PMID: 9552402Jan 1, 1997Paper

Viruses and the cell cycle

Progress in Cell Cycle Research
A Op De Beeck, P Caillet-Fauquet

Abstract

Viruses depend on the host's machineries to replicate and express their genome. Actively replicating cells have large pools of deoxynucleotides and high levels of key enzyme activities that viruses exploit to their own needs. Some viruses have developed strategies for driving quiescent cells into the S phase of the cell cycle, e.g. adenovirus, others, such as parvovirus, wait until the host itself begins to replicate. Viruses may also force the host cell to stay in a favourable phase, e.g. Epstein-Barr virus, or, if necessary, they may inhibit apoptotic cell death, e.g. human cytomegalovirus. In this review, we focus on the different strategies that viruses use to create in infected cells an environment favourable to the accomplishment of the viral life cycle through acting on cell cycle regulators.

Citations

Apr 5, 2002·Journal of Virology·Ralph FeuerJ Lindsay Whitton
Sep 16, 2004·Journal of Virology·Chun-Jen ChenShinji Makino
May 26, 2005·Cytometry. Part a : the Journal of the International Society for Analytical Cytology·David M AsmuthR Allen White
Aug 4, 2005·Journal of Neuro-oncology·Alfredo Quiñones-HinojosaMichael W McDermott
Feb 16, 2010·Veterinary Research·Karin Hoelzer, Colin R Parrish
Mar 16, 2007·FEBS Letters·Sally M HarrisonJulian A Hiscox
Jan 26, 2005·Journal of Cellular Biochemistry·Richard A KatzAnna Marie Skalka
May 14, 2004·Journal of Virology·Chun-Jen Chen, Shinji Makino
Jan 10, 2008·Cellular Microbiology·Robert E MolestinaAnthony P Sinai
May 9, 2016·Archives of Virology·Ahmed S Abdel-MoneimTaha Z M Mahran
Jan 19, 2006·Clinical Microbiology Reviews·Robert S FujinamiJ Lindsay Whitton
Jan 15, 2009·Genetica·L P G D'ArceE T Sakamoto-Hojo
Feb 28, 2002·Journal of Virology·George J PoggioliKenneth L Tyler
Sep 22, 2000·Virology·P GhazalA Angulo

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