The Toxoplasma Acto-MyoA motor complex is important but not essential for gliding motility and host cell invasion
bioRxiv
Jan 15, 2014
Saskia EgarterMarkus Meissner
PMID: 990001800
DOI: 10.1101/001800
Abstract
Apicomplexan parasites are thought to actively invade the host cell by gliding motility. This movement is powered by the parasite own actomyosin system and depends on the regulated polymerisation and depolymerisation of actin to generate the force for gliding and host cell penetration. ...read more
Mentioned in this Paper
MLC1 gene
MLC1 protein, human
Motility
TRAF3IP2 gene
Entry into Host Cell
Glideosome-associated protein 45, Plasmodium falciparum
Regulation of Protein Polymerization
Host Cell
Megalencephalic Leukoencephalopathy With Subcortical Cysts
Parasites
7
3
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The Toxoplasma Acto-MyoA motor complex is important but not essential for gliding motility and host cell invasion
Abstract
Apicomplexan parasites are thought to actively invade the host cell by gliding motility. This movement is powered by the parasite own actomyosin system and depends on the regulated polymerisation and depolymerisation of actin to generate the force for gliding and host cell penetration. ...read more
Mentioned in this Paper
MLC1 gene
MLC1 protein, human
Motility
TRAF3IP2 gene
Entry into Host Cell
Glideosome-associated protein 45, Plasmodium falciparum
Regulation of Protein Polymerization
Host Cell
Megalencephalic Leukoencephalopathy With Subcortical Cysts
Parasites
7
3
Related Papers
Paper Details
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References currently unavailable
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