Resistance to herpes simplex virus - type 1 (HSV-1)

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Resistance to herpes simplex virus - type 1 (HSV-1)

Abstract

Citations

In vivo ablation of CD11c-positive dendritic cells increases susceptibility to herpes simplex virus type 1 infection and diminishes NK and T-cell responses

Entry of herpes simplex virus 1 and other alphaherpesviruses via the paired immunoglobulin-like type 2 receptor alpha.

Growth and cytopathogenicity of herpes simplex virus in a macrophage cell line, RAW264: A good indicator of intraperitoneal pathogenicity

The immune response to herpes simplex virus type 1 infection in susceptible mice is a major cause of central nervous system pathology resulting in fatal encephalitis.

Resistance to pseudorabies virus with enhanced interferon production and natural killer cell activity in mice treated with serum thymic factor

Imiquimod 5-percent cream does not alter the natural history of recurrent herpes genitalis: a phase II, randomized, double-blind, placebo-controlled study.

The case for immunomodulatory approaches in treating HSV encephalitis

Effects of genetic resistance against Herpes simplex virus in vaginally infected mice

Re-evaluating natural resistance to herpes simplex virus type 1

A locus on mouse chromosome 6 that determines resistance to herpes simplex virus also influences reactivation, while an unlinked locus augments resistance of female mice

A NK complex-linked locus restricts the spread of herpes simplex virus type 1 in the brains of C57BL/6 mice

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