The battlefield at arteriovenous crossroads: invading arterial smooth muscle cells occupy the outflow tract of fistulas

Kidney International
Joris I Rotmans, Taisiya Bezhaeva

Abstract

There is an ongoing debate about the anatomical origin of the neointimal cells that are responsible for venous stenotic lesions in arteriovenous fistulas. Liang and co-workers show that vascular smooth muscle cells from the feeding artery contribute substantially to venous intimal hyperplasia in a murine AVF model. In addition, they show that increased Notch signaling is the driving force behind FSP-1-mediated migration of these cells to the venous outflow tract.

References

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Feb 12, 2005·American Journal of Physiology. Heart and Circulatory Physiology·Jo G R De MeyMarijke M W Sanders
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Apr 2, 2013·Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association·Tonia C RothuizenJoris I Rotmans
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Citations

Jul 4, 2017·Cells·Eline E van HaaftenNicholas A Kurniawan

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