Stem cells, hormones and pituitary adenomas

Journal of Neuroendocrinology
Andy Levy

Abstract

We still do not understand the pathogenesis of the majority of pituitary adenomas or why, once formed, their behaviour tends to be so benign. Understanding trophic activity in the normal pituitary may be the key. Despite the fact that changes in indices of cell division and programmed cell death that are too small to measure can produce highly significant fluxes in cell populations, little by little, the fascinating patterns of integrated responses of pituitary cells to hormonal stimuli are now being revealed.

Citations

Sep 3, 2013·Molecular and Cellular Endocrinology·Hugo Vankelecom, Jianghai Chen
Oct 29, 2010·International Journal of Rheumatology·Antonio AversaGiovanni Spera
Apr 9, 2016·Journal of Clinical Medicine·Saishu YoshidaYukio Kato
May 19, 2017·Current Opinion in Endocrinology, Diabetes, and Obesity·Montserrat Lara-VelazquezAlfredo Quinones-Hinojosa
Jul 24, 2010·Neurosurgery·João Paulo C de AlmeidaAlfredo Quiñones-Hinojosa
Dec 15, 2010·The European Journal of Neuroscience·Hugo Vankelecom
Jan 5, 2012·Journal of Biological Rhythms·David G Hazlerigg, Gerald A Lincoln

Related Concepts

Adenoma, Trabecular
M Phase, Mitotic
Pituitary Hormones
Pituitary Carcinoma
Colony-forming Unit

Related Feeds

Apoptosis

Apoptosis is a specific process that leads to programmed cell death through the activation of an evolutionary conserved intracellular pathway leading to pathognomic cellular changes distinct from cellular necrosis