Mitochondrial Proteins Coded by Human Tumor Viruses

Frontiers in Microbiology
Ilaria CavallariVincenzo Ciminale

Abstract

Viruses must exploit the cellular biosynthetic machinery and evade cellular defense systems to complete their life cycles. Due to their crucial roles in cellular bioenergetics, apoptosis, innate immunity and redox balance, mitochondria are important functional targets of many viruses, including tumor viruses. The present review describes the interactions between mitochondria and proteins coded by the human tumor viruses human T-cell leukemia virus type 1, Epstein-Barr virus, Kaposi's sarcoma-associated herpesvirus, human hepatitis viruses B and C, and human papillomavirus, and highlights how these interactions contribute to viral replication, persistence and transformation.

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Citations

May 7, 2019·Frontiers in Cellular and Infection Microbiology·María Maximina B Moreno-AltamiranoFrancisco Javier Sánchez-García
Dec 11, 2019·British Journal of Cancer·Vittoria RaimondiVincenzo Ciminale
May 14, 2020·Retrovirology·Maria OmslandGenoveffa Franchini
Sep 25, 2020·Microbiology and Immunology·Nelly Gakii Muriungi, Keiji Ueda

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Methods Mentioned

BETA
2-hybrid
co-immunoprecipitation
transfection

Software Mentioned

Clustal Omega

Related Concepts

Related Feeds

Apoptosis

Apoptosis is a specific process that leads to programmed cell death through the activation of an evolutionary conserved intracellular pathway leading to pathognomic cellular changes distinct from cellular necrosis

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