Labetalol-induced hepatitis in a patient with chronic hepatitis B infection.

The Journal of Clinical Hypertension
Mark A Marinella

Abstract

Drug-induced hepatotoxicity is an important cause of hepatocellular injury. Hepatic necrosis may range from asymptomatic elevations in transaminases to fulminant hepatic failure and death. A 50-year-old Asian man developed moderate elevations of hepatic transaminases after commencing labetalol therapy for a hypertensive cerebral hemorrhage. Extensive evaluation revealed evidence of chronic hepatitis B infection, of which the patient was previously not aware. Hepatic transaminases normalized after discontinuation of labetalol.

References

Aug 1, 1990·Annals of Internal Medicine·J A ClarkL A Tanner
Sep 26, 1997·Southern Medical Journal·G L Davis

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Citations

May 4, 2013·Alimentary Pharmacology & Therapeutics·J H Lewis, J G Stine
Oct 1, 2009·Arhiv za higijenu rada i toksikologiju·Alenka AksamijaEva Jendris
Dec 22, 2019·Hepatology : Official Journal of the American Association for the Study of Liver Diseases·Cara L MackAlbert J Czaja
Apr 26, 2019·Drug and Therapeutics Bulletin·Nicole KapralBrian Markle
Feb 11, 2005·Current Opinion in Gastroenterology·David Novak, James H Lewis
Dec 16, 2020·Journal of Pharmacy Practice·John A Dougherty, Christopher T Elder

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