochromatosis protein HFE 20 years later: An emerging role in antigen presentation and in the immune system

Linkage of cell-mediated immunity to iron metabolism

Differential effects of iron load on basal and interferon-gamma plus lipopolysaccharide enhance anticryptococcal activity by the murine microglial cell line BV-2

Iron and anemia of chronic disease

Pathogenesis and treatment of anaemia of chronic disease

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