Mitochondrial fission and stemness in prostate cancer

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Mitochondrial fission and stemness in prostate cancer

Abstract

References

Cancer epigenetics: tumor heterogeneity, plasticity of stem-like states, and drug resistance

The mechanisms behind the therapeutic activity of BET bromodomain inhibition

Stem cells. Asymmetric apportioning of aged mitochondria between daughter cells is required for stemness

Autophagy maintains stemness by preventing senescence.

Mitochondrial Dynamics in Regulating the Unique Phenotypes of Cancer and Stem Cells

Cancer Stem Cells: The Architects of the Tumor Ecosystem

Epigenetic Control of Mitochondrial Fission Enables Self-Renewal of Stem-like Tumor Cells in Human Prostate Cancer

Clonal Evolution and Epithelial Plasticity in the Emergence of AR-Independent Prostate Carcinoma

Citations

ARV-825-induced BRD4 protein degradation as a therapy for thyroid carcinoma.

Bromodomain-containing protein 4 inhibitor JQ1 promotes melanoma cell apoptosis by regulating mitochondrial dynamics.

Methods Mentioned

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