T-wave abnormalities of intermittent left bundle-branch block
Ischemia is traditionally considered a cause of intermittent left bundle-branch block (LBBB), and some patients have right precordial T-wave inversion in the normally conducted beats. Clinical correlates of T-wave abnormalities were examined in 46 consecutive patients with intermittent LBBB. Thirty-three patients (72%) had at least transient right precordial (V-14) T-wave inversion suggesting ischemia in normally conducted beats. Seventeen such patients had no evidence of coronary heart disease, including five with normal arteriograms. During LBBB conduction, T-wave abnormalities (upright T-waves I, aVL, V5-6) were frequent (48%) and more common than among patients with permanent LBBB (p less than 0.005). The T-wave abnormalities during LBBB conduction occurred in the absence of coronary heart disease in nine patients, including two with normal arteriograms. Thus, right precordial T-wave inversion may result from recent LBBB itself, associated with T-wave abnormalities during the LBBB, in the absence of coronary artery disease.
Ischemia-induced transient left bundle branch block during exercise documented by 201Tl perfusion imaging
Repolarization abnormalities after catheter ablation of accessory atrioventricular connections with radiofrequency current
Persistent T-wave changes after radiofrequency catheter ablation of an accessory connection (Wolff-parkinson-white syndrome) are caused by "cardiac memory"
Memory T waves after radiofrequency catheter ablation of accessory atrioventricular connections in Wolff-Parkinson-White syndrome
Cardiac memory versus likelihood of ischemic heart disease in hypertensive patients with ventricular repolarization abnormalities after repetitive uniform ventricular extrasystoles
Torsade de pointes secondary to d,l-sotalol after catheter ablation of incessant atrioventricular reentrant tachycardia--evidence for a significant contribution of the "cardiac memory"
Repolarization changes in patients with heart failure receiving cardiac resynchronization therapy-signs of cardiac memory
Effect of short-term cardiac memory on ventricular electrical restitution and QT intervals in humans
Deep T wave inversions after value replacement for chronic aortic regurgitation: possible conversion from a diastolic to systolic overload pattern
Cardiac memory, a surface electrocardiographic clue in the differential diagnosis of ongoing narrow complex tachycardia
Vectorcardiography shows cardiac memory and repolarization heterogeneity after ablation of accessory pathways not apparent on ECG
Rate-related left bundle branch block and cardiac memory in a patient with bradycardia: Case report and literature review
Persistent T-wave changes after alteration of the ventricular activation sequence. New insights into cellular mechanisms of 'cardiac memory'
Slow and long-lasting modulation of myocardial repolarization produced by ectopic activation in isolated rabbit hearts. Evidence for cardiac "memory"
Intermittent left bundle branch block: anatomic substrate as reflected in the electrocardiogram during normal conduction
T wave changes persisting after ventricular pacing in canine heart are altered by 4-aminopyridine but not by lidocaine. Implications with respect to phenomenon of cardiac 'memory'
Prognostic value and temporal behavior of the planar QRS-T angle in patients with nonischemic cardiomyopathy
ECG simulators of infarction. Part II: Pathophysiology and differential diagnosis of pseudo-infarction ST-T patterns
Pacing-induced electrophysiological remodeling in hypertrophic obstructive cardiomyopathy--observations on cardiac memory
Intermittent left bundle branch block: an overlooked cause of electrocardiographic changes that mimic high-grade stenosis of the left anterior descending coronary artery
Cardiac Conduction System
The cardiac conduction system is a specialized tract of myocardial cells responsible for maintaining normal cardiac rhythm. Discover the latest research on the cardiac conduction system here.