The advent of the fibreoptic gastroscope with biopsy facilities has provided the means of obtaining biopsy specimens under direct vision from any part of the stomach. This creates new opportunities for the study of chronic gastritis, and, in particular, its evolution, topographical location, and causal relationships. On the basis of an experience with more than 2,500 biopsy specimens we have outlined a method for their systematic examination and have proposed a classification of chronic gastritis. This classification includes the type of mucosa, the type and stage of activity of the gastritis, and the presence and type of metaplasia. The classification is sufficiently flexible to allow within it quantitative assessment of individual histological features.
Interrelationships among gastric mucosal morphology, secretion, and motility in peptic ulcer disease
Intragastric distribution and gastric emptying of solids and liquids in functional dyspepsia. Lack of influence of symptom subgroups and H. pylori-associated gastritis
Gastric argyrophil (enterochromaffin-like), gastrin, and somatostatin cells after proximal selective vagotomy in man
Helicobacter pylori infection induces a decrease in immunoreactive-somatostatin concentrations of human stomach
Epithelial cell proliferation in human fundic and antral mucosae. Influence of superselective vagotomy and relationship with gastritis
Location of peptic ulcers in relation to antral and fundal gastritis by chromoendoscopic follow-up examinations
Gastroduodenal inflammation in patients with non-ulcer dyspepsia. A controlled endoscopic and morphometric study
Effect of cigarette smoking on extent of acid-secreting area and intestinal metaplasia in the stomach.
Helicobacter colonization and histopathological profile of chronic gastritis in patients with or without dyspepsia, mucosal erosion and peptic ulcer: a morphological approach to the study of ulcerogenesis in man
Bile reflux and degree of gastritis after highly selective vagotomy, truncal vagotomy, and partial gastrectomy for duodenal ulcer
Screening procedures for identifying patients after gastric operations at high risk of developing premalignant histological changes
Foveolar hyperplasia following partial gastrectomy results from expansion of surface mucous cell compartment
Low incidence of clinically significant gastroduodenal pathology despite a high incidence of Helicobacter pylori infection
Eicosanoid synthesis and Helicobacter pylori associated gastritis: increase in leukotriene C4 generation associated with H. pylori colonization
Non-ulcer dyspepsia and Helicobacter pylori in type 2 diabetic patients: association with autonomic neuropathy
Changes in gastric mucosa and luminal environment during acid-suppressive therapy: a review in depth
Low-dose lansoprazole and clarithromycin plus metronidazole vs. full-dose lansoprazole and clarithromycin plus amoxicillin for eradication of Helicobacter pylori infection
Effect of lafutidine, a novel histamine H2-receptor antagonist, on monochloramine-induced gastric lesions in rats: role of capsaicin-sensitive sensory neurons
Helicobacter pylori infection in childhood: results of management with ranitidine bismuth citrate plus amoxicillin and tinidazole
Monochloramine impairs mucosal blood flow response and healing of gastric lesions in rats: relation to capsaicin-sensitive sensory neurons
Colonization by Helicobacter pylori and its relationship to histological changes in the gastric mucosa in portal hypertension
Atrophic Gastritis is a process where gastric glandular cells are lost and replaced with firbous tissues, as a result of chronic inflammation. Learn more about Atrophic Gastritis here.