The academic lineage of Sir John Carew Eccles (1903-1997)

Progress in Neurobiology
Douglas G Stuart, Patricia A Pierce

Abstract

This report reviews the academic lineage of Sir John Eccles; who trained him, whom he then trained and with whom he collaborated, and the subsequent impact of his trainees and collaborators on neuroscience and other areas. In a post-training career at five institutions in four countries (Great Britain, Australia, New Zealand, back to Australia, USA) and during retirement in Switzerland, Eccles trained and collaborated with over 180 people (mostly neuroscientists) from 21 countries. Most of them have had stellar research and training records that span the cellular-behavioral-philosophical spectrum of neuroscience, with a focus from peripheral neuromuscular issues to the forebrain. Some have been equally distinguished in other areas of biomedical science. Eccles' academic contributions and lineage are a valuable colloquium topic in a neuroscience training program. His experimental work spanned much of the 20th C before the recent emphasis on the application of the techniques of molecular biology. He continually sought to integrate information from the cellular to the systems and behavioral levels of analysis and synthesis. He also devoted a substantial amount of his intellectual effort to the mind-brain and other philosophical is...Continue Reading

References

Feb 16, 1977·Experimental Brain Research·P Buser, J C Eccles
Jan 1, 1977·Annual Review of Physiology·J C Eccles
Dec 1, 1992·Proceedings of the National Academy of Sciences of the United States of America·F Beck, J C Eccles
Dec 23, 1989·BMJ : British Medical Journal·R Eccles
Jan 1, 1972·Brain, Behavior and Evolution·G Bertrand
Jan 1, 1982·Archiv für Psychiatrie und Nervenkrankheiten·J Eccles
Jul 11, 1997·Science·R A Nicoll
May 24, 2001·Perspectives in Biology and Medicine·A G Karczmar
Jan 1, 1995·Biographical Memoirs of Fellows of the Royal Society·S Grillner
Jan 1, 1978·Biographical Memoirs of Fellows of the Royal Society·J Eccles, W Feindel
Oct 20, 2001·Journal of the History of the Neurosciences·C Borck
Oct 20, 2001·Biographical Memoirs of Fellows of the Royal Society·B B Boycott
Jul 20, 2002·Progress in Neurobiology·M R Bennett, P M S Hacker
Aug 1, 1952·The Journal of Physiology·L G BROCKJ C ECCLES
Feb 1, 1953·The Journal of Comparative Neurology·C B B DOWNMANA K MCINTYRE
Jan 1, 1952·Cold Spring Harbor Symposia on Quantitative Biology·J W WOODBURY, H D PATTON
Sep 1, 1953·The Journal of Physiology·J C ECCLES, A K McINTYRE
Feb 1, 1960·The Journal of Physiology·A J BULLERR M ECCLES
Aug 1, 1964·The Journal of Comparative Neurology·F D ANDERSONM M CHAMBERS
May 2, 2006·Progress in Neurobiology·Jonathan R Wolpaw, Jonathan S Carp
May 2, 2006·Progress in Neurobiology·Robert E Burke
May 3, 2006·Progress in Neurobiology·William D Willis
May 6, 2006·Progress in Neurobiology·Benjamin Libet
May 24, 2006·Progress in Neurobiology·Hans Hultborn
May 27, 2006·Progress in Neurobiology·Robert M Brownstone
Jun 9, 2006·Progress in Neurobiology·Masao Ito
Sep 18, 1928·The Journal of Physiology·R S Creed
Feb 28, 1929·The Journal of Physiology·J C Eccles

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Citations

Mar 21, 2012·Journal of the History of the Neurosciences·Marianne Fillenz
Jul 26, 2011·Brain Research·Douglas G Stuart, Robert M Brownstone
Jun 28, 2008·Brain Research Reviews·Douglas G Stuart, Hans Hultborn
May 15, 2007·Brain Research Reviews·François Clarac, Edouard Pearlstein
May 27, 2006·Progress in Neurobiology·Robert M Brownstone
May 6, 2006·Progress in Neurobiology·Benjamin Libet
Jun 9, 2006·Progress in Neurobiology·Masao Ito
May 2, 2006·Progress in Neurobiology·Robert E Burke
May 3, 2006·Progress in Neurobiology·William D Willis
May 2, 2006·Progress in Neurobiology·Jonathan R Wolpaw, Jonathan S Carp
Nov 1, 2015·Journal of Neurology·Vincent von Hoeckendorf, Frank W Stahnisch

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